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  • Pa Tho of Prostatic Cancer

    Transféré par

    Jai - Ho
    244 vues2 pages
    V. PATHOPHYSIOLOGY This section summarizes the pathophysiology of prostatic cancer with bone metastasis in an 81-year-old male patient with multiple predisposing factors including age, gender, vitamin D deficiency, and sedentary lifestyle. It describes how vitamin D deficiency can lead to decreased calcium absorption and increased production of dihydrotestosterone, resulting in uncontrolled tumor growth in the prostate and bones. It also lists signs and symptoms such as loss of appetite, abnormal vital signs, and lab abnormalities that resulted from the progression of the disease and complications.

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    Pa Tho of prostatic cancer

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    V. PATHOPHYSIOLOGY This section summarizes the pathophysiology of prostatic cancer with bone metastasis in an 81-year-old male patient with multiple predisposing factors including age, gender, vitamin D deficiency, and sedentary lifestyle. It describes how vitamin D deficiency can lead to decreased calcium absorption and increased production of dihydrotestosterone, resulting in uncontrolled tumor growth in the prostate and bones. It also lists signs and symptoms such as loss of appetite, abnormal vital signs, and lab abnormalities that resulted from the progression of the disease and complications.

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    Attribution Non-Commercial (BY-NC)
    Téléchargez comme DOC, PDF, TXT ou lisez en ligne sur Scribd
    Signaler comme contenu inapproprié
    244 vues2 pages

    Pa Tho of Prostatic Cancer

    Transféré par

    Jai - Ho
    V. PATHOPHYSIOLOGY This section summarizes the pathophysiology of prostatic cancer with bone metastasis in an 81-year-old male patient with multiple predisposing factors including age, gender, vitamin D deficiency, and sedentary lifestyle. It describes how vitamin D deficiency can lead to decreased calcium absorption and increased production of dihydrotestosterone, resulting in uncontrolled tumor growth in the prostate and bones. It also lists signs and symptoms such as loss of appetite, abnormal vital signs, and lab abnormalities that resulted from the progression of the disease and complications.

    Droits d'auteur :

    Attribution Non-Commercial (BY-NC)
    Téléchargez comme DOC, PDF, TXT ou lisez en ligne sur Scribd
    Signaler comme contenu inapproprié
    sur 2

    V.

    PATHOPHYSIOLOGY

    Legend: Disease process Signs and Symptoms Management

    Predisposing Factors:
    Age (81 yrs. Old)
    Precipitating Factors: Gender (Male)
    Poor ejaculation Vitamin D deficiency
    Sedentary lifestyle
    Loss of appetite
    Vitamin D deficiency Decrease
    Esome calcium
    Increase in absorption
    prazole
    magne gastric secretions Bicalutamide
    Increase production of
    sium dihydrotestosterone

    Irritation to the Decrease


    mucosal lining of the serum
    stomach Accumulation of calcium
    dihydrotestosterone in the level of
    prostate

    Ulceration of the GI lining -Zolendronic acid


    -calcium
    gluconate
    Develops rates of more cell
    division than cell death
    Bleeding in the -Sulperazone
    stomach -Tranexemic
    acid

    Cell hypertrophy in the


    Decrease prostate
    -decrease tissue dopamine
    Abnormal
    in RBC perfusion decrease in
    going to the BP of 70/40
    of 11.3
    mmHg
    GI Uncontrolled tumor growth

    - Decrease
    melena peristalsis Abnormal
    Transitional cell Abnormal
    increase
    morphology arrive from in heart decrease in
    urothelial lining of the rate of hematocrit of
    Paralytic ileus 105 bpm
    prostatic urethra
    34.0 and
    hemoglobin
    of 11.3

    A case study on Prostatic Cancer with Bone


    Metastasis (Thorax) (thorax) Page 19
    Proliferation of cell growth
    in multiple zones of the
    prostate
    Absence Hard and
    of bowel small
    sounds amount
    of stool
    Formation of tumor in the
    prostate covering half of the
    lobe

    Continuous proliferation of
    tumor involving less than or
    equal to one lobe

    Tumor involving both lobes


    extending through the
    cefuroxime
    prostatic capsule

    Presence of infection Compression of urethral


    Prostatic enlargement meatus

    Cancer cells proliferates


    in the thorax Benign tumor becomes oliguria Insertion of
    malignant catheter

    Damage to the cilia


    Retention of metabolic
    Invasion to adjacent waste
    structures other than seminal
    Accumulation of copius vesicles like the pelvis
    secretion

    -increase BUN of
    68 mg/dl
    Non- -increase serum
    productive Albuterol Pelvic pain creatinine of 1.40
    cough radiating to mg/dl
    lower
    extremities
    -paracetamol

    A case study on Prostatic Cancer with Bone


    Metastasis (Thorax) (thorax) Page 19

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