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Injuries
Department of Orthopaedic Surgery
Fatmawati General Hospital
LUTHFI GATAM
ORTHOPAEDIC SPINE CONSULTANT
OVERVIEW
Significant percentage of all
emergency room care rendered.
In FGH, over 6 month period, survey
showed chief complaint of
musculoskeletal problem comprised
approx. 11% of all patients (1 in 9).
Musculoskeletal Injuries
ED physician needs a coherent,
systematic approach to orthopaedic
complaints.
Severity ranges from trivial sprains to
life- or limb-threatening trauma.
Often acute trauma is the cause of the
presentation.
Pain and decreased ROM are the main
symptoms.
FRACTURES
Fractures
A partial or complete break in a bone.
Bone is the tissue in the human body that heals
by regeneration without scarring.
For regeneration to occur the bone must be
immobilized to allow uninterrupted formation
of new bone.
Fractures
New Bone Formation:
A hematoma forms between realigned Fx fragments.
Hematopoietic cells in the hematoma secrete growth
factors (GFs).
GFs stimulate formation of granulation tissue at the
Fx ends, slowly resorbing the hematoma.
A primary callus forms, progressing from a soft
callus to hard callus.
Final phase of healing: during remodeling the bone
reassumes its original architecture.
Fractures
Nomenclature of fractures is essential to
successful Fx management in the ED
Adequate description:
Open or closed?
Which bone involved?
Location within the bone?
Direction of the main fracture line?
Number of fragments?
Alignment and displacement of the fragments?
Complications?
Closed Fracture
Open Fractures
ORTHOPEDIC EMERGENCY
Immediate control of hemorrhage.
Splinting +/- reduction.
Copious irrigation.
Early administration of sufficient analgesia,
appropriate antibiotics, and tetanus
prophylaxis.
Emergent consultation w/ orthopaedics for all
Type II and Type III open Fxs (and some Type
Is).
Fractures-Open Classification
I <1cm long, minimal contamination, low energy force
ABX: 1st/2nd Ceph for 3d
II >1cm long, moderate contamination and force
1st/2nd Ceph plus aminoglyc for 3d
ABX:
PELVIC FRACTURES
Pelvic Fractures
Clinical Evaluation
Any patient assessment begins with the ABCs
(Airway, Breathing, Circulation)
Complete neurologic and vascular exam
Have high suspicion of intra-abdominal injuries
Physical exam:
Ecchymosis or contusion around hips, perineum
Pelvic instability with stressing
Suspect if signs of urologic/gyn findings: blood at
urethral meatus, high-riding prostate
High force mechanisms also associated (mvc, femur
fx)
Pelvic Fracture-Management
#1--Control of bleeding vessels
In hemodynamically unstable pts, consider
angiography
#2--Surgical management of the broken
bone can proceed LATER after lifethreatening conditions are controlled.
Management
ABCs
Neurovascular exam (vascular +/- nerve injury)
Splint involved extremity
Reduction decreases pain, bleeding
Fracture Complications
Vascular Injuries
Most commonly occur in open Fxs, Fx-dislocations, or
widely displaced Fxs and at sites where the vessels lie in
close proximity to the bone or @ sites where the vessels
are held in a relatively fixed position.
Fracture Complications
Nerve Injuries
Occur more frequently than vascular injuries in
assoc. w/ Fx.
Can occur 2/2 blunt trauma, along path of
penetrating trauma, or be caused by the Fx
fragments themselves.
Nerves are @ increased risk of injury when they
are superficial to the skin, lie close to the bone, or
span a joint, making them susceptible to stretch
injury.
Fracture Complications
Fat Emboli Syndrome (FES)
Most common form of non-thrombotic
embolism.
Single or multiple long bone fractures in young
or pelvic/hip fractures in elderly predispose to
FES.
20% of patients w/ pelvic or long bone
fractures have detectable fat droplets in their
blood.
Fracture Complications
Fat Emboli Syndrome (FES)
Vast majority remain asymptomatic
Has characteristic clinical course:
1. Fracture sustained.
2. Other than fracture-associated pain, patient is
asymptomatic for 12-36 hours.
3. Sudden onset of life-threatening syndrome characterized
by rapid cardiopulmonary and neurologic deterioration,
agitation, hallucinations, delirium, coma, hypoxia, dyspnea,
tachypnea, and tachycardia leading to DIC and ARDS.
Fracture Complications
Fracture Complications
Fracture Complications
Fracture Complications
CRUSH INJURY
Crush Injuries
First descriptions from military records,
bombings in England and Europe during world
wars
Now more commonly seen in natural disasters,
building collapse, acts of terrorism, after
poisioning, after drug overdoses
Injury results from prolonged continuous
pressure on a body part, typically an extremity
Crush Injuries
Under direct pressure, cellular ischemia incurred
causing loss cellular integrity
Cells leak K+ and myoglobin
Influx of ions into the cells causing irreversible
cell death
Can have large fluid volume shifts
Electrolyte abnormalities:
Hyperkalemia, hyperphosphatemia, myoglobinemia,
hypocalcemia, metabolic acidosis
Crush Injuries
Myoglobin concentrates in
the renal tubules
obstructive nephropathy
acute renal failure
When ARF occurs,
mortality 20-40%
Arrythmias
Concern for sepsis with
devitalized tissue
Crush Injuries-Treatment
Early consideration /recognition
Fluid resuscitation
ARF approaches 100% if hydration
delayed >12 hours
COMPARTMENT
SYNDROME
Compartment Syndrome
Occurs when pressure w/i soft tissues in a
fixed body compartment increases to level
that exceeds venous pressure,
compromising venous blood flow, and
limiting capillary perfusion.
Leads to muscle ischemia and necrosis.
TRUE ORTHOPEDIC EMERGENCY
Compartment Syndrome
Contributing Factors
External:
Conditions that reduced size of muscle
compartment (casts/splints); occlusive
dressing; eschar of burns
Internal:
Conditions that increase compartment volume:
bleeding, swelling, fluid extravasation into
tissue
CS-Recognition
Suspect with long bone fx, crush injuries
Presents as pain out of proportion to
physical findings, +/- hypoesthesia,
pulselessness (late).
Compartment Syndrome
Compartment syndrome
should be suspected in
long bone Fxs and Fxs
associated w/ significant
vascular injuries or
pronounced swelling.
Intra-compartment pressures
must be measured once the
issue of compartment
syndrome is raised.
SPINAL INJURY
Spinal Injuries
Devastating injuries
>80% occur in young males
Motor vehicle accidents, falls from height,
gunshot wound
Worrisome presentations:
Spinal Injuries
Additional risk factors for spinal PAIN:
Metastatic cancer
Osteoporosis, rheumatic dz, steroid use
(compression fracture)
IV drug use (epidural abscess)
Spinal hardware
Spinal Injury
Assessment
ABCs
Immobilize neck and back
GCS, motor/sensory/sphincter tone exam
Imaging
Plain c-spine films (lateral only detects >85%
of cervical spine injuries)
CT/MRI for injuries with neuro deficits and
identifiable spine fractures.
Identifiable spine
fractures require
Orthopedic OR
Orthopaedic spine
consultation
Case Study:
Dislocations
Nomenclature is straightforward:
Most occur @ a joint formed by 2 bones and
the dislocation is named after the affected joint.
Direction of dislocation refers to the position of
the distal bone in relation to the proximal.
Clinically:
Pain, deformity, decreased ROM.
Certain dislocations are associated w/ specific
complications, which must be ruled out in the
routine evaluation of the injurye.g., the axillary
nerve (12%) and the musculocutaneous nerve
(2%) are @ risk in anterior dislocations of the
glenohumeral joint.
Smooth, timely reduction is mandatory.
More dislocations