PATHOPHYSIOLOGY OF PRENANCY INDUCED HYPERTENSION

Precipitating Factors:
Predisposing Factors:
- Primigravida
- Age (31 years old)
- Lifestyle
- 180/100 mmHg
-
Decrease Responsiveness,
Endothelial Damage Effects of Prostaglandin
Decrease Blood pressure,
Virchows Triad

Vasospasm
NURSING DIAGNOSIS:
-Ineffective tissue perfusion r/t
Vasoconstriction of blood vessels
Vascular effect Kidney Effects
-Risk for fetal injury r/t reduced placental
Perfusion secondary to vasospasm
-Social Isolation r/t to prescribed bed rest Decreased Glomeruli filtration
-Risk for Injury Vasoconstriction
rate and increased permeability
-Risk for Bleeding of glomeruli membranes
-Risk for decreased cardiac output
Poor organ
Perfusion
- Pancreas Increased serum blood urea
- Liver nitrogen (BUN), Uric Acid and
- Brain Increased Blood Creatinine
- Retina Pressure
- Placenta
Decreased urine output
Increased pressure in the and Proteinuria
blood vessel

Diffusion of fluid from

Increased pressure in the placenta bloodstream into
interstitial tissue

Decreased oxygen in the Edema

placenta

Decreased oxygen of the fetus

Hypoxia