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Introduction Definition Incidence Presentation, lie, position of Fetus and satges of labour Risk Factor Cause Diagnosis & prevention Management & complication
It is failure of descent of fetal presenting part in birth cannal due to mechanical reasons . It usually occurs at the pelvic brim, but ocassionally it may ocur in the cavity or the outlet. The passage can be a cause when it has abnormal shape or size and the passenger can be the cause when large, malpositioned or presenting congenital anomalies.
Of 5980 hospital deliveries in our country during the year 2003 only 195 i.e 3.3% were admitted for obstructed labour . With common cause of obstructed labor being CPD followed by malpresentation. The mean duration of labour was 45hrs .
Together with hemorrhage, infection and hypertensive disorders of pregnancy, Obstructed Labour Is the major cause of maternal and prenatal mortality in developing countries like Ethiopia. Genital Fistula, Stress Incontinence, PID, seconadry ammenorhea and uterine rupture are some of its complications.
contraction till expulsion. Terms in Labour y Fetal lie- relation of long axis of fetus to that of the mother. It can be transverse or vertical.
y Featl presentation- the portion of the fetal body that
is formost within birth cannal or in close proximity to it. e.g cephalic, breech
y Fetal habitus- a characterstick posture fetus assumes
portion of fetal presenting part to rt or lt, anterior or posterior side of maternal birth cannal. Stages of labour y 1st stage- is concerned with cervical dilatation. a) latent phase- the time till dilitation of 3-5 cm b) active phase- dilitation of cervix 5-10 cm in presence of uterine contraction y 2nd stage- begins with completion of dilitation and ends with delivery, 3rd is removal of placenta.
is disproportion between the presenting part of the fetus and the birth canal.
y So the d/c b/n dystocia(abnormal labor) and
obstructed labour is the former is difficulty in labour, while the later is failure to descent due to mechanical reasons.
prolonged Labour young age of mother (under 17 years of age) female genital mutilation Custom of early marriage Previous obstructed labour
y Maternal
A. Bony obstruction - constructed pelvis - tumors of pelvic bones B .Soft tissue obstruction - uterus; impacted subserous peduculated fibroid - cervix- cervical dystocia - vagina- septa, stenosis ,tumors - ovaries- impacted ovarian tumors
diminishes the capacity of the pelvis can create obstructed labour. This can be:
Pelvic inlet Mid pelvis Pelvic outlet
Contracted inlet:if
AP(conjugate ) diameter:<12cm Transverse diameter:<13cm Diagonal conjugate:<12.5cm Obstetric conjugate:<10.5cm
or
contracted, the obstruction is much greater than when only one is contracted.
y In women with contracted pelvis ,face and
shoulder presentations are in countered 3 x more frequently, and cord prolapse 4-6x more.
y In contacted pelvis, arrested head in the pelvic
inlet ,the entire force exerted by the uterus acts directly on the portion of the membrane that contact the dilating cervix.
y This finding is more common than inlet contraction y It frequently causes transverse arrest of the fetal
head which potentially can lead to a difficult midforceps operation or to cesarean delivery Hence; the midpelvis is likely contracted when the sum of the transverse and posterior sagittal diameters of the midpelvis normal, 10.5 plus 5 cm, or 15.5 cm falls to 13.5 cm or below.
posterior triangles.
anterior triangle must inevitably force the fetal head posteriorly. the fetus increases=>laceration
of the uterus ,mainly during pregnancy grow with the uterus and the baby, as a result, the uterus becomes cramped and crowded ,affecting not only the the development of the baby ,but also impairs the labour of term pregnancy.
y B.
.This can be longitudinal or transverse. . Can result in blockage of normal menstrual flow and cause primary Amenorrhea,and difficulty in labour and delivery.
vaginal stenosis
Which can cause: . difficulty in labour, .long term problems in sex .pain during physical examination
y D. Ovaries ;
ovarian tumors must grow sufficient size to be caught by the pelvic brim when the ovaries ought to rise in to the abdomen with the developing womb. y Months later , when the fetus fully developed ,the mass be big enough to cause an obstruction.
Fetal causes
A. Malpresentation and malposition -persistent occiput- posterior and deep transverse arrest -persistent mentum- posterior and transverse arrest of the face -brow presentation -shoulder presentation -breech presentation Complete Incomplete Frank
B. Large size fetus(macrosemia) C. Congenital anomalies e.g Hydrocephalus Distended fetal abdomen
. distended bladder
This may be normal in early labour,and approximately 10 20% of fetuses at onset of labor.
to anterior position and deliver as Occiputanterior. position and deliver as Face to Pubis.
delivered
Transverse Lie
y At this transverse lie, the long axis of the fetus is
approximately perpendicular to that of the mother, and the shoulder is usually positioned over the pelvic inlet. The head occupies one illiac fossa, and the breech the other. y Result in shoulder presentation.
the occiput is in contact with the fetal back and the chin(mentum)is presenting.
Lt mento-ant
Rt mento-ant
Rt mento-post
Longitudinal lie. Face presentation. Left and right anterior and ri posterior positions.
y Rare presentation y The portion of the fetal head b\n the orbital ridge
unusually large ,engagement of the fetal head. and subsequent delivery cant takes place as long as brow presentation persists=OL
the fundus of utetrus. y It can be:= - Complete breech : both the hips and knees are flexed. - Incomplete breech: extension of one or more hips. -Frank breech : hips are flexed and knees are extended..
gestational age.
y macrosomia was associated with longer first and
second stages of labor, a greater need for oxytocin therapy, and a greater risk for cesarean delivery.
Hydrocephalus: y is a macrocephally from excessive accumulation of CSF ,this may prohibit vaginal delivery.
y Normal fetal head circumstance =b\n 32-38cm y With hydrocephalus >50cm,up to 80 cm y Fluid volume is b\n 500-1500ml,but as much as
may be permitted.
History
y Relevant points to find out from the woman, her
family or the health care worker are: y Her age y Height, gait, and any disability affecting the pelvis or lower limbs y Medical history, in particular rickets, osteomalacia, or pelvic injury y Any history of circumcision
Reasons for any previous operative deliveries(if she had any) y Previous stillbirth or early neonatal death and cause, if known y Any complications during pregnancy y length of time in labour so far
y
In the partograph if line of cervical dilatation cross the alert or action lines contractions increased in frequency and duration, or stopped etc.
signs of physical and mental exhaustion. y Some or all of the following signs and symptoms may also be observed: y Maternal and/or fetal distress y Dehydration and ketoacidosis (sunken eyes, thirsty, dry mouth, dry skin ) y Fever (raised temperature) y Abdominal pain which may be continuous
y Shock(Shock may be due to a ruptured uterus or sepsis) Manifested by 1.Vital sign derangements -Rapid, weak pulse (100 /minute or more) -Rapid respiratory rate (>30 per minute ) -lowblood pressure (systolic <90 mmHg), 2.Diminished urinary output, 3.Cold clammy skin, pallor, 4.Anxiousness, confusion, or unconsciousness.
Abdominal examination Signs of obstructed labour that may be revealed by an abdominal examination are:
y The widest diameter of the fetal head can be felt above
the pelvic brim because it is unable to descend; y Frequent, long and strong uterine contractions that may stop because of uterine exhaustion then the contractions restart with renewed intensity y The uterus become tightly molded around the fetus that can be felt during abdominal palpation
y Bandl s ring may be seen -Bandl s ring is a late sign of obstructed labour.
in severe cases of obstructed labour because the fetus dies from anoxia.
The signs of obstruction that must be looked for in vaginal examination are
y oedematous cervix which is poorly applied to
yexcessive moulding of fetal head yballooning of the lower uterine segment yformation of palpable or visible retraction ring ymaternal and fetal distress.
evident because cervical dilatation will cross first the alert line and then , if no action is taken, will cross the action line despite a history of strong uterine contractions.
mothers reach their genetically determined height, and pelvic size. 2. Universal antenatal care, so that obstructed labour can be anticipated from a mother's history, and any risk factors can identified. 3. The monitoring of labour by skilled staff, so that she can be referred at the first sign of danger, before she obstructs.
Basic principles
y Aim of management, is to save life and prevent further
damage. y Working quickly and according to priorities so that urgent things are done first. y Must be relieved with out delay y Operative delivery is always needed to relieve the obstruction
y Preventive y Curative
y Preventive
y Carful observation y Proper assessment y Early detection & Management of the cause
of obstruction
"By using partograph
1. Resuscitation
Should be started as soon as diagnosis is made
y Control infection
y If there is any sign of infection or the membranes have been
ruptured
" Ampicillin 2g every 6 hours, and
"
2. Preintervantion preparation
y Catheterize the bladder y Empty the stomach y Determine the blood group & haemotocrit
y Abdominal delevery
y When do we use cs ( abdominal delivery)
y If cephalopelvic disproportion is
confirmed y If precondition for instrumental delivery not fulfilled y Malpresention breech presentation y Definite uterine rapture
y Craniotomy
y If the fetus is dead
y Episiotomy
y If the cause of obstruction is tight peritoneum
y Instrumental delivery
y If the fetus is alive, and the cervix is fully dilated and
y 4. postintervention care
consequences to either or both simultaneously, y Maternal effects include; intrapartum infection, uterine rupture, pathological retraction ring, pelvic floor injury and postpartum lower extremity nerve injury. y Fetal effects include; caput succedaneum and fetal head molding.
fluid, traverse the amnion, and invade decidua and chorionic vessels. y Fetal pneumonia, caused by aspiration of infected amniotic fluid, is another serious consequence. y Digital cervical examinations following membrane rupture introduce vaginal bacteria into the uterus. y These examinations should be limited during labor, especially when dystocia is suspected.
creates a serious danger during prolonged labor. y When the disproportion between the fetal head and pelvis is so pronounced that there is no engagement or descent, the lower uterine segment becomes increasingly stretched, and rupture may follow.
ring of Bandl, an exaggeration of the normal retraction ring. y It is often the result of obstructed labor, with marked stretching and thinning of the lower uterine segment.
pelvic inlet but does not advance for a considerable time, tissues of the birth canal lying between it and the pelvic wall may be subjected to excessive pressure. y vesicovaginal,vesicocervical, or rectovaginal fistulas. y Formerly, when operative delivery was deferred as long as possible, such complications were frequent, but today they are rarely seen.
compression from the fetal head as well as to downward pressure from maternal expulsive efforts. y There is accumulating concern that such effects on the pelvic floor during childbirth lead to urinary and anal incontinence and to pop. y The anal sphincter is torn in 3 to 6 percent of deliveries. y Studies also indicate that abnormal pelvic bony architecture is associated with pelvic floor disorders later in life.
secondary to injury at the level of the lumbosacral root, lumbosacral plexus, sciatic nerve, or common peroneal nerve. y Components of the lumbosacral plexus cross the pelvic brim and can be compressed by the fetal head or by forceps. y Fetal macrosomia and malpresentations (occiput posterior) predispose women to foot drop. y The most common mechanism of injury, however, is external compression of the peroneal nerves usually caused by inappropriate leg positioning in stirrups especially during a prolonged second stage of labor.
succedaneum frequently develops on the most dependent part of the fetal head.
i.e. Caput succedaneum: Swelling of the fetal scalp due to pressure from the cervix. The swelling may be exaggerated in obstructed labour.
oxytocin labor stimulation, and delivery with a vacuum extractor. y Previously considered an overlapping of the parietal bones, ultrasonographic evidence shows molding to be an unbending or straightening of the parietal bones in combination with an inward movement of the occipital and frontal bone apices . y These investigators further described a locking mechanism by which the free edges of the cranial bones are forced into one another, preventing further molding and providing protection for the fetal brain.
may lead to tentorial tears, laceration of fetal blood vessels, and intracranial hemorrhage. y Analyzed nine neonates born with asymptomatic subdural hemorrhages and found that three followed normal vaginal deliveries. y studies indicate that severe molding could lead to fatal subdural hemorrhage as a result of tears involving the dura mater septa, especially the tentorium cerebelli. Such tears were observed in both normal and complicated deliveries.
imperfectly ossified. This important feature may explain the differences observed in the course of labor when two apparently similar cases initially present with identical measurements of the pelvis and the fetal head. y Skull fractures occasionally are encountered, usually following forcible attempts at delivery. The skull also may fracture with spontaneous delivery or even with cesarean delivery . y The fractures are either a shallow groove or a spoonshaped depression just posterior to the coronal suture.
Separation of the muscular wall of the uterus usually occurs during labor occasionally happen during the later weeks of
pregnancy
During pregnancy weak scar after previous operations on the uterus History of cesarean section (vaginal birth after csection) myomectomy excision of a uterine septum previous perforation of uterus(D&C, hysteroscopy, forceps delivery
During labor: uterine hyper-stimulation(oxytocin with pitocin induction or augmentation of labor) obstructed labor(macrosomia, fetopelvic dispropotion) intrauterine manipulation( manual removal of an adherent placenta) forcible dilatation(cervical tear) a weak scar(C-section or other operations)
Incomplete rupture complete rupture depending on whether the peritoneal coat is torn
through or not
1. Rupture of scar be gradual that symptom is very slight in incomplete rupture abdominal pain wrongly attributed to the onset of labor severe pain and shock occurs in complete (suddenly pain) fetal distress bleeding in vagina
2.Spontaneous rupture during obstructed labor prolonged labor violent uterine actions, *pathologic retraction ring disporpotion, malpresentation(transverse lie) fetal distress a sharp, tearing pain in lower abdomen pulse rapid blood pressure fall fetus may be felt in the abdominal cavity
3. Rupture by oxytocin drugs: be follow the administration of oxytocin the danger is less if the drug is given as a dilute intravenous drip given in an increasing fashion
giving morphome, blood transfusion,glucose solution) immediate laparotomy hysterectomy wide-spectrum antibiotics
y Gabes text book of gyne & obs. y Williams text book of obs. y Mid wifery education module-2nd Edition by WHO y Ethiop.J.Health Dev. 2003; 17(3)2.175-180 y Current Diagnosis & Treatment in OBS & GYN