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injection may produce a brief exacerbation of pain before relief is noted and should be
provided with analgesic medications. When full function has been recovered,
reduction of stressful activities should be advised.
2. Calcific tendinitis
General considerations
Calcium deposition in the degenerative rotator cuff may lead to a variant form of
tendinitis in the shoulder region. Asymptomatic bilateral calcium deposits in the
shoulder tissues are common finding in persons over age 40. The pathogenesis is
unclear. The deposits may enlarge or rupture into the subacromial bursa.
Clinical findings
a. Symptoms and signs
The presentation of acute calcific tendinitis or bursitis is dramatic, with
excruciating pain and severe restrection of shoulder motion. The patient may
refuse even the gentlest examination for fear of motion-induced muscle spasm.
b. Imaging studies
X-rays reveal either focal calcium deposits within the rotator cuff or a large cap
of calcium overlying the humeral head, which represents dissemination of calcium
into the subacromial bursa.
Treatment
Treatment of acure calcific tendinitis includes immediate injection of a
corticosteroid and lidocaine solution into the tendon near the calcium deposit or
into the bursa if calcium has entered that structure. Multiple needle punctures into
the calcium deposit may break up the deposit and provide dramatic relief.
Mobilization of the shoulder should procced as described in the preceding section.
3. Biceps tendinitis
Essentials of daignosis
-localized tenderness over the bicipital groove
-pain during supination of the forearm against resistance.
General considerations
A common inflammatory lesion producing shoulder pain involves the biceps tendong
in the bicipital groove. Biceps tendon inflammation usually affects individuals whose
occupation involves repetitive biceps flexion against resistance or whose recreational
activities include forceful throwing of a ball. Pain is prominent over the anterior
aspect of the arm and is aggravated by shoulder motion. Symptomms are worse at
night and improve with rest. Deltoid muscle spasm may be present and may limit both
active and passive motion.
Clinical findings
Biceps tendinitis can be distinguished from rotator cuff tendinitis by localization of
tenderness to the bicipital groove. Forearm supination against resistance with the
elbow flexed at the patients side elicits extreme tenderness in the region of the
bicipital groove when the tendon is palpated near the shoulder. Instability of the
tendon in the groove is occasionally manifested by a snapping sensation as the arm is
abducted and externally rotated. Subluxation of the tendons can be provoked for
diagnostic verification by Yergasons maneuver, in which the patient actively flexes
the elbow against resistance while the physician rotates the humerus externally. An
unstable tendon will pop out of the groove.
Treatment
Treatment of bicipital tendinitis includes cessation of offending activities and shortthem immobilization of the shoulder in a sling ; a trial of aspirin or nonsteroidal antiinflammatory agents ; and, occasionally, local injection of corticosteroids. Repeated
corrticosteroid injections may result in tendon attrition or rupture and should be
avoided. Surgery is occasionally required to stabilize a subluxating tendon.
When discomfort has subsided, progressive mobilization is begun with
exercises similar to those described in the section in rotator cuff tendinitis.
4. Adhesive capsulitis (frozen shoulder)
Essentials of diagnosis
-diffuse shoulder tenderness
-restriction of shoulder joint motion
General considerations
A common cause of shoulder pain in middle-aged and elderly patients is adhesive
capsulitis, or so-called frozen shoulder. This disorder may complicate other
inflamatory shoulder ailments, particularly in individuals immobilized for prolonged
periods. It may also occur without any identifiable inciting trauma and has been
associated with cardiovascular disease, rheumatoid arthritis, and degenerative cervical
spine disease. Though the exact pathogenesis is unknown, the end result is a
chronically inflamed, contracted fibrotic capsule densely adherent to the humeral
head, the acromion, and the underlying biceps and rotator cuff tendons. Normal
bursae are obliterated by scarring.
Clinical findings
A. Symptoms and signs
The onset of symptoms is usually gradual and heralded by complaints of diffuse
tenderness with disproportionately severe restriction of active and passive motion.
Motion is not improved by lidocaine or corticosteroid injection.
B. Imaging studies
Arthrography reveals a contracted joint capsule and no bursal filling. X-rays may
reveal severe osteoporosis of the humeral head.
Trearment
The success of various treatment is difficult to assess, as the natural history of
adhesive capsulitis is spontaneus resolution. Subsidence of pain and return of
nearly full motion can be anticipated, though the process may persist for 6 months
to several years. Efforts to speed return of function have included intensive
physical therapy, oral corticosteroids and anti inflammatory agents, and a
procedure called infiltration brisement, which consists of pressure injection of the
joint with 50 mL of saline and corticosteroid solution in order to break the
adhesions binding the capsule to the surrounding structures.
Clearly, the best treatment of this condition is prevention. Prolonged disuse or
immobilization of a painful shoulder must be avoided. Early mobilization is
stressed, with initiation of gentle range of motion exercises and persistent
encouragement and guidance by the physician and the physical therapist.