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  • PATHOPHYSIOLOG DHF

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    susan_salas200183
    71 vues6 pages
    Virus goes into the circulation May reduced platelets or an increase in blood hematocrit Causes viremia which explains appearance of generalized flushing Infects cells and generate cellular response Enters bone marrow and arrest the maturation of megakaryocutes Initiates destruction of the platelet increase potential for hemorrhage Stimulates intense inflammatory response Release of exogenous pyrogens Redness and heat Fever Dizziness, nausea and vomiting, abdominal pain, Cir

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    PATHOPHYSIOLOG dhf

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    Virus goes into the circulation May reduced platelets or an increase in blood hematocrit Causes viremia which explains appearance of generalized flushing Infects cells and generate cellular response Enters bone marrow and arrest the maturation of megakaryocutes Initiates destruction of the platelet increase potential for hemorrhage Stimulates intense inflammatory response Release of exogenous pyrogens Redness and heat Fever Dizziness, nausea and vomiting, abdominal pain, Cir

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    71 vues6 pages

    PATHOPHYSIOLOG DHF

    Transféré par

    susan_salas200183
    Virus goes into the circulation May reduced platelets or an increase in blood hematocrit Causes viremia which explains appearance of generalized flushing Infects cells and generate cellular response Enters bone marrow and arrest the maturation of megakaryocutes Initiates destruction of the platelet increase potential for hemorrhage Stimulates intense inflammatory response Release of exogenous pyrogens Redness and heat Fever Dizziness, nausea and vomiting, abdominal pain, Cir

    Droits d'auteur :

    Attribution Non-Commercial (BY-NC)
    Téléchargez comme DOC, PDF, TXT ou lisez en ligne sur Scribd
    Signaler comme contenu inapproprié
    sur 6

    PATHOPHYSIOLOGY

    RISK FACTORS
    Precipitating factors:
    Fever, Nausea, Vomiting, bleeding
    Virus goesFactors:
    Predisposing into the circulation
    Modifiable: Age, Environment,
    geography
    Non-modifiable:gender

    Bite of an aedes aegypti mosquito carrying a virus and incubates for 6-7 weeks

    Virus goes into the May reduced


    circulation platelets or an
    increase in blood
    hematocrit

    Causes viremia which


    Infects cells &
    explains appearance of
    generate cellular
    generalized flushing
    response

    Enters bone marrow and


    arrest the maturation of
    megakaryocutes

    Initiates destruction of the


    platelet

    Skin petechiae, epistaxis, Increase potential for


    Hematochezia hemorrhage
    Stimulates intense inflammatory response

    Release of exogenous pyrogens The body releases anti-


    inflammatory mediators
    (Histatin, Kinins)

    Increase WBC
    (Neutrophils & A sign of
    Macrophages) infection Vascular response

    Redness and heat


    Release of
    endogenous
    pyrogens

    Dizziness, nausea and


    vomiting, abdominal
    pain, Circulatory
    FEVER Collapse Shock

    Muscle Blood vessels contract to


    contract to prevent body heat loss DEATH
    produce
    additional heat

    Shivering and chills

    Dengue Hemorrhagic Fever

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