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SA node
AV node
Bisa mengeluarkan
impuls 40-50x/menit
Berkas His
Serabut Purkinje
Ventrikel
1 kotak kecil
= 0.04 detik
5 kotak kecil
= 1 kotak sedang
= 0.2 detik
5 kotak sedang
= 1 kotak besar
= 1 detik
MEMBACA EKG
I. Sebutkan iramanya :
Sinus Rhythm
The Heartbeat
Arrhythmia
Tachyarrhythmia
(rate >100 x/min)
Bradyarrhytmia
(rate < 60 X/min)
Management
Haemodynamically unstable patients :
Given oxygen via facemask if the patient is
hypoxic on air.
Keep NBM until definitive therapy has been
started to reduce the risk of aspiration in case of
cardiac arrest or when the patient lies supine for
temporary wire insertion.
Secure peripheral venous access.
Bradyarrhytmias causing severe haemodynamic
compromise (cardiac arrest, asystole, SBP<90
mmHg, severe pulmonary oedema, evidence of
cerebral hypoperfusion) require immediate
treatment and temporary pacing.
Sinus bradycardia
The sinoatrial node (SAN) discharges <60/min. P
waves are normal but slow. It may be normal (e.g. in
sleep, healthy resting hearts).
Causes
Young athletic individual
Drug-(-blockers, morphine, amiodarone,
calcium channel blockers, lithium,
propafenone, clonidine)
Hypothyroidism
Hypothermia
Management
If hypotensive or pre-syncopal :
- Atropine 600 g-3 mg IV bolus repeating as
necessary.
- Isoprenaline 0.5-10 g/min IV infusion.
- Temporary pacing
- Avoid and take steps to correct prepitants
- Stop any drugs that may suppress the sinus or
AV nodes.
Atrioventricular block
This can occur at the AVN (nodal) or His-Purkinje
system (infranodal). Common causes are ischaemic
heart disease, conduction system fibrosis (aging),
calcific aortic stenosis, congenital, cardiomyopathy,
hypothermia, hypothyroidism, trauma, radiotherapy,
infection, connective tissue disease, sarcoidosis,
and anti-arrhytmic drugs. AV block is further
classified :
Management
Interventricular conduction disturbance on their
own do not require temporary pacing. However,
when associated with haemodynamic disturbance
or progression to higher levels of block (even if
intermittent) must consider insertion of a
transvenous pacing wire. The need for longer term
pacing is dependent on the persistence of
symptoms and underlying cause.
Precipitating factors
Underlying cardiac disease
Ischaemic heart disease
Acute or recent MI
Angina
Mitral valve disease
LV aneurysm
Congenital heart disease
LV aneurysm
Congenital heart disease
Abnormalities of resting ECG
Pre excitation (short PR interval)
Long QT (congenital or acquired).
Irama
Tidak teratur
Irama Teratur
Sinus Tachycardia
Atrial Flutter
Supraventricular
Tachycardia
Atrial Fibrillation
SVT :
-due to re-entry mechanism
-narrow QRS complex
-regular
-retrograde atrial depolarization
-P wave ?
Atrial Fibrillation :
-from multiple area of re-entry within atria
-or from multiple ectopic foci
-irregular, narrow QRS complex
-very rapid atrial electrical activity
(400-700 x/min).
-no uniform atrial depolarization
Atrial Flutter :
-The result of a re-entry circuit within
the atria
-Irregular / regular QRS rate
-Narrow QRS complex
-Rapid P waves (300x/min), sawtooth
SR
VES
Sinus rhythm
with
Multifocal VES
VES
VES
SR
SR
SR
SR
SR
SR
QRS lebar
Irama
tidak teratur
Irama Teratur
Ventricular
Tachycardia
Ventricular
Fibrillation
Ventricular Tachycardia
Torsade de Pointes
Ventricular Fibrillation
VT
VF
Management of Tachyarrhytmia
TACHYCARDIA
Stabil
Tidak
stabil
Tidak stabil
Cardioversi :
VT
Non VT
Defibrilasi :
VT / VF
Stabil
QRS sempit
Irama Teratur
Supraventricular
Tachycardia
(SVT)
Adenosin
6 mg 12 mg -12mg
Irama
Tidak teratur
Atrial Fibrilation
(AF)
Diltiazem
15 25 mg
10 mg / 8jam
Stabil
QRS lebar
Irama
tidak teratur
Irama Teratur
Ventricular
Tachycardia
Amiodarone 150 mg
AF dgn MPW ?
VF ?
Consult expert
. ECG
diagnosis of
tachycardia
Types of
supraventricular
tachycardia.