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Cardiac
Tamponade
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. pericardium is a double-walled sac containing the heart and the roots of the great
vessels. The pericardial sac has two layers, a serous layer and a fibrous layer. It
encloses the pericardial cavity which contains pericardial fluid.
The pericardium fixes the heart to the mediastinum, gives protection against infection and
provides the lubrication for the heart
The pericardium is a tough double layered fibroserous sac which covers the heart. There
are two layers to the pericardial sac: the outermost fibrous pericardium and the inner
serous pericardium.
Fibrous pericardium
The fibrous pericardium is the most superficial layer of the pericardium. It is made up
of dense and loose connective tissue, which acts to protect the heart,
Serous pericardium
The serous pericardium, in turn, is divided into two layers, the parietal pericardium, which
is fused to and inseparable from the fibrous pericardium, and the visceral pericardium,
When the large amounts of fluid accumulate (pericardial effusion) or when the
pericardium becomes scarred and inelastic, one of three pericardial compressive
syndromes may occur:
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Definition
Cardiac tamponade is a medical emergency.
Cardiac tamponade is the accumulation of pericardial fluid, blood, pus, or air
within the pericardial space that creates an increase in intrapericardial
pressure, restricting cardiac filling and decreasing cardiac output. Cardiac
tamponade can be fatal if it is not quickly diagnosed and treated
promptly. The diagnosis is based on clinical suspicion and supported by
evidence of hemodynamic compromise on echocardiography.
Epidemiology
The frequency of tamponade is unclear. One estimate from the United States places it at
2 per 10,000 per year. It is estimated to occur in 2% of those with stab or gunshot
wounds to the chest.
Pathophysiology
The outer layer of the heart is made of fibrous tissue which does not easily
stretch, so once fluid begins to enter the pericardial space, pressure starts to
increase.
If fluid continues to accumulate, each successive diastolic period leads to less
blood entering the ventricles. Eventually, increasing pressure on the heart forces
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the septum to bend in towards the left Pathophysiolo Pathophysiology gy
ventricle, leading to a decrease instroke volume. This causes the development
of obstructive shock, which if left untreated may lead to cardiac arrest (often
Causes:
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accidental perforation after cardiac catheterization, angiography, or insertion of a
pacemaker
punctures made during placement of a central line, which is a type of catheter that
administers fluids or medications
cancer that has spread to the pericardial sac, such as breast or lung cancer
a heart attack
kidney failure
Risk factors
malignancy
aortic dissection
purulent pericarditis
large idiopathic pericardial effusion
Clinical picture:
weakness
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trouble breathing or taking deep breaths
rapid breathing
abdominal pain
confusion
fever
Diagnosis:
Cardiac tamponade often has three signs can be recognized. These signs are
commonly known as Beck’s triad. They include:
low blood pressure and weak pulse because the volume of blood your heart is
pumping is reduced
extended neck veins because they’re having a hard time returning blood to
your heart
a rapid heartbeat combined with muffled heart sounds due to the expanding
layer of fluid inside your pericardium
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Other diagnostic tests may include:
a thoracic CT scan to look for fluid accumulation in the chest or changes to
the heart
a magnetic resonance angiogram to see how blood is flowing through your
heart
x ray
ECG
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Differentials
o Constrictive pericarditis
o Restrictive cardiomyopathy
Cardiogenic shock
●Constrictive pericarditis – Constrictive pericarditis is the result of scarring and
consequent loss of elasticity of the pericardial sac. Pericardial constriction is
typically chronic, but variants include subacute, transient, and occult constriction.
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feature of both cardiac tamponade and constrictive pericarditis is greatly
enhanced ventricular interaction or interdependence, in which the hemodynamics
of the left and right heart chambers are directly influenced by each other to a
much greater degree than normal.
Treatment:
Hospital management
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References
1. Spodick, DH (Aug 14, 2003). "Acute cardiac tamponade". The New England Journal
of Medicine.349 (7): 684–90. doi:10.1056/NEJMra022643.PMID 12917306.
2. : Richardson, L (November 2014). "Cardiac tamponade". JAAPA : Official Journal of
the American Academy of Physician Assistants. 27 (11): 50–
1.doi:10.1097/01.jaa.0000455653.42543.8a.PMID 25343435.
3. Kahan, Scott (2008). In a Page: Medicine. Lippincott Williams & Wilkins.
p. 20. ISBN 9780781770354.Archived from the original on 2016-10-02.
4. Schiavone, WA (February 2013). "Cardiac tamponade: 12 pearls in diagnosis and
management". Cleveland Clinic Journal of Medicine. 80 (2): 109–
16.doi:10.3949/ccjm.80a.12052. PMID 23376916.
5. Sagristà-Sauleda, J; Mercé, AS; Soler-Soler, J (26 May 2011). "Diagnosis and
management of pericardial effusion". World Journal of Cardiology. 3 (5): 135–
43.doi:10.4330/wjc.v3.i5.135. PMC 3110902.PMID 21666814.
6. Bodson, L; Bouferrache, K; Vieillard-Baron, A (October 2011). "Cardiac
tamponade". Current Opinion in Critical Care. 17 (5): 416–
24.doi:10.1097/mcc.0b013e3283491f27. PMID 21716107.
7. : Porth, Carol; Carol Mattson Porth (2005).Pathophysiology: concepts of altered
health states (7th ed.). Hagerstwon, MD: Lippincott Williams & Wilkins.ISBN 978-0-
7817-4988-6.
8. Gwinnutt CL, Driscoll PA (2003). Trauma Resuscitation: The Team Approach (2nd
ed.). Oxford: BIOS. ISBN 978-1-85996-009-7.
9. Isselbacher EM, Cigarroa JE, Eagle KA (Nov 1994). "Cardiac tamponade
complicating proximal (retrograde) aortic dissection. Is pericardiocentesis
harmful?".Circulation. 90 (5): 2375–8.doi:10.1161/01.CIR.90.5.2375. PMID 7955196.