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Submitted by:
Joher Bolante Mendez, Jr.
MD-II
Submitted to:
Dr. Remo Benliro, Jr.
Dr. Agnes Segunda Gerasmo
Dr. Henry Gonzales
Instructors
THE CASE
I. Vital Information
• Age: 40 y/o
• Sex: Male
• Occupation: Farmer; Barangay Councilor
Vital Signs
• BP: 120/80 mmHg
• Cardiac Rate- 85 bpm
• RR: 18 cpm
• Temp: 37.0 degrees centigrade
PE Findings:
• Anicteric sclerae pinkish conjunctivae
• No nasal congestion or polyp
• Intact ear drum both ears, (-) cerumen
• (-) Cervical Lymph Adenopathy or neck bruit
• (-) enlarged tonsils; Symmetrical Chest Expansion
• (-) rales (-) wheezing
• Cardiac findings with regular cardiac rate and rhythm no murmur
• Abdomen flat NABS (-) tenderness (-) masses
• Both upper and lower extremities unremarkable findings.
Neuro Exam:
• Intact other Cranial nerves, (-) horizontal Nystagmus
• Motor 5/5 on all extremities and 100% sensory on all areas.
• Reflexes ++
• Visual Acuity 20/20
INTRODUCTION
Dizziness
• “dizzy” (“hilo”, “liyo”)
• general term that describes sensations of imbalance and unsteadiness
• 4 Categories (Post & Dickerson, 2010)
Vertigo
• sensation of spinning, whirling, or falling, either of the self (I’m spinning) or the environment
(the room is spinning)
• a symptom, rather than a disease
• onset is abrupt and accompanied by nausea, vomiting, and disequilibrium
• type of dizziness that involves a false sensation that one's self or the surroundings are
spinning, swaying or tilting, usually accompanied by loss of balance & nystagmus
• Physiologic Vertigo: sustained head rotation
• Pathologic Vertigo : vestibular function
• Types and Causes
TYPE STRUCTURES INVOLVED CAUSES / ETIOLOGIES
• Benign paroxysmal positional
vertigo (BPPV)
• Vestibular neuritis
lesions affecting the • Meniere's disease
PERIPHERAL VERTIGO inner ear and cranial nerve • Acoustic neuroma
• Aminoglycoside toxicity
(Subjective Vertigo) VIII
• Semicircular canal dehiscence
I’m spinning
syndrome
• Perilymphatic fistula
• Herpes zoster oticus
(Ramsay Hunt syndrome)
• Vestibular migraine
• Brainstem stroke
lesions affecting the • Multiple sclerosis
CENTRAL VERTIGO brainstem and cerebellum • Ischemic or hemorrhagic
damage to the cerebellum
(Objective Vertigo)
The room is spinning
Epidemiology
• Studies show that about a third of cases of dizziness are vertigo.
• Most common are viral, BPPV or Meniere's disease.
• Prevalence estimates for vertigo are ~5%, for BPPV 1.6%. Meniere's disease
~0.5%
• Philippine General Hospital (2015)
• Department of Otorhinolaryngology, there were 103 cases out of 3056 new
patients seen are related to vertigo
• Department of Family Medicine, out of 20, 902 new patient consults, there were
528 related to to dizziness and vertigo
• Department of Neurosciences, out of 4547 patients, there were 42 consults for
vertigo and dizziness
• University of Santo Tomas, OPD (2014)
• 688 patient visits for dizziness
APPROACH TO DIAGNOSIS
A. SIGNIFICANT FINDINGS
PATIENT’S HISTORY
PHYSICAL EXAMINATION
PERTINENT NEGATIVES
• (-) diplopia
• (-) vomiting
• (-) weakness
• (-) blurring of vision
• (-) facial asymmetry
Differential diagnostic characteristics of central versus peripheral vertigo. Brainstem signs
may include motor and sensory deficits, hyperreflexia, positive Babinski sign, dysarthria, and
limb ataxia.
B. DIFFERENTIAL DIAGNOSES
• Vestibular Neuritis
• Labyrinthitis
• Benign Paroxysmal Positional Vertigo
• Meniere’s Disease
• Vestibular Neuritis
• inflammation of the inner ear and/or the nerve connecting the inner ear to the brain.
• generally caused by a viral infection, usually, Herpes Viruses.
RULE IN
• Meniere’s disease also known as idiopathc endolymphatic hydrops, is a disorder of the inner
ear resulting in the clinical triad of vertigo, tinnitus, and hearing loss. There is an abnormal
inner ear fluid balance caused by a malabsorption in the endolymphatic sac or a blockage in the
endolymphatic duct.
• Endolymphatic hydrops, a dilation in the endolymphatic space, develops, and either increased
pressure in the system or rupture of the inner ear membrane occurs, producing symptoms
Meniere’s disease.
▪ Middle stage
▪ vertigo episodes continue, but are usually less severe.
▪ tinnitus and hearing loss, get worse
▪ some patients during this stage may experience periods of complete remission
▪ periods of remission can last several months.
▪ Late stage
▪ vertigo episodes become even less frequent, and in some cases never come back
▪ balance problems, though, and may continue
▪ Patients will feel especially unsteady when it is dark and they have less visual input to
help maintain balance.
▪ Hearing and tinnitus typically get progressively worse.
PATHOPHYSIOLOGY
Hearing Loss
Vertigo Tinnitus
EVALUATION / WORK-UP
D. C-Spine X-Rays
• An X-Ray of the cervical spine may be performed to check for broken bones or any neck
injuries.
G. Spinal Tap
• This test involves the removal of fluid from the spinal cord to test for infection, bleeding
in the brain and spinal cord or
H. Positional testing
• We try to move the patient's head in different directions to check for inner ear problems.
A physical exam may also confirm Nystagmus. Tests on balance and coordination may
also be executed to see if the brain is performing properly.
I. Hearing test.
• This is to assess if the middle ear, cochlea and auditory nerve are functioning properly.
(Weber-Rinnetest)
K. Blood test
• Blood sugarlevels may be checked to see if this contributes to the patient's vertigo; CBC.
L. Electrocardiogram(ECG)
• This is to check for damage in different areas of the heart.
•
M. Rotary-chair testing
• This test also measures inner ear movement by assessing eye movement. It is usually
better tolerated than the ENG.
• The patient sits in a chair in a small, dark booth. Electrodes are placed near the eyes and
a computer-guided chair rotates gently back and forth at varying speeds.
• The movement stimulates the inner balance system and causes nystagmus (eye
movements) that are recorded by a computer and monitored with an infrared camera.
• Rotary chair testing does not provide specific diagnostic information about each ear
individually - unlike the ENG.
APPROACH TO MANAGEMENT
1. Nonsurgical Management
• Teach patients to move the head slowly to prevent worsening of the vertigo.
• Nutrition and lifestyle changes can reduce the amount of endolymphatic fluid.
• Encourage the patients to stop smoking because of the blood vessels constricting
effects.
• Stress Reduction
• Changing positions slowly to avoid disorientation.
• Controlling high blood pressure and high cholesterol through a low salt diet.
• Focusing on distant objects instead of one's feet while walking.
• When riding a car, sitting in the front seat and focusing on distant objects.
• Updating eye glasses and hearing aid prescriptions.
• Avoiding activities that involve moving the head up and down repetitively or keep the
head tilted back a long time, such as painting.
Physical therapy
• Repositioning maneuvers.
2. Pharmacologic Management
• Drugs for nausea - prochlorperazine has been shown to be effective in the treatment of
nausea during a vertigo episode.
• Nicotonic Acid
• Antihistamines
• Diphenhydramine hydrochloride (Benadryl, Allerdryl)
• Dimenhydrinate (Dramamine, Gravol)
• Antiemetics
• Chlorpromazine hydrochloride (Thorazine, Novo- Chlorpromazine)
• Droperidol (Inapsine)
• Promethazine (Phenergan)
• Ondansetron (Zofran)
3. Surgical Management
• Endolymphatic sac decompression - a small portion of bone is removed from over the
endolymphatic sac. Occasionally, a shunt is placed (a tube that drains excess fluid from
the inner ear).
• Hearing aid - a patient with Meniere's disease who has suffered hearing loss from the
affected ear may benefit from a hearing aid. A hearing aid is an instrument to help in
hearing.
• Labyrinthectomy - a portion of the inner ear is surgically removed. This takes away
both the hearing and balance function of the affected ear. This procedure is only done if
the patient is either totally, or almost totally deaf in that ear.
• Vestibular nerve section - the vestibular nerve is cut. This nerve connects the balance
and movement sensors in the inner ear to the brain. A vestibular nerve section is aimed at
preserving hearing in the affected ear, while addressing the problems with vertigo.
SUMMARY OF CARE / APPROACHES
PATIENT CENTERED
• Avoid hazardous activities when you are dizzy, such as driving an automobile or operating
dangerous equipment, or climbing a step ladder, etc.
• Avoid rapid changes in position, especially from lying down to standing up or turning around
from one side to the other.
• Avoid extremes of head motion (especially looking up) or rapid head motion (especially
turning or twisting).
• Eliminate or decrease use of products that impair circulation, e.g. nicotine, caffeine, and salt.
• Minimize your exposure to circumstances that precipitate your dizziVness, such as stress and
anxiety or substances to which you are allergic.
• Annual Audiometry.
• Treat infections, including ear infections, colds, flu, sinus congestion, and other respiratory
infections.
FAMILY FOCUSED
• “Fall-proofing” their home by covering sharp corners with foam and removing rugs and
masts from slippery areas and stair landings.
• Encourage or help in motivating the patient to stop drinking alcohol or if not then reduce the
alcohol intake.
• Try to communicate openly with the patient with regards to use of illicit drugs.
• Inform or give the people from the neighborhood an idea about what is the present condition
of the patient. With this, they could help the patient to change his lifestyle and be guided if
an episode of vertigo happens outside their house.
• Recommend to the LGU or barangay officials to have a program that will entertain or
conduct a program that aims for awareness, information, and participation that will benefit
debilitated people or has a condition like Tomas in the community.
IMPACT OF ILLNESS
BRGY COUNCILOR
As a brgy coucilor, social interaction and duties come together. Their role is to act as
peace officers in the maintenance of public order and safety. If symptoms come on and
off, work will be affected and duties as a peace-officer will be at stake.