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Baroreceptors
Periodic changes
Accelerations
Decelerations
BASELINE VARIABILITY
N.B. fetal maturation > 32 Wks (as vagus delay in its maturation)
ACCELERATIONS
PURE ACCELERATION -1
Shape:
Timing:
contraction
Range:
25 bmp)
COMPENSATORY -2
ACCELERATION
Uniform
With
Moderate (20
<
Mechanism:
Unknown (fetal stimulation ??)
Pathophysiology: sympathetic over-activity (inc
unopposed)
:Cause
Preterm
Breech
thick ms)
Drugs
(as atropine) (3
Significance: INNOCUOUS
(2
Decelerations
Early
Shape:
Timing:
Late
Uniform slope
No lag time (with contraction)
Shape:
sec)
Timing:
Uniform
Lag time
Duration:
< 1 min
Range:
Moderate (20-25)
:Mechanism
Head compression
(pelvis Fundus if breech iatrogenic PV)
Duration:
< 1 min
Range:
Mild ( < 20 bpm)
:Mechanism
Utero-placental insufficiency
:Pathophysiology
:Pathophysiology
IC pressure
Dura stimulation
(-) HR regulating center in Mid brain
Deceleration
: Cause
2nd stage of labor
( fully dilated do PV (1
: Cause
Inadequate placental feedline (severe PET IUGR P. Abruption )
Uterine hypertonus (IUP > 30 mmHg) .(resting<10 - cont.50-70)
Inadequate maternal feedline -
Head compression
Fetal Hypoxia
examination)
Breech
presentation)
(2
CPD
(3
progress)
PV examination (4
:Significance
:Significance
NEVER NORMAL
Early in labor (exclude breech) Persistent + failure of descent exclude CPD Disappearance + persistence Pressure ?? CNS damage
:OMINOUS SIGNS
:OMINOUS SIGNS
Not associated with
Reduced variability
(Fetal hypoxia - Baseline changes)
Baseline changes (inc then dec)
then long return lag time
Persistent or recurrent
:Non operative
Stop uterine stimulation
Position
Circulatory flow
Oxygen
Mg sulphate
(Semi-sitting Lt side)
(fluid load shock ttt)
(intermittent tight face mask)
(2-3 gm IV)
VARIABLE DECELERATIONS
(MOST COMMON: 80% OF LABOURS)
Shape:
Timing:
Duration:
Range:
Variable
Variable usually < 1 min at the Nadir
Variable (Mild Moderate severe)
Mechanism:
Pathophysiology
&V
Initial pressure
occlusion of umbilical V Blood supply in the fetus
Fetal BP (abrupt)
(+) Baroreceptors
HR (tachycardia)
(pre deceleration Acc)
More pressure
occlusion of umbilical A (with already occluded V)
Resistance against heart
BP (abrupt)
(+) Baroreceptors
HR (bradycardia)
Deceleration
Relief
Umbilical V opens BP
(+) Baroreceptors
HR (bradycardia)
Return to baseline
:Significance
Most are not OMNIOUS
Indicate intact fetal CNS 2nd stage Mild moderate (physiologic: PCO2 of fetal Blood) ;Most are not ominous However, it's the most frequent cause of intrapartum fetal demise
RANGE SIGNIFICANCE
Mild
( < 50% )
Transient
Mild/Moderate Persistent
Moderate +
poor prognostic
signs
Severe
poor prognostic
signs
MILD
Nadir
Duration
Management
80 <
sec 60 30
Reassure
MODERATE
70 >
80 / 70 <
sec 60 30
sec 60 <
Manage as an Ominous
Assure intact
Normal &
terminate
SEVERE
70 >
sec 60 <
Ominous
MANAGEMENT
Mild/Moderate
Normal variability
Moderate
Severe
Non-operative
Close observation
Operative
Failure of non-operative in 20
min
Continue in-Utero resuscitation
Expedite delivery -
Poor prognostic
signs
COMBINED DECELERATIONS
Any pattern not conforming to one of the 3 classic patterns
Most frequent: Variable + Late
(Return to baseline delays > 20 sec)
Management: as the more ominous component
PROLONGED DECELERATIONS
Shape:
Uniform non-uniform Mixture
Duration:
2 min
Timing: Any time
(during several contractions)
Range: severe
(> 25 bpm)
:Cause
Uterine hypertonus (1
Caine drugs (2
Prolonged cord compression (3
Pathophysiology & ttt
Caine drugs
:Cause
Local infiltration
Epidural analgesia
:Pathophysiology
CNS rather than myocardial effect
Uterine hypertonus
:Treatment
Stop drug
Stop syntocinon
:Cause
Drug induced
(syntocinon PGs Caine drugs)
:Pathophysiology
Fetal hypoxia due to defective placental
perfusion
ominous fetal hypoxia
(less tolerated in placental insufficiency )
:Treatment
Maternal supportive measures
Uterine relaxants (rarely indicated)
Wait 15 min
Maternal supportive measures (fluid O2)
Dont deliver
if delivery occurred before the pattern
resolved
neonatal collapse & convulsions may occur
Baseline
(bpm)
160110
109100
180161
Variability
(bpm)
5
5>
for 4090
minutes
Decelerations
Accelerations
None
Present
The absence of
accelerations
with otherwise
normal trace is
of uncertain
significance
Early deceleration
Abnormal
100 >
180 <
Sinusoidal
pattern
10 minutes
5>
for 90
minutes
Normal
Suspiciou
s
Pathologi
cal
.The date and time clocks on the EFM machine should be correctly set
.Traces should be labelled with the mothers name, date and hospital number
Any intrapartum events that may affect the FHR should be noted at the time on the FHR
trace, which should be signed and the date and time noted (for example, vaginal
.examination, FBS or siting of an epidural)
Any member of staff who is asked to provide an opinion on a trace should note their
findings on both the trace and the womans medical records along with the date, time
.and signature
Following birth, the healthcare professional should sign and note the date, time and
.mode of birth on the FHR trace
The FHR trace should be stored securely with the womans medical records at the end
of the monitoring process.
Interpretation of FHR traces/cardiotocographs
The recommended definitions and classifications of the FHR trace/cardiotocograph produced during
EFM are shown in tables 5 and 6.
Table
For women having continuous EFM, a documented systematic assessment based on these
.definitions and classifications should be undertaken every hour
During episodes of abnormal FHR patterns when the woman is lying supine she should be advised to
.adopt the left-lateral position
Prolonged use of maternal facial oxygen therapy may be harmful to the baby and should be avoided.
There is no research evidence evaluating the benefits or risks associated with the short.term use of maternal facial oxygen therapy in cases of suspected fetal compromise
In the presence of abnormal FHR patterns and uterine hypercontractility not secondary to oxytocin
infusion, tocolysis should be considered. A suggested regimen is subcutaneous terbutaline
.0.25 mg7
In cases of suspected or confirmed acute fetal compromise, delivery should be accomplished within a
.time appropriate for the clinical condition
Where assisted birth is contemplated because of an abnormal FHR pattern, in cases of suspected
fetal acidosis FBS should be undertaken in the absence of technical difficulties or any
.contraindications
Where there is clear evidence of acute fetal compromise (for example, prolonged deceleration greater
than 3 minutes), FBS should not be undertaken and urgent preparations to expedite birth
.should be made
.Fetal blood samples should be taken with the woman in the left-lateral position
.The classification of FBS results shown in table 7 is recommended
Interpretation
7.25
7.247.21
7.20
If the FHR trace remains unchanged and the FBS result is stable after the second test, a third/further
.sample may be deferred unless additional abnormalities develop on the trace
.Where a third FBS is considered necessary, consultant obstetric opinion should be sought
:Contraindications to FBS include
maternal infection (for example, HIV, hepatitis viruses and herpes simplex virus)
fetal bleeding disorders (for example, haemophilia)