AD Overviews AD fact sheet
BioInitiative Report 2012
Beta-amyloid plaque develops
Shrinkage of brain tissues
Tangles and clumps in nerves, disintegration
Alzheimer's disease is not a memory disorder;
more accurately, it is a degeneration of neural
and supporting tissue in the brain. It is
characterized by the presence of fiber tangles
within nerve cells, plaques or clusters of
degenerating nerve endings, accumulation of
beta-amyloid (a beta) proteins, as well as
decreases in important neurotransmitters --
especially acetylcholine.
Failure of memory functions is just one effect
of the process. Other cognitive and brain
functions gradually deteriorate.
EMF has been studied in relationship to
Alzheimer's Disease (AD) because of higher
incidence of this disease in EMF-exposed
workers. Several possible different, or
perhaps combined mechanisms, have been
proposed to explain this incidence: melatonin
effects, calcium efflux and neurotransmitter
alterations, breech of the blood-brain barrier,
aggravation of toxic insults.
This map provides links to overviews on the
topic as well as studies on various factors
associated with EMF and its impact on this
specific pathology.
Occupations with EMF risk
Blood-brain barrier and AD
Electric utility workers
BioInitiative Report 2012: Alzheimer's Important factors in AD Railway workers
Telephone workers
Disrupted biologic clock, AD Machinists
Compilation of abstracts on EMF and AD Welders
AD and EMF studies Increased a beta Carpenters
Six Studies: EMF and Alzheimer's Disease Seamstresses
Electricity and AD
Dressmakers
A beta protein, EMF Tailors
Occupational exposures and AD: a meta-analysis
RF, AD, transthyretin
Magnetic fields, dementia
Occupational exposures and AD: a collection of abstracts
Modern life, younger dementia
Increased a beta, electrical workers
Melatonin in humans
Light, ELF, circadian rhythm Full body EMF, Memory, Alzheimer's
and EMF studies
Video display units, melatonin Pulsed EMF increases b. amyloid
Day exposure, night melatonin Alzheimer's Disease 900 MHz, beta amyloid, other toxins
and ELF may increase neurodegeneration
Cellphone use, ELF, melatonin
Night exposure, night melatonin
EMF AD, EMF, Mechanisms and Prevention
Railway workers, ELF, melatonin Neurodegeneration
High power lines, female melatonin and occupational risks Alzheimer's disease
Is EHS a Pre-AD State?
EMF, signal transduction, melatonin receptors Increased dementia
3 studies of worker risk
Melatonin may protect against Alzheimer's disease
Occupational exposures
Environmental risk factors
Alzheimer's mouse study response Cell phones, power lines, AD Occupational MF exposures
Living near power lines, more AD risk MF exposures, electrical workers
Power-Line EMFs, New Focus on AD Occupational histories, HMO patients
Blood flow, barrier
Living near power lines, more AD risk, study and EMF studies ELF-EMF, AD and neurodegenerative disease
Mix of effects
Barrier damage Swedish twins, work exposures, dementia, ELF
Presentation on EMF and EHS
Prefrontal blood flow
Reduced local blood flow Home: Oscillatorium
Newest version this map
Interview on EMF, EHS and Alzheimer's Disrupted cerebral blood flow Date of this update: 04-08-16
Altered permeability of b/b barrier
"Several mechanisms have been proposed
Melatonin, AD, circadian rhythm disruption and studied in order to explain ELF-EMF
Mix with toxins Atrazine, ELF, mast cells potential actions on biological systems,
See Also: EMF Map on Melatonin Hypothesis involving melatonin and biosynthetic enzymes
EMF and metals in mouth
in the pineal gland (melatonin hypothesis),
Mercury release and cellphones oxidative stress, or Ca2+ efflux (release of
Melatonin... helps prevent... Alzheimers disease... There calcium ions from a sample into a
is strong evidence from epidemiologic studies that high surrounding solution) in immune system cells
( 10 milligauss or mG), longterm exposure to extremely Brain structure and neurons. Other potential pathways,
low frequency (ELF, 60 Hz) magnetic fields (MF) is and EMF studies Reduced neurites which may be involved in the relationship
associated with a decrease in melatonin production. Microglial activation between ELF-EMF and AD include apoptosis
BioInitiative Section 12 and necrosis in brain cells, effects on
MW/RF, MDA, brain biomagnetic particles reported in the human
ELF, cortical neurons, increased ROS brain or differential levels of electrosensibility
ELF-EMF levels between 0.6 and 1.2 T have been shown among the general population, but their
to consistently block the protective effects of melatonin. MW/RF, mitochondrial DNA, 8-OH-dG potential nexus with AD remain unknown."
BioInitiative Section 13 ELF, increased MDA, decreased melatonin activity Ana Garcia, et al