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Definitions:
Impairment:
Any loss or abnormality of psychological, physiological, or anatomical structure or
function.
Disability:
Where an impairment causes restriction of the ability to perform any activity in the
normal manner or a manner considered to be with in the limit of normality.
Handicap:
Impairment or disabilities that limits or prevent the achievement of a role that is
normal for that individual.
Cerebral Palsy:
Non progressive lesion-sustained- wit in the immature growing brain which cause
impairment of Mainly the motor function & posture. The injury develop in fetal or
early life (1st 2 year of life) & it become obvious by the 5 years of age.
Exclude :-lesions occur in older children, spinal cord lesion, progressive disorder of
the brain e.g degenerative brain disease.
==================
Etiology:
1-Prenatal 40%
Genetic –AR & AD-
Congenital cerebral malformation e.g Failure of migration of gray cells, Tubal
defect, Cong. Cyst, Cong. Absence of the pyramidal tract.
Teratogenic e.g Alcohol
Cong. Infection -TORCH-
Maternal PKU
2-Perinatal 55%
I.C.H
Peri-venticular
Leukomalaccia
Birth asphyxia
Cerebral artery infarction
3- Postnatal 5%
Kernicterus
Meningitis
Hypoxic ischemic injury
Cerebral traumatic injury
Metabolic (HyperNa –Hypoglycemia)
Status epelipticus
Asphyxia
N.B:
1-Most common cause of CP is Idiopathic!
Risk Factors:
IUGR
Prematurity 80:1000 Infants of B.wt below 1500g ==because it may cause
I.V.Hg==
1:1000 B.wt above 2500
Sever birth asphyxia
Kernicterus
Feeding difficulties (Nasogastric Feeding)
Classifications:
1- Spastic 85%
(hypertonic)-Increase in DTR- Babeneski- clonus.
Diplagia 20%, Heme-, Mono-, Para-, Qaudr-35%
-Most common Type is Diplagic-?
2-Choreo athetotic 9%
It’s a basal ganglia lesion.
It include: Increase DTR- Movement of big muscles- Not a persistent feature
cause after while it will be over come by spasticity-
To diffrencaite between it & chorea: In chorea it occurs when they start
moving e.g stating to write or starting to comb their hair.
3-Unclassified 3%
4-Hypotonic 2%
To differentiate between it & LMNL you will find Hyper-reflexia (Increase
DTR)
5-Mixed.
1-Pyramidal (Asymetrical Tonic Reflex, babenski, Hypertonia, Increase
DTR)
2-Extrapyramidal (Hypotonia)
======================
N.B:
Tetraplagic: 4 limbs either equal or UL<LL
Paraplagic: LL
Hemiplagic: 1/2 body. UL<LL
Diplagic:4limbs LL<UL
Triplagic: 3 limbs
Monoplagic: One limb
Double Hemiplegics: Both side of the body, where one side is affected more than
the other.
========================
Clinical picture:
In general:
Spasticity.
Increase the Tone : Clasp knife (Indicate lesion motor cortex or pyramidal)
Increase DTR
+/- clonus
Extensor planter responses.
Symptoms:
Arousal problems:
Irritability.
Sleep disturbance.
Lethargy.
Inconsolable crying.
Feeding problem:
Sucking problem
Refused to feed
Choking
Drooling
Spits
Signs:
Accidental or during investigation.
Failure M.S or delayed (Gross: walk, stand, sit, stand) (Manipulation:
Transfer, Holding, reaching)
Persist primitive neonatal reflexes e.g (Early) Moro<6moths, Grasp<4moths.
Abnormal posture or muscle tone e.g Stiff, persistence of fisting beyond age
2 months.
Presentation:
Obvious at age 6 months.
-Signs of hemiparesis. Which include the presence of one hand that is
fisted.
-Decrease the flopping or increase the tone of one wrist when th ULs
are getting shaken.
-Extra beats of clonus.
Early in infancy:
Failure to develop protective response of the lateral propping.
Asymmetrical Parachute response ()سؤال
Uneven crawling.
Early childhood:
Delay walking to 2Y&1/2 TO 3 Y.
Run posturing the U.L in flextion & internal rotation.
Late childhood:
Arm held close to the chest in a position of adduction, flexion, internal
rotation, Leg adducted at the hip. Particularly flexed at the Knee & The Foot
planter flexion.
Scliosis.
Face Normal
In Rt. Side hemiplagia of Post Natal origin ===< Speech is affected. BUT
NOT in congenital origin as the speech center from the other side will take
over.
Convulsions are common.
MR-/+
Cause
Due to Symetrical Bilateral cerebral lesion mainly corticospinal tract to
the LL.
All 4 limbs LL< UL (Symmetrical involvement)
Premature
CP
Intelligent is Normal
Convulsion -/+
IV. Atheosis:Dyskinetic
Abn. Of the basal ganglia.
As in case of Kernectrus (Uncong. Bilirubin) pass BBB & deposit/ destroy
the Basal ganglia.
Present late at age 3 years.
Intelligent is NORMAL
Speech : Dysarthria
Deafness
Convulsion is Rare.
V. Ataxia:
Rare
Cause:
genetic is rare.
Aquired defect of cerebellar function.
C/P:
Arms-interfere with movement.
Legs-unsteady tremulous gait.
If ALL limbs are involved LL<UL
+ve Nystagmus
+ve intention tremors.
It Improve with age.
==================
Diagnosis:
1. Can NOT be Dx at birth as the nervous system is Not mature enough
for clinical signs to be manifested.
2. Spasticity from the background of hypotonia
3. Increase DTR
4. Persistence of primitive reflexes.
5. Delay Developmental M.S- Gross Motor : grasp & walk.
N.B: When Ex the tone in CP, we should keep the head central. Because,
asymmetrical tonic neck reflex may give false impression.
=============================
Management:
Team work. For the child & the family.
1st evaluation 2nd re-assessment.
Multi disciplinary child development center:
Physiotherapist: To achieve max. potential early to facilitate normal
movment & to teach mother.
Occupational Therapist: To help him to sit in position of Max
advantage to eye & head control….AND to minimize the spinal curve.
Specialist health visitor
Psychologist
Speech therapy
Social worker
Dentist
Orthopedic ( Release Tendon achillis to stabilize hip joint)
Pediatrician who can lead with pt. & family
===================
Prognosis
In sever CP----death due to recurrent infection. 50% By age 10years.
Mild Cp----Good prognosis.
=================
Important Hx points in CP:
Delay MS.
Associated problems./ Admit for Exam on regular plan :p
S/R:
1. Related to feed-growth parameters-velocity of growth i.e Increase in Wt.
2. GIT: Anorixai –N/V- Diarrhea
3. Resp:Chest Infection.
4. CVS: In extreme malnutrition==< Dehydration & Tachycardia.
5. GU: UTI
6. M.sk: deformities- skin lesions & ulcer i.e bad hygiene-
NB : IF convulsion you have to look for skin lesions to R/O N.F, T.S, Sturge
web Syndrome i.e neurocutaneous disease.!!
PMHx: previous admissions, Frequency of admission & was it for adjust
medication or NGT. OR chest infection.
Anti-pre-post natal
Feeding: before how?, Now how?, balbar palsy Sx.
Vaccine- Social-FHx
*==================================*
N.B:
This Sheet is done from Dr. S.M Jaber's Sheet & Session (Pager # 2511)-Its ENOUGH to
study CP from HERE