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CHAPTER I
INTRODUCTION AND DATA BASE
This chapter deals with the introduction about cerebrovascular disease, the objectives of
this study, the demographic data, and the health history of the patient, Gordons Functional
Health Patterns, and the family genogram.
INTRODUCTION
Cerebrovascular diseases (CVD) are a group of conditions that affect the circulation of
blood to the brain, causing limited or no blood flow to affected areas of the brain. One example
is atherosclerosis, wherein, high cholesterol levels coupled with inflammation in areas of the
arteries in the brain can cause the cholesterol to build up in the vessel in the form of a plaque.
Thus can limit, or completely obstruct, blood flow to the brain, causing a stroke, transient
ischemic attacks, which can result to a variety of other health complications such as Dementia.
Hypertension is also one of the most important causes that damage the endothelial lining of the
blood vessel which exposes the collagen to circulating platelets causing them to aggregate and
initiate a repairing process which is not always complete and perfect. Uncontrolled chronic
hypertension permanently changes the vulnerable fluctuation of blood pressure. A fall in blood
pressure during sleep can lead to a marked reduction of blood flow in the narrowed blood vessel
causing ischemic stroke in the morning. Conversely, a sudden rise in blood pressure due to
excitation during the daytime can cause tearing of the blood vessels resulting in intracranial
haemorrhage. Primarily people who are elderly, diabetic, smoker, or with a history of ischemic
heart disease, have increased risk for cerebrovascular accidents such as an Ischemic, or even a
hemorrhagic stroke. This would affect other organ systems secondary to the neuro-deficits, like
the muscular system, affected by atrophy of disuse from a plegia.
CVD is the most disabling of all neurologic diseases. Approximately 50% of survivors
have a residual neurologic deficit and greater than 25% require chronic care. Cerebrovascular
disease mortality in the Philippines was studied from the existing vital statistics for 2000-2008.
Death rates from cerebrovascular diseases increased enormously both in men and women. This
increase in mortality was seen in all age groups. The age-standardized mortality rate in men rose
from 33.3 in 2000 to 78.0 in 2005, and that of women from 15.4 to 34.5. The male to female
ratios in the age-standardized death rates increased during this 9-year period. Age-standardized
mortality increased clearly in the male population but decreased in the female population of the
Philippines. This excess mortality in males is mostly due to the increased cerebrovascular disease
death rate. This is a clear example of how chronic non-communicable diseases are becoming
major health problems in countries where they previously have not been prevalent. Immediate
preventive measures are needed in order to control cardiovascular diseases in countries, such as
ours, where disease rates are rapidly increasing.
Nursing care has always been a major part of in and out-patient management in most
morbidity cases, such as Cerebrovascular Disease like stroke. Having seen the increasing number
of CVD cases in our country, this has led to our interest in the study of CVD as a case. It is
through this case report that we can apply the appropriate nursing care through skills to alleviate
patients symptoms, and provide quality care.
OBJECTIVES
At the end of 30-minutes case presentation, student nurses will be able to:
Knowledge
-
Skills
-
Apply the Nursing Process in formulating appropriate nursing care plan for patient with
Cerebrovascular Disease.
Construct a realistic and timely manner plan of care to patient with cerebrovascular
disease.
Attitude
-
DEMOGRAPHIC DATA
Patient Mr. Strokesman, a 67 year old, male, married; Filipino; Seventh-Day Adventist;
born on June 15, 1949; a retired teacher, residing in San Juan Pontevedra Negros Occidental.
Admitted for the first time in Adventist Medical Center Bacolod, date September 4, 2016,
while due to slurred speech and left-sided weakness. His provisional diagnosis was: to consider
CVD Infarct, HCVD, DM II, and is finally diagnosed with Acute Infarct, Right Corona Radiata
by his attending physician, Dr. Reynold A. Wong. On September 13, 2016 at around 7:10 pm,
Mr. Strokesman was discharged.
NURSING HISTORY
History of Present Illness
Informant: Mr. Strokesman and his son
According to Mr. Strokesmans son, on September 4, 2016 early morning, Mr.
Strokesmans sat on his chair beside his bed after he woke up while he was inside his room, he
wanted to go outside so he stood up to open the door, but suddenly fell to the floor. His family
members heard a bang and immediately rush to it. They found the patient lying on the floor but is
conscious. According to his son, he didnt feel anything (e.g. dizziness, nauseated, etc.) before
his fall. But they noticed that he had slurring of speech and signs of weaknesses so they
immediately rushed him to CLMMRH, but because they werent given any attention, they
decided to transfer the patient to Adventist Medical Center-Bacolod. According to the patients
son, the patient was just feeling fine during the transportation, it them, the son and the other
members of the family who were panicking. They immediately brought him to Emergency
Department of AMC-B with a chief complaint of slurring of speech and left sided weakness.
FAMILY GENOGRAM
Fathers Side
U
Mothers Side
70
64
89
59
59
66
HTN
VA
HTN
67
HTN & DM
Acute Infarct, Right
Corona Radiata
64
63
60
HTN
LEGEND:
Male
HPN
Hypertension
Female
DM
Deceased
Unknown
Age
VA
Vehicular Accident
68
52
75
76
80]
DM
HPN
27
VA
10
CHAPTER II
THE DISEASE ENTITY
This chapter contains the chief complaint and the medical diagnosis of the patient, the
anatomy and physiology of the brain, and the pathophysiology of the disease.
Chief Complaint: Slurred speech and left-sided body weakness
Medical Diagnosis: Acute Infarct, Right Corona Radiata
11
to the Endocrine System. The midbrain and pons are also part of the unconscious brain. The
thalamus serves as a central relay point for incoming nervous messages.
The cerebellum is the second largest part of the brain, after the cerebrum. It functions for
muscle coordination and maintains normal muscle tone and posture. The cerebellum coordinates
balance.
The conscious brain includes cerebral hemispheres, which are separated by the corpus
callosum. In reptiles, birds, and mammals, the cerebrum coordinates sensory data and motor
functions. The cerebrum governs intelligence and reasoning, learning and memory. While the
cause of memory is not yet definitely known, studies on slugs indicate learning is accompanied
by a synapse decrease. Within the cell, learning involves change in gene regulation and increased
ability to secrete transmitters.
The Brain
During embryonic development, the brain first forms a tube, the anterior end which
enlarges into three hollow swellings that form the brain, and the posterior of which develops into
spinal cord. Some parts of the brain have changed little during vertebrate evolutionary history.
Parts of the Brain as seen from the Middle of the Brain
Vertebrate evolutionary trends include:
1. Increase in brain size relative to body size.
2. Subdivision and increasing specialization of the forebrain, midbrain and hindbrain.
3. Growth is relative in size of the fore brain, especially the cerebrum, which is
associated with increasingly complex behavior in mammals.
12
13
The cortex in each hemisphere of the cerebrum is between 1and 4mm thick. Folds divide
the cortex into four lobes: occipital, temporal, parietal, and frontal. No region of the brain
functions alone, although major functions of various parts of the lobes have been determined.
The occipital lobe (back of the head) receives and processes visual information. The
temporal lobe receives auditory signals, processing language and the meaning of words. The
parietal lobe is associated with the sensory cortex and processes information about touch, taste,
pressure, pain, and heat and cold. The frontal lobe conducts three functions:
1. Motor activity and integration of muscle activity
2. Speech
3. Thought processes
Most people who have been studied have their language and speech areas on the left
hemisphere of their brain. Language comprehension is found in Wernickes area. Speaking
ability is in Brocas area. Damage to Brocas area causes speech impairment but not impairment
of language comprehension. Lesions in Wernickes area impair ability to comprehend written and
spoken words but not speech. The remaining parts of the cortex are associated with higher
thought processes, planning, memory, personality and other human activities.
14
Predisposing Factors:
-
Precipitating Factors:
Tx:
- Atorvastatin
Narrowing of blood
vessels (Coronary Artery)
Sedentary Lifestyle
Diet (High Fat,
Cholesterol)
Insufficient insulin
secretion/production of the
pancreas
Atherosclerosis
Increased peripheral
vascular resistance
Increased viscosity of
blood
HCVD
Severe elevation in BP
without progressive target
organ
Sx:
BP
Monitoring:
160/60
170/90
mmHg
Hypertension
Obstruction/interruption of
O2 supply due to
decreased blood supply
Dx:
RBS
Monitoring: 54
214 mg/dL
Tx:
- Glimiperide
- Humulin R
Sx:
- Increased thirst
- Fatigue
- Slow-healing sores
15
If treated:
- Nursing management
- Diet management
- Pharmacologic
medications
- O2 therapy
Recovery
Tx:
- Losartan+Amlodipine
- Amlodipine
- Nitroglycerine
If untreated:
Further complication
can occur:
- Ischemia
- Angina Pectoris
- MI
Decreased O2 supply to
Basal Ganglia,
INFARCTION
Death
Sx:
- Slurring of Speech
(Dysphasia)
- Dysphagia
- Left sided weakness
16
CHAPTER III
THE MANAGEMENT
This chapter contains the nursing physical assessment, diagnostic studies results and
significance, drug study, nursing care plan, and the discharge summary.
17
are pinkish in color in both eyes. Ears are symmetrical in shape and no lesions or discharges
observed. Nose is symmetrical in shape, presence of NGT tube at the left nostril, no presence of
discharges observed. No tenderness noted upon palpation on the frontal and maxillary sinuses.
Lips appear to be slightly dry.
Neck
No masses found upon inspection and palpation of cervical lymph nodes. No distension
of jugular vein noted and no unusual pulsations upon inspection. No deviations of trachea noted
upon palpation. No irregularities of the thyroid gland noted upon inspection and palpation.
Respiratory System
Respiratory rate of patient upon assessment is 18 cpm. Patient is with nasal cannula, O2
running at 2L/min. Stridor is heard upon inspection and observation of the patients breathing.
Cardiovascular System
Heart rate of patient upon assessment is 97 bpm. No unusual pulsation observed on the
patients jugular vein. No abnormal heart sounds heard upon auscultations.
Gastrointestinal and Urinary System
Patient is with NGT and urinary catheter. No tenderness observed upon palpation of the
abdominal area and no unusualities heard, such as hyperactive bowel sounds, upon auscultation
of the patients abdomen.
18
Neurologic System
Upon assessment, patient was oriented to time, person, and place. Appropriate thought
process is observed by being able to answer questions appropriately, although some words are
hard to make out due to the patients slurring of speech; patient readily answers when asked with
some questions. Patient is able to recall what happened before and after he fell down, and
memories way before his accident. Patient is friendly to the staffs and socializes with them. The
patient has minimal left-sided weakness and is able to move his left arm and left leg with a
limited range of motion. The patients right arm and legs is with full range of motion. Pain reacts
upon pain infliction by jolting or moving away from the source of pain.
Endocrine System
Patient was diagnosed with Diabetes Mellitus type 2 on the year 2011, upon consultation
to a private medical doctor.
19
K (Potassium)
(L)
may indicate
dehydration, stress,
diabetic acidosis,
excessive ingestion
K (Potassium)
Result
of glucose.
September 13, 2016 5:17 AM
Normal Value
Significance
Increased glucose
level may indicate
RBS Glucometer
116 mg/dL
60 110 mg/dL
presence of
Diabetes Mellitus,
or excessive stress.
September 12, 2016 5:15 PM
Decreased glucose
level may indicate
RBS Glucometer
54 mg/dL
60 110 mg/dL
insulin overdose or
rebound
hypoglycemia.
FBS (Glucose)
Increased in glucose
214 mg/dL
Diabetes Mellitus,
149 mg/dL
Result
129 mg/dL (H)
presence of
or excessive stress.
September 5, 2016 6:30 AM
Normal Value
Significance
74.00 106.00
Increased glucose
mg/dL (H)
20
presence of
Diabetes Mellitus
Hematology Section
Result
HCT
0.34
LYMP%
0.10
0.25 0.35
EO%
0.12
0.01 0.03
POLYS
0.71
Result
HCT
0.34
0.05 0.65
infections or inflammations.
September 7, 2016 7:00 AM
Normal Value
Significance
Decreased hematocrit levels
0.37 0.47
RBC
3.90
46
WBC
10.14
5 10
LYMP%
0.07
0.25 0.35
EO%
0.05
0.01 0.03
21
MCH
0.81
31.30
Chest X Ray
0.05 0.65
26 31
September 5, 2016
Impression:
Clear lungs
True cardiac size not ascertained
Atherosclerotic aorta
22
Drug Study
PARENTERAL MEDS
Generic/Brand Name:Citicoline Sodium /
Zynapse
Dosage/Route: 1g q8
Indication: Citicoline is indicated in CVD in acute recovery phase in severe s/sx of
cerebrovascular insufficiency and in-cranial traumatism and their sequallae. Citicoline in CVA,
stimulates brain function.
Mechanism of Action: Citicoline increases blood flow and O2 consumption in the brain. It is
also involved in the biosynthesis action.
Side Effects/Adverse Reactions: Fleeting and discrete hypotension effect, increased
parasympathetic effects, low blood pressure, itching or hives, swelling in face or hands, chest
tightness, tingling in mouth and throat.
Nursing Consideration: - Monitor for adverse effect.
- Monitor patients neurologic status.
- Note presence of slurring in speech.
Patient Teaching: - Instruct patient to take the medication as prescribed.
-
Teach the patient that citicoline may be taken with or without food.
Instruct patient to report immediate feelings of chest tightness, tingling
in mouth and throat, headache, diarrhea and blurring of vision.
23
superinfections.
- Assess for allergic reactions.
- Monitor blood studies.
- Monitor electrolytes.
- Assess bowel pattern daily.
- Monitor for bleeding.
Patient Teaching: - Instruct patient to report rash, fever, or chills. A rash is the most common
allergic reaction.
ORAL MEDS
Generic/Brand Name: Atorvastatin Calcium /
Lipitor
CoA reductase inhibitor
Dosage/Route: 40mg/tab ODHS P.O.
Indication: Prevention of CV disease in adults without clinically evident coronary disease but
with multiple risk factors for CAD such as age >55 yr, smoking, hypertension, low HDL-C,
family history of early CAD; to reduce the risk of MI and risk for revascularization procedures
and angina.
Mechanism of Action: Inhibits HMG-CoA reductase, the enzyme that catalyzes the first step
in the cholesterol synthesis pathway, resulting in a decrease in serum cholesterol, serum LDLs
(associated with increased risk of CAD), and increases serum HDLs (associated with
decreased risk of CAD); increases hepatic LDL recapture sites, enhances reuptake and
catabolism of LDL; low triglyceride levels.
Side Effects/Adverse Reactions: CNS: Headache, asthenia
GI: flatulence, abdominal pain, cramps, constipation,
nausea, dyspepsia, heartburn, liver failure
RESPIRATORY: sinusitis, pharyngitis
Other: rhabdomyolysis with acute renal failure, arthralgia,
myalgia
Nursing Consideration: Assessment
-
24
Intervention
-
normal levels
Withhold atorvastatin in any acute, serious condition (severe
infection, hypotension, major surgery, trauma, severe metabolic or
endocrine disorder, seizures) that may suggest myopathy or serve
25
Generic/Brand Name:Losartan+Amlodipine /
Tozam
Angiotensin II Antagonists
Dosage/Route: 500mg/5mg 1tab BID (Hold for BP <130mmHg) P.O.
Indication: Indicated to treat mild to moderate hypertension.
Mechanism of Action: Amlodipine inhibits the movement of Ca ions across the cell
membrane into vascular smooth muscles and myocytes. Losartan is an angiotensin II receptor
(type AT1) antagonists antihypertensive which acts by blocking the actions of angiotensin II of
renin-angiotensin-aldosterone system.
Side Effects/Adverse Reactions: Headache, dizziness, back pain, myalgia, respiratory tract
disorders, asthenia/fatigue; first-dose hypotension, rash, cough, angioedema; neutropaenial GI
disturbances; transient elevation of liver enzymes; taste disturbances and hyperkalemia,
hypotension, bradycardia, conductive system delay, CCF.
Nursing Consideration: - Monitor blood pressure frequently during initiation of therapy.
Because drug-induced vasodilation has a gradual onset, acute
hypotension is rare.
-
26
hypotension
- Regularly assess the patients renal function
Patient Teaching: - Advise patient to avoid salt substitutes; these products may contain
potassium, which can cause high potassium level
-
Generic/Brand Name:Acetylcysteine /
Exflem
derivative
Dosage/Route: 600mg 1tab BID 500cc H2O P.O.
Indication: Adjunct therapy for abnormal viscid thickened mucous secretions in patients with
pneumonia, bronchitis, bronchiectasis, primary amyloidosis of the lung, tuberculosis, cystic
fibrosis, emphysema, atelectasis, pulmonary complications of thoracic surgery, or CV surgery.
Mechanism of Action: Reduces the viscosity of pulmonary secretions by splitting disulphide
linakages between mucoprotein molecular complexes. Also, restores liver stores of glutathione
to treat acetaminophen toxicity.
Side Effects/Adverse Reactions: CNS: abnormal thinking, fever, drowsiness, gait
disturbances
CV: chest tightness, flushing, hypertension, hypotension,
tachycardia
EENT: rhinorrhea, ear pain, eye pain, pharyngitis, throat
tightness
GI: nausea, stomatitis, vomiting
Respiratory: bronchospasm, cough, dyspnea, rhonchi
Skin: clamminess, diaphoresis, pruritus, rash, urticarial
Other: anaphylactoid reaction, angioedema, chills
Nursing Consideration: - Monitor cough type and frequency
-
27
28
Watch for signs of rhabdomyolysis, such as dark, red, or colacolored urine; muscle tenderness; generalized weakness; or muscle
stiffness or aching
Patient Teaching: - Caution patient to avoid hazardous activities until drugs effects are
known.
-
Platexan
aggregation inhibitor
Dosage/Route: 75mg/tab OD P.O.
Indication: Indicated to reduce thrombotic events in patients with atherosclerosis documented
by recent stroke, MI, or peripheral arterial disease
Mechanism of Action: Inhibits the binding of adenosine diphosphate (ADP) to its platelet
receptor, impending ADP-mediated activation and subsequent platelet aggregation, and
irreversibly modifies the platelet ADP receptor.
Side Effects/Adverse Reactions: CNS: confusion, fatal intracranial bleeding, hallucinations
CV: hypotension
EENT: epistaxis, rhinitis, taste disorder
GI: haemorrhage, abdominal pain, constipation, diarrhea,
dyspepsia, gastritis, ulcers
GU: UTI, hematuria
Hematologic: purpura
Musculoskeletal: arthralgia, myalgia, arthritis
Respiratory: bronchospasm, respiratory tract bleeding
Skin: rash, pruritus, eczema, Stevens-Johnson syndrome
Nursing Consideration: - Drug may cause fatal thrombotic thrombocytopenic purpura that
requires urgent treatment, including plasmapheresis
Patient Teaching: - Advise patient it may take longer than usual to stop bleeding. Advise him
to refrain from activities in which trauma and bleeding may occur, and
encourage him to wear a seat belt when in a car.
29
occurs.
Tell patient to inform all health care providers, including dentists, before
undergoing procedures or starting new drug therapy, that he is taking
drug.
Drug has strong taste. Mixing oral form with orange juice mask
the taste
- Assess patients airway
- Provide support ventilation, if needed
- Assess and support cardiac function
Patient Teaching: - Advise patient to report any unusualities
30
(Provasc)
Channel Blocker
Dosage/Route: 10 mg/tab tab OD P.O.
Indication: Indicated to treat hypertension and prevent chronic stable angina or vasospastic
angina
Mechanism of Action: Inhibits the movement of calcium ions across the membrane of cardiac
and arterial muscle cells, slowing the conduction of the cardiac impulse, and decrease cardiac
work, decrease cardiac oxygen consumption and increases on to cardiac cells.
Side Effects/Adverse Reactions: CNS: fatigue, dizziness, light-headedness, asthenia
CV: edema, flushing, palpitations
GI: dyspepsia, nausea, abdominal pain
GU: sexual difficulties
Musculoskeletal: muscle cramps
Respiratory: dyspnea
Skin: rash, pruritus
Nursing Consideration:- Monitor blood pressure frequently during initiation of therapy.
Because drug-induced vasodilation has a gradual onset, acute
hypotension is rare.
-
31
TREATMENT MEDS
Generic/Brand Name: Nitroglycerine (NTG
Classification:Vasodilator - Nitrate
Patch)
Dosage/Route: 5mg OD ACW
Indication: Nitroglycerine is indicated in preventing chronic angina attacks and hypertension
Mechanism of Action: Relaxes (widens) blood vessels to allow blood to flow more easily.
This reduces the heart's workload and the amount of oxygen needed by the heart.
Side Effects/Adverse Reactions: CNS: dizziness, syncope, weakness
CV: orthostatic hypotension, tachycardia, palpitations
EENT: S.L. burning
GI: nausea, vomiting
Skin: cutaneous vasodilation, contact dermatitis, rash
Nursing Consideration: - Administer nitroglycerin patch with extreme caution to patients
with hypotension or hypovolemia since the drug may precipitate a
severe hypotensive state.
-
STAT MEDS
32
Generic/Brand Name:Insulin(HumulinR)
Classification:Antidiabetic Pancreatic
hormone
Dosage/Route: 5 units/mLSubq
Indication: Treatment of type 1 (insulin-dependent)diabetesand type 2 (non- insulindependent) diabetes
Mechanism of Action: Insulin is a hormone secreted by the beta cells of the pancreas that, by
receptor-mediated effects, promotes the storage of the body's fuels, facilitating the transport
of metabolites and ions (potassium)through cell membranes and stimulating the synthesis
of glycogen from glucose, of fats from lipids, and proteins from amino acids.
Side Effects/Adverse Reactions: EENT: blurred vision
GI: dry mouth
Metabolic: hypoglycaemia, hyperglycemia,
hypomagnesemia, hypokalemia
Skin: rash, urticaria, pruritus, swelling, redness, stinging,
warmth at injection site
Respiratory: increased cough, respiratory tract infection,
dyspnea, reduced pulmonary function
Other: lipoatrophy, lipohypertrophy, anaphylaxis,
hyspersensitivity reactions
Nursing Consideration:- Monitor patient for hyperglycemia (rebound, or Somogyi, effect)
Patient Teaching: - Make sure that the patient knows that drug relieves symptoms but doesnt
cure the disease
-
skipping meals
Advise patient not to smoke within 30 minutes after insulin injection
33
characteristics appearance.
5. Gave due OTF q3 100cc.
34
Ineffective cerebral tissue perfusion related to interruption of blood flow secondary to multiple small old infarcts as
manifested by changes in motor and language deficit
Cues (Objectives): The patient has left sided weakness and slurred speech noted, BP of 160/80, and patient is on bed rest. GCS of
15, CT scan impression of probable acute infarct on the right side of the pons.
Expected Outcome: After 3 days of nursing intervention, the patient will be able to demonstrate stable vital signs of BP- 140/80,
and maintain GCS of 15.
Nursing Interventions
1. Assess higher functions, including speech, alertness, and
Rationale
Changes in cognition and speech content are an indicator of location
reactivity.
position.
6. Maintain bedrest, provide quiet environment, and restrict
35
Activities that require holding the breath and bearing down( Valsalva
maneuver) can result in bradycardia (temporarily reduced cardiac
and laxatives)
10.Monitor laboratories studies as indicated
anticoagulant.
Evaluation: Goal Partially met. The patient still manifest unstable vital signs of 140/60, 150/70, 150/80, 160/80. Patients GCS
assessment is still 15 with noted slurring of speech and left sided weakness.
References: Valenti, L., Rozinski, M., &Tamblyn, R. (1998). Lippincotts Review Series: Critical Care Nursing. Lippincott
Williams and Wilkins: Philadephia
Doenges, M., Moorehouse, M., &Murr, A. (2008). Nurses Pocket Guide: Diagnoses, Prioritized Interventions, and
Rationales. iGroup Press Co., Ltd.: Thailand
Potawan, H.B. (2012, November). Ineffective Cerebral Tissue Perfusion. Retrieved October 4, 2016, from Scribd:
https://www.scribd.com/doc/114128541/Ineffective-Cerebral-Tissue-Perfusion
Activity intolerance related to left body weakness as evidenced by decreased muscle strength.
Cues (Objectives): The patient is on complete bed rest, with left side weakness, without bathroom privileges, Braden Scale is 16, of
BP-160/80 GCS of 15.
Expected Outcome: At the end of 3 days duty, patient will be able to maintain good skin integrity, and good muscle strength.
Nursing Interventions
Rationale
1.Monitored the vital signs, assess braden scale, and GCS of To have a baseline data, identification of nursing intervention, and to
the patient every hour.
2.Assisted patient reposition at least every hour (supine, or
36
overexertion.
Evaluation: Goal partially met. At the end of 3 days of duty, the patient is still in bed rest, with GCS 15, left sided body weakness,
BP of 160/60, HR of 97, Braden Scale of 16 and with absence of pressure sores.
References:Valenti, L., Rozinski, M., &Tamblyn, R. (1998). Lippincotts Review Series: Critical Care Nursing. Lippincott Williams
and Wilkins: Philadephia
Doenges, M., Moorehouse, M., &Murr, A. (2008). Nurses Pocket Guide: Diagnoses, Prioritized Interventions, and
Rationales. iGroup Press Co., Ltd.: Thailand
Nurse Theory.(n.d.). Activity intolerance. Retrieved October 4, 2016 from Nurse Theory:
http://www.nursetheory.com/activity-intolerance/
Nurseslabs. (2012, December). NCP CVD impaired physical mobility. Retrieved October 3, 2016, from Nurseslabs:
http://nursinginterventionsrationales.blogspot.com/2013/07/impaired-physical-mobility.html
Risk for impaired skin integrity related to decreased mobility as evidenced by left sided weakness
Cues (Objectives): The patient is on bed rest, with no bathroom privileges, and needs assistance in turning, GCS 14 and Braden
Scale is 16 (Sensory-4, Moisture-3, Activity 1, Mobility 3, and Nutrition-3).
Expected Outcome: After 3 days of duty in the ICU, the patient will be able to maintain tissue integrity as evidence by no skin
breakdown.
Nursing Interventions (OME)
Rationale
37
intervention. Health skin differs from person to person, but must have
a good turgor, feel warm and dry to touch, be free of injury, and have
circulation.
Reduces the pressure on tissues, avoiding skin breakdown.
pillows.
8. Maintain linens dry and free of wrinkle, crumbs.
9. Rise lower extremities occasionally.
10. Educate patient and caregiver the reason(s) of pressure
38
Doenges, M., Moorehouse, M., &Murr, A. (2008). Nurses Pocket Guide: Diagnoses, Prioritized Interventions, and
Rationales. iGroup Press Co., Ltd.: Thailand
Nurseslabs. (2012, December 2016). NCP CVA Unilateral Neglect. Retrieved October 3, 2016, from Nurseslabs:
https://www.scribd.com/doc/117005199/NCP-CVA-Unilateral-Neglect
Deficient self-care (bathing, hygiene, toileting, dressing, grooming, and feeding) related to body weakness, as evidenced by
complete bed rest.
Cues (Objectives): The patient is with left sided weakness, no bathroom privileges, with nasogastric tube on left nostril, and needs
assistance in performing ADL, Braden Scale of 16 with a GCS of 14.
Expected Outcome: After 8 hours of nursing intervention, the patient will be able to achieve and maintain good hygiene as
evidence by satisfying appearance and good grooming.
Nursing Interventions
1. Assessed ability to carry out ADLs on regular basis.
Rationale
Provides data regarding mobility and ability to perform activities
the patients.
2. Assessed specific cause of each deficit (e.g. weakness).
enable self-care.
Planning conserves energy and minimizes the workload of the heart
bathing.
4. Performed AM care.
5. Provide privacy during bathing, dressing and during
to pump blood.
To provide comfort and enhance well-being.
The need for privacy id fundamental for most patients.
39
buttocks and perineal area with water and pat dry with
tissue.
11. Emphasize the importance of maintaining good hygiene
40
Discharge Summary
Nursing Goals
Medication
Orders
Rationale
prescribed To have a
-Verbalize compliance
as:
to prescribe
medication regimen
6pm)
Citicoline (Zynapse) 1g/tab OD
AM AC breakfast (7am)
Solifenacin Succinate (Vesicare)
5g 1tab OD AM x 2 days more
Sept 14 (7am)
Atorvastatin (Lipitor) 40mg
1tab OD HS (8pm)
Clopidogrel (Platexan) 75g 1tab
OD PC lunch (1pm)
Losartan + Amlodipine 50/5mg
1tab OD AM (8am)
NAC (Exflem) 600mg/5ml
water ODHS x 3 days then pm
OD PC Lunch (1pm)
KCL tab (K Lyte) 600mg/tab
BID x 4 doses until sept 14
(8am-6pm)
Glimiperide (Norizec) 2mg/tab
OD AC before Breakfast, hold
for RBS<100 (7am)
of the patient
41
Verbalize
To improve
motor function
importance of tongue
& maintain
exercise
muscle tone.
&
light
activities.
Therapy
To decrease
restore
viscosity of
muscle
strength
&
have
adequate
motor
body
&
secretions on
functional activities.
the airways
Encourage
patient
to
Verbalize
To achieve &
understanding on the
maintain good
importance
of
hygiene to
maintaining
good
prevent
hygiene.
development
of infections
and avoid foul
Follow up check-up on Room 306 odors.
Medical Plaza, September 13,
OPD
Identify
updates on
and to check if
there are
42
progress
Report significance of
To have good
nutrition and
healthy
optimal body
body
condition
-----------------------------------------
Spiritual
conditioning
To have Gods
guidance and it
of
healthy spiritual
life by worshipping
to be healed
God
faster
and
everyday
praying
43
CHAPTER IV
GENERAL EVALUATION OF THE STUDY
This chapter comprises the implication of the study (Nursing Practice, Nursing
Education, Nursing Service, and Nursing Researcher), the personal reaction of the group, and the
bibliography.
A. Implication of the Study
We the researchers would like to recommend the following to:
Nursing Practice
To the nursing practice, the researchers would like to recommend for the continuity of
education to its practitioners in concepts and skills required for practice, focusing especially on
the population, PHN practice, advocacy for more public health resources, and collaboration with
academic partners for education and research.
Nursing Education
To the nursing education, the researchers would like to recommend the need for highlyeducated nurses. The ways in which nurses were educated during the 20 th century are no longer
adequate for dealing with the realities of health care in the 21 st century. As patient needs and care
environments have become more complex, nurses need to attain requisite competencies to
deliver high-quality care. These competencies include leadership, health policy, system
improvement, research and evidence-based practice, and teamwork and collaboration, as well as
competency in specific content areas such as community and public health and geriatrics.
44
Nursing Service
To the nursing service, the researchers would like to recommend an assurance to a
sufficient continuing supply of new applicants, nurse educators and national nursing
organizations should adopt recruitment strategies that attract not only recent high school
graduates but also nontraditional prospective students, such as those seeking late entry into a
profession or seeking to change careers, and minorities. Closer collaboration between nurse
educators and nurses who provide patient services is essential to give students an appropriate
balance of academic and clinical practice perspectives and skills during their educational
preparation.
Nursing Researchers
To the nursing researchers, the researchers would like to encourage future nursing
researchers to take interest in cases like this, to develop a more quality type of care for such
patients based on the appropriate knowledge gained through research. Also to delve a little
deeper on the case in order to make appropriate care needed for the patients and be able to share
gained knowledge and insights to co-researchers as well regarding the case being studied to help
expand the knowledge of others in order to achieve a more quality kind of care.
B. Personal Reaction
We are indeed fortunate that we were able to care for a client with Cerebrovascular Disease.
Although it is one of the most common diseases, it has been a great opportunity that we were
able to gain experience, study and dig a little deeper about the background and the formulation of
care essential for the client with the said disease. Before, we had a little understanding about
45
Cerebrovascular Disease and because of this study, we gained better and clearer discernment
how the disease progressed.
This study also adds our perception in the importance of each prescribed medications and
special consideration that is given for clients with Cerebrovascular Disease. Throughout this
study, we were able to develope our personal skills and the formulation of our discharge
summary to our chosen patient.
For the purpose of this study, we hope that it will also guide and expand the horizon of the
reader in their breakthrough of their clients care.
46
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