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RESUMEN
A pesar del cuerpo humano encontrar abundante fuentes de antioxidantes tales como el cido
ascrbico, cido rico y glutatin, experimentos en Vitro han encontrado que el consumo de
sulfurafanos ha reducido el ndice de muerte celular programada a travs, de la activacin celular
prolongada por estimulacin y recesin del oxgeno y el gen NFE2L2. Los agentes xenobioticos
metablicos se han reducido significativamente produciendo una desintoxicacin de perxidos
reactivos, incluyendo el perxido de hidrgeno y el peroxinitrito.
PALABRAS CLAVES:
ABSTRACT
Sulforaphan is a compound of the group of organic sulfides, and occurs in nature as a form of anti-
natural damage agent, this is created when the Myrosinase enzyme transforms Glucoraphanin into
Sulforaphane after molecular damage or tissue damage has occurred in plants of the family
Brassicaceae. Mixing compounds into a reactive oxygen species (EOR) by molecularly binding to
xenobiotoc elements in the human body.
Despite the human body finding abundant sources of antioxidants such as ascorbic acid, uric acid
and glutathione experiments in Vitro have found that consumption of Sulforaphan has reduced the
rate of programmed cell death through prolonged cellular activation by stimulation and recession
Oxygen and the NFE2L2 gene. Metabolic xenobiotic agents have been significantly reduced
producing a detoxification of being reactive peroxides including hydrogen peroxide and
peroxynitrite.
KEY WORDS
Glucoraphanin, Myrosinase enzyme , oxidative stress, Peroxinitrite, NFE2L2 gen, cancer