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Abstract
The level of fertility in natural fertility populations can be though of as a
function of five components: age-pattern of marriage, permanent sterility,
fecundability, postpartum non-susceptible period and intrauterine mortality. A wellknown model for analyzing the proximate determinants of fertility is Bongaarts
model, which, however, refers to controlled fertility and does not account for sterility.
This paper modifies Bongaarts model so as to take into account permanent sterility.
The modified model is applied to four historical European populations so as to
establish the contribution of each of the proximate determinants to the total fertility
rate of these populations.
90
See for example J.Bongaarts and R. Potter, Fertility, Biology and Behaviour, London, 1983; J.
Bongaarts, F. Odile and L. Ron, The proximate determinants of fertility in sub-Saharan Africa,
Population and Development Review 10 (1984) 511-517.
91
2.3 Sterility
Referring to female sterility demographic studies usually distinguish two kinds
of permanent sterility: primary and secondary. Primary sterility for a woman means
that she is not capable to conceive and carry a foetus to terms at any point of her life.
Secondary sterility on the other hand, means that a woman was fecund for a time
period but ceased to be so because of various reasons, the most common being the
process of ageing which renders every woman sterile by the age of 50 or soon
thereafter. Other reasons for secondary sterility may be a trauma of childbirth or the
effect of a disease (Wrigley et al. [21]:358)
Primary sterility can only be measured (demographically) if everyone marries
at her teens. This is hardly the case, however, in European populations where many
marriages occur later in life and thus many women who marry, say, in their 30s and
remain childless cannot be characterised as primary sterile since they may not have
been so if they had married earlier. Consequently, in a population where teenage
marriage is not universal primary sterility cannot be measured based on marriages that
proved childless.
Secondary sterility on the other hand, could be readily approximated from
longitudinal analysis, such as the results of family reconstitution. One could look at
the birth history of a group of women who married at any age and observe the
proportions that become gradually sterile as these women progress from one age
group to the other up to the age 50. Nevertheless, this would only make sense if the
examined population was acting under natural fertility. If any kind of birth control is
applied reliable measures of secondary sterility cannot be derived, since couples can
stop childbearing at their own will.
Nevertheless, there is a sterility measure that can be calculated for any
population, based on the very plausible, even for a modern population, assumption
that no married couple remains voluntarily childless. This is the entry sterility, that is
the proportion of women in a given age at marriage group who were not fecund
(Wrigley et al. [21]:360). Entry sterility reflects both primary and secondary sterility
and its complement is entry fecundity. Its importance, though, lies in the fact that
93
TFR=Cm*Cc*Ca*Ci*TF
(1)
Other factors such as intra-uterine mortality and coital frequency may be responsible for the
difference between the first and second birth interval but these factors are of minor significance.
95
m( a ) g ( a )
g (a)
20
where i=average duration of post-partum
18,5 i
infecundability in months.
TF: Total Fecundity Rate.
To Bongaards model we add one more variable: entry sterility.
Cs= index of sterility (equals 1 if no woman of reproductive age is sterile and 0 if all
women are sterile).
Cs
(1 s(a)) g (a)
g (a)
Where
s(a) = age-specific proportions of entry sterility among couples (by wifes age at
marriage)
g(a) = age-specific marital fertility rates.
96
TFR=Cm*Cc*Ca*Cs*Ci*TF
(2)
(3)
TF
TFR
C m Ci C s
(4)
However when the TFR of a population is known and someone tries to estimate Total Fecundity from
the model (that is when one uses equation (4)) the interaction between the three indexes (Cm, Ci and
Cs) should be less than when one considers TF fixed at 15.3 and uses equation (3) to estimate TFR.
98
99
Figure 1: ASMFRs as percentage of the rate of the 20-24 age group. Data from
English parishes 1800-24, Paros 1904-13, Mykonos 1899-1908, German villages
1875-99 and natural fertility standard.
100
90
80
70
England
60
Paros
50
Mykonos
Germany
40
Standard
30
20
10
0
20-24
25-29
30-34
35-39
40-44
45-49
Source: for the four historical populations as of table 1; for the natural standard: Henry [11].
The only population that deviates visibly from the natural fertility standard
curve is that of rural Germany in the last quarter of the 19th century. Its curve is not
convex but it is not concave either. This is an indication that part of the population of
the 14 German villages who married in the studied period had started applying some
kind of family limitation but this practice was not widespread in the whole population
of these villages. We most probably have to do with a population in the onset of
fertility transition.
100
102
% childless
70
Paros
60
Mykonos
50
rural Germany
40
English parishes
30
20
10
0
15-19
20-24
25-29
30-34
35-39
40-44
45-49
Source: Data on Paros refer to the marriage cohort 1894-1950 and have been taken from Gavalas [8].
Data on Mykonos are based on marriages that took place from 1859 to 1919 or soon thereafter and
have been taken from Hionidou [12]: 226. Data on rural Germany have been taken from Knodel
1988:270 and refer to complete marriages that took place from 1750 to 1899. Data on English parishes
refer to marriages that took place between 1538 and 1837 and have been taken from Wrigley et al. [21]:
384.
5. Conclusions
It is well-known today that the variation in the fertility level of natural fertility
populations is due to five factors: age-pattern of marriage, permanent sterility,
fecundability, postpartum non-susceptible period and intrauterine mortality. These are
known as proximate determinants of natural fertility and have been modelled
mathematically by Bongaarts for analyzing the fertility-inhibiting effects of each of
the proximate determinants and their contribution to the total fertility rate of a
population (Bongaarts [2]).
Sterility as well as miscarriage (i.e. intrauterine mortality) are considered
relatively unimportant determinants of fertility because they do not vary much among
populations (Bongaarts [3]). However, in natural fertility populations, where no or
little birth control is practised, even small fluctuations in one of the proximate
determinants of fertility can produce changes in the overall fertility level of the
population. We examined four historical populations that by all indications can be
103
g (a) m(a) * 5
1000
(5).
f (a)
m( a )
and consequently f (a) g (a) m(a)
This formula derives from g (a)
(6)
(7)
where f(a) is a schedule of age-specific fertility rates and which Bongaarts [3] used to
estimate age-specific marital fertility g(a) in populations where data on marital
fertility were lacking.
The same procedures for the calculations of TFR, TMFR, Cm and Cs have
been followed for the rest of the populations presented in this paper.
Table A.1: Computation of Total Marital Fertility, Total Fertility and index of
marriage (Cm) from g(a) and m(a).
Age-group
15-19
20-24
25-29
30-34
35-39
40-44
45-49
Total
Paros
1904-1913
g(a)
313
417
366
330
278
106
8
1818
TMFR= 9.1
Cyclades
1907
M(a)
0.14
0.61
0.82
0.87
0.84
0.81
0.74
105
g(a)*m(a)
44.3
254.2
300.9
285.4
233.7
85.9
5.9
1210.4
TFR = 6.1
And therefore C m
1210.4
0.67
1817.5
Table A.2: Computation of index of sterility (Cs) from s(a) and g(a).
Age
15-19
20-24
25-29
30-34
35-39
40-44
45-49
Total
Source: see text.
Paros 18941950
1-s(a)
1.00
0.97
0.90
0.80
0.67
0.25
0.00
And therefore C s
Paros 19041913
g(a)
313
417
366
330
278
106
8
1818
(1-s(a))*g(a)
312.9
402.7
328.0
263.6
185.3
26.4
0.0
1518.9
1518.0
0.84
1817.5
References
[1] Bongaarts, J. A., method for the estimation of fecundability, Demography 12 (4),
645-660 (1975).
[2] Bongaarts, J. A., framework for analyzing the proximate determinants of fertility,
Population and Development Review 14 (1), 105-132 (1978).
[3] Bongaarts, J. A., The fertility-inhibiting effects of the intermediate fertility
variables, Studies in Family Planning 13 (6/7), 179-189 (1982).
[4] Bongaarts, J., & Potter R., Fertility, Biology and Behaviour, London: Academic
Press (1983).
[5] Bongaarts J., Odile, F., and & Ron, L., The proximate determinants of fertility in
sub-Saharan Africa, Population and Development Review 10 (3), 511-517 (1984).
[6] Bongaarts, J., The relative contribution of biological and behavioral factors in
determining natural fertility: A demographers perspective, In: Gray, R., Leridon, H.
& Spira, A. (eds.), Biomedical and demographic determinants of reproduction.
Oxford: Clarendon Press (1993).
[7] Frank, O., Infertility in sub-Saharan Africa: Estimates and implications,
Population and Development Review 9 (1) 137-144 (1983)
[8] Gavalas, V.S., Demographic reconstruction of a Greek island community:
Naoussa and Kostos, on Paros, 1894-1998, Ph.D. thesis. London School of
Economics and Political Science, London (2001).
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