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Symptoms Present/A Rationale Justification

bsent
Chest pain Present According to Harrisons (2015). Pain is the - 2 days prior to admission at
most common presenting complaint in SPMC, patient has sudden
patients with STEMI. The pain is deep and onset of pressing chest pain
visceral; adjectives commonly used to graded 8-9 out of 10 non
describe it are heavy, pressing, and crushing, radiating associated with
although, occasionally, it is described as dyspnea.
stabbing or burning. - Increased in Troponin levels
(0.98 ng/ml)
Chest pain caused by acute coronary - final diagnosis of ACS
syndromes can come on suddenly, as is the STEMI
case with a heart attack. Other times, the
pain can be unpredictable or get worse even
with rest, both hallmark symptoms
of unstable angina. People who experience
chronic chest pain resulting from years
of cholesterol buildup in their arteries can
develop an acute coronary syndrome if a
blood clot forms on top of the plaque
buildup. (American Heart Association, 2015)

Nausea and Present Occurs due to pain, redirection of blood - 2 days prior to admission
Vommiting away from the gastrointestinal system, and patient experience nausea and
increased output of the sympathetic nervous vommiting.
system particularly due to stimulation of
abdominal splanchnic and vagal afferent
nerves. (Herlihy T, Mclvor ME, Cummings
CC, et al Am J Cardiol 60: 2022 Jul 1987 )
Shortness of Present Caused by reduced cardiac output leading to - 2 days prior to admission
breath ventricular failure. Heart can no longer patient experience dyspnoea
Dyspnoea adequately pump blood around the body, associated with acute coronary
causing a back-up of deoxygenated blood syndrome and increase cardiac
rate of 173 .
in the pulmonary circulation.
(Johansson ME, Wickman A, Skott O, Gan
LM, Bergstrm G, Blood pressure is the
major driving force for plaque formation in
aortic constricted ApoE-/- mice. J Hypertens
2006: 24: 20012008. )
Cardiogenic Present Acute damage to the myocardium results in - Patient is diagnose with ST-
shock reduced cardiac output. This leads to acute elevation myocardial
hypoperfusion and hypoxia of the tissues and infarction (STEMI).
organs, despite the presence of an adequate - Prior to admission patient
intravascular volume. This is a medical has increase cardiac rate of
emergency requiring immediate resuscitation 173 leading to ventricular
by treating the underlying cause: patient is tachycardia; cardio version
therefore admitted to Intensive Care Unit. was done @ 100 joules.
- Bp: 60/40

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