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Childhood Obesity
Obesity is an ongoing public health issue for adults and children. Children are rapidly
increasing in size and health risks are increasing as they grow. Stanhope and Lancaster (2014)
define obesity in children as, a BMI at or above the 95th percentile for children of the same age
and sex when plotted on the CDC growth charts (p. 348). It is hypothesized that factors
contribute to obesity in our youth today; including lifestyle, environmental concerns, dietary
choices, and genetics. In children ages 2 to 12; would an increase in activity and modified
nutritional intake decease BMI scores compared to children in the same age group who do not
have a change in lifestyle and dietary intake? This paper will examine some of the contributing
factors for childhood obesity and interventions needed to reduce the prevalence. Children need to
be guided into healthy lifestyles so they can develop into healthy adults. The school nurse is a
Stanhope and Lancaster (2014) identify the approximate prevalence of obesity in children
as, 17% for ages 2-5 years, 34% for ages 6 to 11 years, and 31% for ages 12 to 19 years (p.
348). There are many health risks associated with childhood obesity. Hypertension, high
cholesterol, and diabetes are among these risks. In a research article by Brady (2015), there are
vast hemodynamic factors associated with childhood obesity. The cardiovascular system
attempts to adapt to the increase in adipose tissue and remodeling occurs. Left ventricular
function. This research articles goes on to explain the negative effects on the heart due to LVH,
including volume overload and increased intravascular volume, which increase the negative
effects on the cardiovascular system. Hypertension can lead to other cardiovascular diseases and
CHILDHOOD OBESITY 3
increase the risk of negative cardiovascular outcomes. Brady (2015) writes, Obese children also
have greater carotid intima media thickness, an ultrasound measurement associated with vascular
disease, when compared to normal weight children (p. 2). Families need to be educated in order
to understand the severity of the health risks related to childhood obesity. An article by Karnik
and Kanekar (2015), indicates that education should be provided at an appropriate level for the
children. A study conducted by Raistenskis, J., Sidlauskiene, A., Cerkauskiene, R., Burokiene,
S., Strukcinskiene, B., and Buckus, R. (2015) analyzes the relationship of environment on
childhood obesity. The participants were between the ages of 11 and 14 years old. The study
compares the amount of daily physical activity of children who live in a small town to children
who live in the city. There were 118 children from a small town and 414 children from the city
noted in the study. Anthropometric measurements, including body mass index (BMI) and body
fat percentage were included in the study. The World Health Organization (WHO) child growth
underweight. Sedentary activities (screen time) were also compared with living environment by
providing a questionnaire with 47 activities to each participant. The study concluded that the city
children engaged in more daily physical activity than the small town children; with 25.5% of the
small town children being either overweight or obese compared to 18.6% of the city children
(Raistenskis et al., 2015). The children from both locations were noted to have high sedentary
time (screen time) periods and the data was not significant in that area of the study. The research
did indicate that youth physical activity programs in the small town area may need to be
CHILDHOOD OBESITY 4
incorporated. The study lacked information regarding nutrition and socioeconomic factors.
Karnik and Kanekar (2015) write, sedentary lifestyle is an important factor for obesity, as many
children spend most of their time in front of television sets, play video games, and watch
computers (p. 5). Lifestyle modifications are another area of education for parents of obese
children or children at risk for becoming obese. Exercise programs can be implemented within
the home with no cost to the family. Family participation in exercise can also create a positive
In addition, other environmental factors may increase the risk for childhood obesity. A
history and intake should be documented by the school nurse. Families should be educated on the
risks of common environmental risk factors. Chemicals used in production of plastics may put
children at risk. In a study completed by Vafeiadi, M., Roumeliotaki, T., Myridakis, A.,
Chalkiadaki, G., Fthenou, E., Dermitzaki, E., and Chatzi, L. (2016), bisphenol A (BPA) is
associated with childhood obesity. The study includes 235 two and a half year olds and 500 four
year olds. The participants were measured for weight and length at birth, and again at ages 2 to 4
years old for weight and height. BMI was calculated at both birth and 2 to 4 years of age. Urine
concentrations of BPA were analyzed among both age groups. A population of pregnant women
were also included in the urine study. At the age of 4, participants underwent cardiometabolic
testing, including a 5 min. resting blood pressure measurement and non-fasting blood sampling.
Lipids and adiponectin were among the blood testing completed. Vafeiadi et al., (2016),
indicated that BPA had no effect on weight at birth or by the age of 2.5 years. However, there
was an increase in BMI among the 4 years old age group with exposure to BPA prenatally and in
early childhood. BPA is an endocrine-disrupting chemical (Vafeiadi et al., 2016). There was
no significant difference in blood pressure or lipids and adiponectin levels in the study groups.
CHILDHOOD OBESITY 5
This study only included short term effects of BPA and socioeconomic status for the participants
was not indicated. Vafeiadi et al., (2016) also noted, BPA urine concentration varies throughout
the day and multiple daily samples were not collected. Education regarding environmental risks
should be provided at all well child visits and by the school nurse for all children regardless of
the risk for obesity. Low income families or families without health insurance may be directed to
Demmelmair, Grote, Prell, & Weber (2016), protein intake was examined in relation to
childhood obesity. The study was orchestrated with a population of 2,154 children. The
hypothesis by Koletzko et al., (2016) was named, The Early Protein Hypothesis and
hypothesized that early high protein intake caused increased plasma concentrations of insulin
releasing amino acids and therefore stimulated secretion of insulin-like growth factor (p. 303).
The study included self-reports from parents of 21 month olds and indicated that excessive
protein among the 21 month olds affected BMI scores for children into their 5th year of life. This
study lacked significant data regarding the sources of protein, other than noting that cows milk
was a common factor among the participants. Depending upon a childs growth curve, 2% or
skim milk may be indicated instead of Vitamin D milk. Healthy food choices and dietary intake
recommendations can be directed by the school nurse for students and their families. Education
and guidance from a pediatrician or dietician are also helpful. Healthy food choices implemented
at an early age reduce the risk for poor nutritional habits as children grow.
Guzey, Wardle, & Wichstrm, (2016) conducted a study with 652 participants, ranging in age
from 4 to 8 years old. Children began the study at age 4 and were reassessed, including BMI
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scores; at age 6 and again at age 8. Genotype, adipose and appetite traits were analyzed. Salvia
samples were collected and analyzed for genetic loci. There were alleles across 32 single
nucleotide polymorphisms (SNPs) included in the study (Steinsbekk et al., 2016). The SNPs
were used to calculate genetic risk for developing obesity. When the participants were 6 years
old, their parents were given a 5 item Likert scale questionnaire. The Childrens Eating Behavior
Questionnaire (Steinsbekk et al., 2016) was used to analyze appetite. Although BMI was only
measured a total of three times throughout the study, Steinsbekk et al., (2016) concluded that
children with an increased genetic risk for obesity based on the SNPs, had a higher BMI and
percentage of body fat. The study noted that appetite had no influence on weight gain. The data
regarding the genetic variations with the 32 SNPs was also noted to be incomplete (Steinsbekk et
In conclusion, childhood obesity is an ongoing public health issue. The school nurse is a
major resource for children and their families. The school nurse can provide education regarding
prevention and management of obesity. The school nurse can also initiate information for health
management with primary care providers and additional community resources. Financial services
to purchase healthy foods are available to low-income families. The school nurse plays a vital
role in primary prevention of childhood obesity by influencing children and teaching healthy
food choices and the importance of daily exercise. Providing screenings and completing health
history intakes is a means of secondary prevention by the school nurse. The school nurse utilizes
tertiary prevention by providing care for the children who are already battling with childhood
obesity and by working with their families to promote continuity of care while collaborating with
References
Brady, T. M. (2015). The role of obesity in the development of left ventricular hypertrophy
doi:10.1007/s11906-015-0608-3
Karnik, S., & Kanekar, A. (2015). Childhood obesity: a global public health crisis. International
Koletzko, B., Demmelmair, H., Grote, V., Prell, C., & Weber, M. (2016). High protein intake in
young children and increased weight gain and obesity risk. American Journal of Clinical
Raistenskis, J., Sidlauskiene, A., Cerkauskiene, R., Burokiene, S., Strukcinskiene, B., &
Buckus, R. (2015). Physical activity and sedentary screen time in obese and overweight
Stanhope, M., & Lancaster, J. (2014). Foundations of nursing in the community: Community-
Steinsbekk, S., Belsky, D., Guzey, I. C., Wardle, J., & Wichstrm, L. (2016). Polygenic risk,
appetite traits, and weight gain in middle childhood. JAMA Pediatrics, 170(2), e154472.
doi:10.1001/jamapediatrics.2015.4472
Vafeiadi, M., Roumeliotaki, T., Myridakis, A., Chalkiadaki, G., Fthenou, E., Dermitzaki, E.,
Chatzi, L. (2016). Association of early life exposure to bisphenol A with obesity and
doi:10.1016/j.envres.2016.01.017