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INTRODUCTION
A. Background
sporadic miscarriage do not have pattern and many that are caused by
and many happen at 12th weeks, that are more than 80 %, there are many
disorder, womb disorder (weak of womb mouth, tumor or miom and womb
for the end of that pregnant and artificial abortion is abortion is happened by a
abortion can definited which bleeding happen from uterus in pregnant before
20th weeks, when the result of conception is still in uterus and there is servic
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fast and correct the complication will be minimilized, But if isn’t handed so
fast will cause the mother died. Because of that abortion incomplit is an
Abortion incomplit abortion has complication that can threat mother safety
because there mast bleeding which can make died because of hypovolemic
shock if this condition can’t be fast and exact core. Some mother who get
incomplit abortion can get physical shock, not only the mother but to family
too.
happening many of women get stress because don’t know what is happened to
often must get rid. The pregnant will continue and the patient can be amuse of
about 5 % case at her pregnancy will be shorter and the baby will be born
preterm.
The biggest abortion are more happened difficult often known for
mother who was stated positive pregnancy and 60-75 % the miscarriage
months after birth. In early abortion, dismiscal of fetus is often started with
fetus died even though in old women abortion. The fetus often still live before
get out.
Ever though the data was got in Bougenville ward Ambarawa Regional
Hospital since September 2007, November 2007 is 32 among that the abortion
account 12 20 32 100 %
Regional Hospital.
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B. Purpose
1. General purpose
2. Special purpose
e. Can evaluate intervention nursing has been given to the women with
abortion incomplit
f. Can know the supporting factor and giving nursing care to the women
C. Advantage
Can compare between the theory and she fact in field about abortion
2. For hospital
can increase healt quality service special giving nursing care to the
3. For institution
CHAPTER II
BASIC CONCEPT
A. Definition
pregnancy 22 weeks) and weight less 500 gram (Saifuddin, 2002 : 10)
1000 gram.(manuaba,2001;584)
live of uterus, as age pregnancy 28 weeks and before the fetus 1.00 gram
2005 : 649)
pregnancy threat or dismissal conception before the fetus can live out
B. Kinds of abortion
out the fetus still can be prevented with giving the hormonal medicine
on medical indication)
C. Etiology
1. Abnormally chromosome
a. Chromosome
c. External factor
1) Infection of endometrium
2. Abnormally of placenta
a. Infection at placenta
miscarriage
3. Mother disease
The growth the fetus condition abnormally on type of myoma uteri, servic
5. Stimulate at the mother which cause the very surpise constraction, the
medicine, afraid, laparatomy or the fetus directly because damage and the
medicine.
before 8 weeks
7. Movement body which coplous on age pregnancy the first or finished (age
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pregnancy under 2 months, because future the fetus at womb yet strong
easy released
D. Pathophysiology
cause bleeding so the fetus less the nutrient and oxygen a part which released
considered as a strong things, so the uterus has the effort to come out with
contraction.
part conception result. Type bleeding variation between little and go on log
and long sum which big with clod so bleeding mentioned can be shock syock,
The first will happen bleeding on desidua basalic and than along
necrosis in around, this mention can cause conception result released a part on
all, so is a storage things on the uterus this condition can cause the uterus
condition on pregnancy less from 8 weeks conception result that usually out
all because villi coriales yet pierce desidua deepened on pregnancy between 8
to 14 weeks the placenta not is released perfect which cause much bleeding on
pregnancy 14 weeks general sign which out on various type there are packed
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amnion empty or measles in depth small thing without type which cleared
(blighted ovum), may be the fetus has been long dead (missed abortion).
When the mudigah dead is not out on time, so it can cover layer
blood clot. The uterus content’s name is mola kruerta. This type become mola
carnosa when blood pigment has been absorb and on residue happen
and corion.
On the focus which has been dead and isn’t out can happen process
as fetus papiroseus.
And then the other on the died fetus which is not quickly out
happened meration, can loose skin cranium become flabby and big stomach
because filled liquid and all the fetus the colors are red.
E. Pathway
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F. Supporting Examination
1. Pregnancy test.
Positive if the fetus still lives even 2-3 weeks after abortion.
261)
F. Clinical Manifestation.
waist
G. Supporting Examination
live or dead
H. Focus Intervention
contraction
game
pulse
Intervention
intravaskuler more
Intervention :
f. Monitor activity uterus, status the fetus there are pain pressure
2001, 495).
Intervention
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infection
desuria, turbid
2001 : 492)
Intervention
blood
related to anorexia
Intervention
often
patient
Intervention
analgesic
edecuate
protrude a cough
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TABLE OF CONTENT
TITTLE OF PAGE.............................................................................................. i
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vi
APPROVAL PAGE............................................................................................. ii
ACKNOWLEDGMENT..................................................................................... iv
CHAPTER I INTRODUCTION
A. Background.................................................................................... 1
B. Purpose.......................................................................................... 4
1. General Purpose....................................................................... 4
2. Specific Purpose...................................................................... 4
C. Advantage...................................................................................... 4
A. Definition....................................................................................... 6
B. Kinds of abortion........................................................................... 7
C. Etiology......................................................................................... 8
D. Pathofisiology................................................................................ 9
E. Pathway......................................................................................... 11
F. Supporting Examination................................................................ 12
G. Management.................................................................................. 12
H. Clinical Manifestation................................................................... 13
I. Focuc intervention......................................................................... 14
REFERENCES.................................................................................................... 18
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