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APRIL 5–8, 2018

20 mg IV prednisolone. On the fifth day of treatment, minimal pericar-

Topic: AJC » Cardiac Imaging - dial effusion was detected in the ECO (Figure 2).
When symptoms of cardiac disease occur in a RA patient, cardiac in-
Echocardiography volvement associated with drug use should be kept in mind and the drug
should be discontinued immediately.

PP-612
Topic: AJC » Arrhythmias and
Acute Pericarditis Due to the Use of Sulphalazine in Rheumatoid
Arthritis Patient. Hakki Simsek, and Ahmet Ferhat Kaya. Dicle Antiarrhythmic Therapy
University Cardiyology Hospital, Diyarbakir.

Pericarditis is a life-threatening condition that can be seen in rheu- PP-622


matic diseases. It may also depend on the medication used as well as
the disease itself. A Life-Threatening Condition: Hyperkalemia-Induced Complete
A 47-year-old woman was admitted to the emergency department with Heart Block. Uğur Aksu1, Zakir Lazoglu2, Kamuran Kalkan1,
complaints of chest pain, palpitation and shortness of breath. 6 years before Selim Topcu2, and Ibrahim Halil Tanboga2. 1Erzurum Regional
methotrexate and corticosteroid treatment were started with diagnosis Training and Research Hospital, Erzurum; 2Ataturk University,
RA. And 3 weeks before sulphasalazine treatment was added. Physical Erzurum.
examination showed decreased heart sounds and dyspnea and orthop-
nea. There was minimal ral in the bilateral lung sub-zones. Blood pressure Hyperkalemia is a frequently encountered electrolyte abnormality. Peaked
was 110/70 mm Hg, pulse rate was 96/min and fever was 37°C. His labo- T waves are usually the first ECG findings. If left untreated, progres-
ratory showed leucocyte: 18,000, Hgb: 11.5 g/dl, platelet count: 365,000, sion of hyperkalemia leads to slowing of conduction, wide, low p waves,
erythrocyte sedimentation rate (ESR): 34 mm/h, CRP: 18.5 mg/dl. On long PR interval, and a wide QRS complex. Ultimately it might cause
ECG we found sinus tachycardia (102/min). Transthoracic malignant ventricular arrhythmias and asystole. Renal dysfunction and
echocardiography revealed pericardial effusion in the moderate to severe electrolyte imbalances caused by cardiac procedures and chronic medi-
(Figure 1). The patient’s fluid was evacuated with pericardiocentesis. There cations may be precipitate hyperkalemia. Therefore physicians should
was no identified any microorganism in the patient’s pericardiocentesis be aware of this procedures and medications during medical history taking.
fluid and blood cultures. Pericarditis due to sulfalazine use was diagonsed In this case, we present a case of malignant AV block resolved by di-
finally. Sulphalazin therapy was cessasieted and the patient was given alysis, in a patient who was receiving medical treatment for cardiac failure.
Case report: A 69-year-old female was admitted to the hospital with
a new onset dyspnea. Two months ago, she underwent to coronary an-
giography due to severe coronary artery stenosis and her home drug
regimen included metoprolol 50 mg daily, perindopril 10 mg daily, aspirin
100 mg daily, clopidogrel 75 mg daily and atorvastatin 20 mg daily. The
patient’s vital signs on admission were temperature, 36,4°C; pulse rate,
P
30 beat per minute and regular; blood pressure, 140/90 mm Hg, equal
in both arms. Respiratory rate 24 breaths/minute. She was in acute re-
O
spiratory distress with fluctuating consciousness, oxygen saturation 85% S
while she was breathing room air. Initial ECG showed wide QRS complex
with an S wave that merges with a peaked T wave without an isoelec- T
Figure 1. tric ST segment (Figure 1A). After initial examination patient clinical
and electrocardiographic features hyperkalemia was suspected and calcium
E
gloconate was administered. After a while patient’s heart block was re-
solved (Figure 1B). Laboratory tests revealed a significantly high potassium
R
(K) level (8,1 meq/ml). Patient was immediately transferred to hemo-
dialysis unit for normalization of K levels. Through 3 hours of
hemodialysis, serial control ECGs revealed that hyperkalemia related wide A
QRS complex pattern at a rate of 75 beats per minute were transform-
ing to a normal sinus rhythm at a rate of 95 beats per minute (Figure 1C)
B
as the K levels were brought to normal range. S
T
R
A
C
T
S
Figure 2. Figure 1.
® th
The American Journal of Cardiology APRIL 5–8, 2018 14 INTERNATIONAL CONGRESS OF UPDATE IN CARDIOLOGY AND e161
CARDIOVASCULAR SURGERY / Poster
APRIL 5–8, 2018

Increasing cardiac interventions and anti hypertensive medication use report of a 1-year natural course. Tex Heart Inst J 2005;32:589–
are frequently encountered reasons for acute renal failure. Moreover, 594.
cardiac failure may also result in prerenal azotemia. Therefore, cardiac 7. Yazkan R, Çeviker K. Pulmoner arterin cerrahi gerektiren edinsel
history, medication use and interventions requiring intravenous con- hastalıkları. Türk Göğüs Kalp Damar Cerrahisi Dergisi 2015;23:792–
801.
trast media are points of interest for emergency room, targeting early
recognition of precipitating factors and prompt initiation of appropri-
ate therapy.
Topic: AJC » Interventions for Peripheral
Arterial Diseases
Topic: AJC » Diagnosis and Treatment of
Pulmonary Hypertension PP-632
Endovascular Approach for Acute Limb İschemia: Mechanical
Thrombectomy With Angiojettm Thrombectomy
PP-625 System. Onur Saydam1, Deniz Serefli1, Aysen Yaprak Engin1,
Pulmonary Artery Aneurysm. Şahin Karakılıç. Dokuz Eylül Mehmet Atay2, and Ayse Gul Kunt1. 1Tepecik Education and Research
University Hospital, Izmir. Hospital, İzmir; 2Bakırköy Dr. Sadi Konuk Training and Research
Hospital, İzmir.
Objective: Pulmonery artery aneurysm is a rare abnormality of pulmo-
nary arteries.1 Etiology of disease are caused trauma, infections, Behcet’s Acute arterial occlusion (AAO) is defined as a sudden loss of limb per-
disease, pulmonary hypertension, congenital heart disease and neoplasm.2 fusion and considered a vascular emergency. AAO is associated with
Isolated or idiopathic pulmonary artery aneurysms are more rare.3,4 Big increased morbidity, significant disability, and emergent operation in high-
part of patients are asymptomatic.5 Another part of patients has symp- risk patients. The most common cause is in situ thrombotic occlusion
toms; cough, hemoptysis, chest pain and the other non-spesific symptoms. and initiating event is a preexisting history of peripheral artery disease
We write this presantation about a patient who has pulmonary artery an- (PAD). AAO require emergent vascular surgery consult. Open Surgery
eurysm with infundibular stenosis. is still the gold standard. However recent advances in endovascular in-
Methods: This patient is 58 aged women who earlier had aortic an- terventions suggest that the endovascular approach can represent an
eurysm and treated endovascular technique. Her physical examination alternative therapeutic strategy especially high-risk patient with preex-
was smoothly. Her control echocardiography results are pulmonary arter isting chronic peripheral arterial disease. We present a 65-year-old man,
aneurysm, pulmonar valv failure, 52/20 mm Hg gradient and pulmonar with a history of chronic obstructive pulmonary disease and peripheral
stenosis. The next examination was right heart catheterisation angiog- arterial disease who was consulted to our clinic with 2-day history of
raphy. Images showed patient’s aneurysms size 7 cm and an infundibular progressive limb pain after radical cystectomy operation. Computed to-
stenosis. Aneurysm treated a conduit with bioprosthetic pulmonar valv. mography (CT) angiography revealed acute left iliac artery and common
P And the infundibular stenosis was repaired with resection. Right ven-
triculotomy closed with bovine pericardium.
femoral artery occlusion. Initially the patient received subcutaneous low
molecule weight heparin. Considering his severe clinical status and pre-
O Results: Aortic aneurysm caused a treatment with endovascular re- existing peripheral arterial disease, we decided to perform percutaneous
mechanical thrombectomy. Retrograde cannulation of right common
paired patient’s control examination showed us an asymptomatic
S pulmonary artery aneurysm. Most rare pulmonary arter aneurysm with femoral artery and left superficial femoral artery with 7 Fr introducer
sheath. Pelvic arteriography revealed total occlusion of left iliac artery
T infindibular stenosis are replaced by a valved conduit. Postoperative process
was smoothly. She discharged with no problem. and common femoral artery occlusion. Under heparinization mechani-
E Conclusions: When pulmonary truncus’ diameter is over 30 mm, it’s
called pulmonary artery aneurysm.1 Autopsy results shows 1/14,000 rate
cal thrombectomy with AngioJetTM Thrombectomy System was
performed. After mechanical thrombectomy severe stenosis was de-
R and it’s a rare disase. Pulmoner artery hypertension, some infections (etc. tected in left common iliac artery and thromboembolic formation in
common femoral artery. 8 mm × 37 balloon expandable stent was de-
tbc, syphilis), syndromes (etc. marfan, hughes-stovin), traumas are related
between pulmonary artery aneurysm. Idiopathic one is more rare than ployed at left common iliac artery and 7 mm balloon angioplasty was
performed at common femoral artery. Control arteriography showed total
A others.1–7
Usually it’s asymptomatic, when it shows symptoms, like cough, he- revascularization of iliac and femoral arteries and the patients no longer
B moptysis, fever, breath disorders, chest pain, more dangerous.6,7 Most
important complication is disection which cause of death. Thats’s the
demonstrated the above-mentioned symptoms. Duration of the proce-
dure was approximately 35–40 min. These less invasive techniques
S reason why pulmonary artery aneurysm have to fix by surgery.6,7 constitute an option that is bettertolerated in medically compromised pa-
tients. Unlike open surgery during endovascular procedures acute
T preexisting peripheral arterial pathologies can be detected and solved in
1. Shih HH, Kang PL, Lin CY, Lin YH. Main pulmonary artery aneu- same session.
R rysm. J Chin Med Assoc 2007;70:453–455.

A 2. Hammad AM, Al-Qahtani SM, Al-Zahrani MA. Huge pulmoner artery


aneurysm. Can Respir J 2009;16:93–95.
Management of Hypertension in Patients with Chronic Kidney
Disease. Burak Sayin. Asst. Prof. Dr, Department of Nephrology,
C 3. Bartter T, Irwin RS, Nash G. Aneurysms of the pulmonary arteries.
Chest 1988;94:1065–1075.
Baskent University.
Introduction
T 4. Fang CC, Tsai CC. Idiopathic pulmonary artery aneurysm. J Formos
Med Assoc 1996;95:873–876.
Chronic kidney disease (CKD) may be described as documented kidney
damage and/or loss of kidney function with a glomerular filtration rate
S 5. Zhao YJ, Cheng XS. An analysis of 21 cases of idiopathic dilata-
tion of the pulmonary artery. Zhonghua Nei Ke Za Zhi 1992;31:24– (GFR) lower than 60 ml/min/1.73m2 for over 3 months of period. Chronic
25. kidney disease is a growing public health problem that has been affect-
6. Smalcelj A, Brida V, Samarzija M, Matana A, Margetic E, Drinkovic ing more than 500 million people worldwide. CKD has a close and
N. Giant, dissecting, high-pressure pulmonary artery aneurysm: case reciprocal relationship with hypertension, because they are both a cause

e162 The American Journal of Cardiology® APRIL 5–8, 2018 14th INTERNATIONAL CONGRESS OF UPDATE IN CARDIOLOGY
AND CARDIOVASCULAR SURGERY / Poster

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