@siradrianbond - Coronavirus Exposed Part 1 - Communist Coverup or Pandemic Bioweapon of Mass de

Coronavirus Exposed, Part 1:
Communist Coverup, or Pandemic

Bioweapon of Mass Destruction?
Coronavirus 2019-nCoV, able to enter and infect human cells’ ACE2 receptor via its spike protein.
The official story about Coronavirus 2019 nCoV is that it “appears to

have originated in the Huanan Seafood Wholesale Market in Wuhan, a
https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

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Chinese city about 650 miles south of Beijing that has a population of
more than 11 million people.” This tale has been officially reported as
early as January 9th by CCP’s state-owned and operated news
channel, Xinhuanet, New-type coronavirus causes pneumonia in
Wuhan: expert, reported by local Chinese authorities to the US
National Library of Medicine database, Outbreak of Pneumonia of
Unknown Etiology in Wuhan China: the Mystery and the Miracle and to
the International Journal of Infectious Diseases database, The
continuing 2019-nCoV epidemic threat of novel coronaviruses to
global health — The latest 2019 novel coronavirus outbreak in Wuhan,
China.
Typically not included in most mainstream news stories, however, is

the fact that the claimed epicenter of the outbreak is just 8.6 miles
from Wuhan Institute of Virology, which houses China’s only P4-Level
Biosafety Laboratory capable of storing, studying, or engineering
Pathogen Level 4 microbes such as other coronaviruses, Ebola, Severe
Acute Respiratory Syndrome, SARS, H5N1 influenza virus, Japanese
encephalitis, and dengue. Bill Gurtz of the Washington Times reports,
“the deadly animal virus epidemic spreading globally may have
originated in a Wuhan laboratory linked to China’s covert biological
weapons program, according to an Israeli biological warfare expert.”
The journalist states that an unnamed U.S. official revealed that false
rumors have been circulating for weeks on the Chinese Internet
claiming the new coronavirus is “part of a U.S. conspiracy to spread
germ weapons” — possibly preparing propaganda outlets to counter
future charges the new virus escaped from one of Wuhan’s civilian or
defense research laboratories.
The article refers to statements provided by Dany Shoham, a former

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Israeli military intelligence officer who holds a doctorate in medical
microbiology, and served as a senior analyst with Israeli military
intelligence for biological and chemical warfare in the Middle East and
worldwide from 1970 to 1991.“Coronaviruses (particularly SARS) have
been studied in the Institute and are probably held therein”, Shohan
reveals, as has anthrax, adding that “certain laboratories in the
Institute have probably been engaged, in terms of research and
development, in Chinese [biological weapons]. Work on biological
weapons is conducted as part of a dual civilian-military research and is
“definitely covert.” Troublingly, even a State Department report issued
last year raised suspicions that China has been engaged in covert
biological warfare work. “Information indicates that the People’s
Republic of China engaged during the reporting period in biological
activities with potential dual-use applications, which raises concerns
regarding its compliance with the BWC,” the report said, adding that
the United States suspects China failed to eliminate its biological
warfare program as required by the treaty.
Thus, it seems rather astute to examine the details of government-

and media-disseminated reports in contrast to the background of
activity conducted at Wuhan Institute of Virology, as well as look into
the specifics of the new coronavirus in comparison with viruses
already isolated, identified, stored, studied, and/or engineered at the
Institute’s Biosafety Laboratory, in an effort to glean the truth.
Claims of surprise by Chinese scientists and State officials are

arguably inauthentic
Let’s begin by examining the glaring discrepancies in the official story

to the underlying and background reality of coronaviruses, especially

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in the SARS-scarred land of China. The Sun reports that the current
consensus centers on the belief that the origin of the coronavirus
outbreak is linked to bat soup sold at the market. However, the article
states that experts “had thought the new virus wasn’t capable of
causing an epidemic as serious as [previous deadly outbreaks of SARS
and Ebola] because its genes were different,” something that simply
isn’t true. In 2006, one of China’s preeminent virologists, Professor
Zhengli Shi, co-authored the study, Review of Bats and SARS,
concluding that “a SARS epidemic may recur in the future and that
SARS-like coronaviruses (SL-CoVs) that originate from different
reservoir host populations may lead to epidemics at different times or
in different regions…. The recent discovery of a group of diverse SL-
CoVs in bats support the possibility of these events….”

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Bowl of hot, delicious bat soup served at Huanan Seafood Wholesale Market in Wuhan, China.
A concurrent article published in the South China Morning Post on

January 22, 2020, entitled Coronavirus weaker than SARS but may
share link to bats, Chinese scientists say reports the latest findings on
the coronavirus by scientists at China’s Center for Disease Control and

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Prevention. “The scientists’ findings, published on Tuesday, suggested
that the danger posed by the pneumonia-like virus may have been
underestimated by the research community.” However, Prof. Zhengli
and her co-authors published a study early last year on March 2, 2019
entitled Bat Coronaviruses in China which explicitly warned,
“During the past two decades, three zoonotic coronaviruses have

been identified as the cause of large-scale disease
outbreaks⁻Severe Acute Respiratory Syndrome (SARS), Middle East
Respiratory Syndrome (MERS), and Swine Acute Diarrhea
Syndrome (SADS). SARS and MERS emerged in 2003 and 2012,
respectively, and caused a worldwide pandemic that claimed
thousands of human lives, while SADS struck the swine industry in
2017. They have common characteristics, such as they are all
highly pathogenic to humans or livestock, their agents originated
from bats, and two of them originated in China. Thus, it is highly
likely that future SARS- or MERS-like coronavirus outbreaks
will originate from bats, and there is an increased probability
that this will occur in China. Therefore, the investigation of bat
coronaviruses becomes an urgent issue for the detection of early
warning signs, which in turn minimizes the impact of such future
outbreaks in China” (emphasis added).
The South China Morning Post article continues with the beguiling
assertion, “Previously, most scientists believed the new virus could not
cause an epidemic as serious as that of SARS because its genes were
quite different. But the new study found that, like SARS, the virus
targeted a protein called angiotensin-converting enzyme 2 (ACE2).”
Apparently, the virology scientific community not only failed to heed
Prof. Zhengli’s explicit, recent dire warnings about the “high likelihood”

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that future SARS- or MERS-like coronavirus outbreaks would originate
from bats — they also ignored Zhengli’s incredibly pertinent report
published ten years ago in July, 2010, Identification of key amino acid
residues required for horseshoe bat angiotensin-I converting enzyme
2 to function as a receptor for severe acute respiratory syndrome
coronavirus. The study’s abstract can’t be clearer on the
immunological risks associated with protein ACE2, with its obvious
liability for usurpation by viral agents with a little modified genome
sequencing:
“Angiotensin-I converting enzyme 2 (ACE2) is the receptor for

severe acute respiratory syndrome (SARS) coronavirus (SARS-
CoV). A previous study indicated that ACE2 from a horseshoe bat,
the host of a highly related SARS-like coronavirus, could not
function as a receptor for SARS-CoV. Here, we demonstrate that a
3 aa change from SHE (aa 40–42) to FYQ was sufficient to convert
the bat ACE2 into a fully functional receptor for SARS-CoV. We
further demonstrate that an ACE2 molecule from a fruit bat, which
contains the FYQ motif, was able to support SARS-CoV infection,
indicating a potentially much wider host range for SARS-CoV-
related viruses among different bat populations.”
This old but remarkable study concludes that only a minor genome
sequence change was required to convert a non-susceptible bat ACE2
protein into a functional receptor for SARS-CoV, something that could
easily happen in nature. “Considering that there are more than 60
different horseshoe [bat] species around the world (Flanders et al.,
2009; Rossiter et al., 2007), it is possible that one or some of them
may serve as the natural reservoir of SARS-CoV and/or its progenitor
virus(es).” Why is it that current State virologists are apparently

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ignorant of these essential discoveries of yesteryear?
The South China Morning Post article cited above summarizes two
primary known facts about the new coronavirus: first, that a “virus
found in fruit bats is [the] common ancestor of the two strains
[Coronavirus 2019-nCoV and SARS],” and that this “new strain has
[an] unusually high ability to bind to a human protein.” And the new
study on Coronavirus 2019-nCoV by the joint research team from the
Chinese Academy of Sciences, the People’s Liberation Army, and
Institut Pasteur of Shanghai indeed found that, like SARS, the virus
targeted the ACE2 protein. It’s just as Prof. Zhengli predicated a
decade ago: “…the fact that an ACE2 protein from a megabat, the fruit
bat Rousettus leschenaultia, can function as a receptor for SARS-CoV
would suggest that the host range for SARS-CoV or SL-CoVs may be
much wider than originally thought.”
So what happened — did the virology and surrounding scientific

community drop the ball on these well-established findings and
warnings, or what? After all, at least as February, 2008, they knew
three key facts about ACE2:
1. Severe acute respiratory syndrome (SARS) is caused by the

SARS-associated coronavirus (SARS-CoV), which uses ACE2 as
its receptor for cell entry. SL-CoVs and SARS-CoVs share identical
genome organizations and high sequence identities, with the main
exception of the N terminus of the spike protein, known to be
responsible for receptor binding in CoVs.
2. Whereas the SL-CoV spike protein was unable to use any of the
three ACE2 molecules as its receptor, and the SARS-CoV spike
protein failed to center cells expressing the bat ACE2, the

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chimeric spike protein the study created did gain its ability to
center cells via human ACE, and
3. A minimal insert region (amino acids 310 to 518) was found to be
sufficient to convert the SL-CoV S from non-ACE2 binding to
human ACE2 binding, indicating that the SL-CoV S is largely
compatible with SARS-CoV S protein both in structure and in
function.
We know they knew these facts way back in 2008 because Prof.
Zhengli published the findings of these facts in her report, Difference
in Receptor Usage between Severe Acute Respiratory Syndrome
(SARS) Coronavirus and SARS-Like Coronavirus of Bat Origin. Therein
the scientists concluded, “Knowing the capability of different CoVs to
recombine both in the laboratory and in nature, the possibility that SL-
CoVs may gain the ability to infect human cells by acquiring spike
protein sequences competent for binding to ACE2 or other surface
proteins of human cells can be readily envisaged.” Thus, it seems
strange and perhaps even disingenuous that the new joint CCP
government-joint Coronavirus 2019-nCoV task force is seemingly
ignorant about coronavirus targeting the ACE2 protein, apparently
pretending it’s only just now discovered this. After all, Zhengli’s 2008
report was quite clear about the role that this ACE2 protein would play
in future pandemics: the study “strengthened our belief that ACE2
from certain bat species could be able to support SARS-CoV infection
because of the predicted genetic diversity of bat ACE2 variants in
different bat species.”
What is the Wuhan Institute of Virology’s National Biosafety

Laboratory, where is it, and why is it pertinent?

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Wuhan National Biosafety Laboratory, the only P4 lab in China, headquartered at Wuhan Institute of
Virology.
At any rate, the forgoing storyline is the official word on Coronavirus

2019-nCoV, manifesting itself somehow in a seafood market in Wuhan.
But what else might be found in Wuhan? After all, Wuhan is the capital
city of the Hubei Province, home to some 11 million Chinese citizens.
Well, curiously underreported is the fact that China’s first high-level
biosafety laboratory is located just 8.6 miles away. “Used to study
class four pathogens (P4), which refer to the most virulent viruses that
pose a high risk of aerosol-transmitted person-to-person infections,”
Wuhan National Biosafety Laboratory is the darling, cutting-edge hi-
tech baby of the Wuhan Institute of Virology, Chinese Academy of
Sciences, and is the only such lab in China where dangerous, highly
communicable viruses such as Ebola, SARS, MERS, H5N1 influenza
virus, Japanese encephalitis, dengue, and assorted coronaviruses can
be “safely” toyed with.

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China’s National Biosafety Laboratory, located at Wuhan Institute of Virology, is only 8.6 miles away from
the claimed epicenter of the Coronavirus 2019-nCoV outbreak. Do you believe in coincidences?
What’s odd is that despite completing the decade-long construction

and having the official inauguration of this P4 laboratory on January
31, 2015 — announced by the General Office of Hubei Provincial
People’s Government, it wasn’t until 2 and 1/2 years later in January
2018, that the Chinese government announced that the lab was
actually in operation. And ahead of the lab’s second opening in
January 2018, biosafety experts and scientists from the United States
expressly warned “that a SARS-like virus could escape,” much in the
same way the SARS virus had escaped multiple times from a lab in
Beijing.
UPDATE — JANUARY 29, 2020: What’s also odd, and outright

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suspicious, is that as of January 29, 2020, the location of Wuhan
Institute of Virology (where the National Biosafety Laboratory is
headquartered) on Google Maps has inexplicably moved since I first
viewed it on January 24, 2020 and published this article on January
27, 2020. Its new location is now over twice the distance from the
claimed epicenter of the novel coronavirus, Huanan Seafood
Wholesale Market. Even its satellite imagery of the original site has
been altered as well. Good thing I took screenshots.
A Google Map Image captured Jan. 24, 2020 of Huanan Seafood market 8.6 miles distant from Wuhan
Institute of Virology, where China’s only Level P4 Biosafety Laboratory is headquartered.

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Another Google Map Image captured Jan. 29, 2020 displaying Wuhan Institute of Virology now strangely
moved approximately 15 miles southwest of its original location. What a difference five days make, eh?
Side-by-side comparison of original Google Maps location of Wuhan Institute of Virology, captured by
screenshot on Jan. 24, 2020, and its altered location as of Jan. 29, 2020. What’s going on here?

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A Google Maps Satellite Image captured January 24, 2020, clearly showing the urban Wuhan Institute of
Virology situated across the street from the humongous China Earthquake Administration building.
Another Google Maps Satellite Image captured January 29, 2020, now showing the Wuhan Institute of
Virology completely camouflaged in a patch of forest in a rural area 15 miles southwest. What gives,

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Google?
Whether Wuhan Institute of Virology actually remains at its original

location displayed a few days ago — or has suddenly packed up and is
now holed up in the nearby woods like a crouching tiger or hidden
dragon — former Israeli military intelligence officer and microbiologist,
Dany Shoham, exposes the institute as “one of four Chinese
laboratories engaged in some aspects of the biological weapons
development.” He adds that although the institute is under the
Chinese Academy of Sciences, it has certain laboratories within it that
are linked to the Chinese defense establishment. Indeed, the annual
State Department report on arms treaty compliance stated last year
that China engaged in activities that could support biological warfare.
In fact, in 1993, China declared a second facility, the Wuhan Institute
of Biological Products — located 21.6 miles away from Wuhan Institute
of Virology, and only 9 miles away from Huanan Seafood Wholesale
Market — as one of eight biological warfare research facilities covered
by the Biological Weapons Convention (BWC) which the communist
country joined in 1985. “This means the SARS virus is held and
propagated there, but it is not a new coronavirus, unless the wild type
has been modified, which is not known and cannot be speculated at
the moment,” Shoham explains.

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Wuhan Institute of Biological Products — a known biological warfare research facility — is located just 9
miles from Wuhan Seafood Wholesale Market, the claimed epicenter of the Coronavirus 2019-nCoV
outbreak
Wuhan Institute of Virology is also connected with the recent, major

scandal in Canada where two Chinese virologists working at Canada’s
only Pathogen Level 4 virology laboratory, the National Microbiology
Lab (NML) in Winnipeg were caught stealing and smuggling some of
the most deadly viruses on earth, including the Ebola virus, back to
China. The suspects — a Chinese couple, virologist Dr. Xiangguo Qui
and biologist Dr. Keding Cheng — are now believed to be connected to
China’s biological warfare program. Her husband, Dr. Qiu, was head of
the Vaccine Development and Antiviral Therapies Section in the
Special Pathogens Program at the NML. Dr. Keding Cheng, also
affiliated with the NML, specifically the “Science and Technology
Core,” is primarily a bacteriologist who shifted to virology. According to
ZeroHedge, “the couple is responsible for infiltrating Canada’s
NML with many Chinese agents as students from a range of
Chinese scientific facilities directly tied to China’s Biological
Warfare Program, including the Wuhan Institute of Virology and

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the Center for Disease Control and Prevention in Chengdu Military
Region.
And guess what one of the stolen viruses was? Yup, coronavirus. On
May 4, 2013, NML’s Scientific Director Frank Plummer received a
shipment of coronavirus from a Dutch virologist, who in turn had
received it from an Egyptian virologist treating a Saudi Arabian who
contracted it. The Canadian lab grew stocks of the virus, and then
experimented upon animals to see what they could infect with it. It is
from this stash of reserves that the coronavirus was stolen and
smuggled by Dr. Qui, Dr. Cheng, and by alledged Chinese Biological
Warfare Program agents recruited from the Wuhan Institute of Virology
who were disguised as virology students at the University of Manitoba.
Similarly, and perhaps connected, is the recent indictment of Charles

Lieber, Chair of Harvard University’s Department of Chemistry and
Chemical Biology. Prosecutors claim he had a contract with Wuhan
Institute of Virology. Reports CBS, “It appears China paid Lieber
hundreds of thousands of dollars over the years for his involvement
with the Chinese entities and for his work on research for Chinese
gain,” said U.S. Attorney for Massachusetts Andrew Lelling.” Lieber lied
about his links to Wuhan Institute of Virology, report Tonya Alanez and
Travis Andersen of the Boston Globe. “Federal authorities said Charles
Lieber, a prominent nanoscientist and a prolific inventor and
entrepreneur, received hundreds of thousands of dollars from his
Chinese connections.” Details about the extent of Lieber’s illicit and
illegal conspiracy with the institute have yet to emerge. So this means,
significantly, that not only are there Chinese nationals allegedly being
recruited from Wuhan Institute of Virology to penetrate foreign P4
biosafety laboratories abroad and smuggle the spoils back home, but

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it also appears Americans and Canadians may be complicit in aiding
the Chinese biowarfare program.
In short, although there are actually two laboratories in Wuhan linked

to the Chinese biowarfare program — only one is certified for
coronaviruses and only one is caught in the midst of all the recent
international espionage intrigue — the new Pathogen Level 4-rated
National Biosafety Laboratory at Wuhan Institute of Virology. And
whether this enigmatic facility is a philanthropic, health services-
related institute, a covert, biological warfare research installation, or
some combination of the two — remains to be officially disclosed. So
what on earth could the scientists sequestered at Wuhan National
Biosafety Laboratory have been up to in their brand new, state-of-the-
art biotech base for two and a half years, if it wasn’t officially in
operation? And what have they been doing since their second opening
in 2018?
Scientists at Wuhan National Biosafety Laboratory research coronaviruses, Ebola, and other deadly
pathogens.

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Well, storing, researching, and experimenting with numerous fulminant
disease pathogens, of course. After all, the lab is “preservation center
for virus seeds, a fulminant disease pathogen storage facility, a
reference laboratory of WHO, a node for disease network, and finally…
a core in China’s emerging disease research network.” Basically, in all
of China, Wuhan National Biosafety Laboratory is the only place to
store and experiment with the most lethal, most virulent, most rapidly-
spreading disease pathogens known to humanity. The lab is in “the
central region of Central China, with mountains at three directions,
convenient transportation and relatively independent environment”
[sic]. And convenient it is, as you can play with Ebola, SARS,
Hantavirus, and assorted coronaviruses in the morning…and then hop
in your car and have some bat soup for lunch at the Huanan Seafood
Wholesale Market on the other side of the Yangtze River. Maybe BYOB
— bring your own bat?
Once Wuhan Institute of Virology formally put their brand new Cellular
Level Biosafety Level 4 Laboratory into operation, we can safely take
their word that they followed up on their promise to “conduct research
for natural focal viruses including Ebola virus and other emerging
viruses, such as researches [sic] on rapid detection system, molecular
epidemiology, infectious disease etiology, therapeutic antibody,
vaccine and drug evaluation, and assessment on biological risk
factors, thus building a biosafety platform in China for emerging and
fulminant infectious diseases in terms of isolation and identification of
pathogen, building of infection models, vaccine development,
biological containment and research on mechanism of interaction
between pathogen and the host.” And one thing we know they worked
on is the Origin and evolution of pathogenic coronaviruses, pioneered

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by none other than the enormously qualified, highly-decorated, and
widely-respected Professor Zhengli Shi, Senior Scientist and Principal
Investigator.
Who is Professor Zhengli Shi and what is her relevance to Wuhan

Institute of Virology and the National Biosafety Laboratory?
Professor Zhengli Shi, Senior Scientist and Principal Investigator of Wuhan National Biosafety Laboratory.

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Do you believe in coincidences? Because it just so happens that Prof.
Zhengli has been ardently researching and experimenting with
coronaviruses for years at Wuhan Institute of Virology — even before
ground was broken over a decade ago on the new P4 National
Biosafety Laboratory. Interestingly, the scientist seems uniquely
perfect for her role — like a “Neo” figure in a laboratory version of The
Matrix. In fact, Prof. Zhengli has been Senior Scientist and Principal
Investigator of Wuhan Insititute of Virology for the last 20 years,
initially starting as a Research Assistant in 1990 before upgrading to
Research Scientist in 1993, serving in that role until 1995. Aside from a
5-year leave from 1995 to 2000 to get her PhD at University of
Montpellier in France, she’s been at the Institute for an amazing 30
years.
Notably, starting in 2014, Prof. Zhengli began to win particularly large

sums of grant funding for the express purpose of researching and
experimenting with coronaviruses — often receiving numerous,
overlapping grants for the same time period. What’s just as interesting
is where a lot of this funding originated — the US government. On
January 6, 2014, Prof. Zhengli received a US$665,000 grant from the
National Institute of Health for a study named The Ecology of Bat
Coronaviruses and the Risk of Future Coronavirus Emergence (NIAID
R01 AI1 10964) and then four days later on January 10, 2014, an
additional US$559,500 grant from the United States Agency of
International Development for research studied entitled Emerging
Pandemic Threats PREDICT 2_China (Project No. AID-OAA-A-14–
00102).
On top of these lucrative American grants she concurrently received

similarly significant grants from the National Basic Research program

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of China, the Chinese Academy of Science, the National Natural
Science Foundation of China, and from the Strategic Priority Research
Program of Chinese Academy of Sciences totaling over US$2,500,000
for researching interspecies transmission of zoonotic viruses, the
identification, genetic evolution and pathogenesis of bat viruses, the
genetic variation of pathogens in Africa, the evolution mechanism of
the adaptation of bat SARS-related coronaviruses to host receptor
molecules, the risk of interspecies infection, genetic evolution and
transmission mechanism of important bat-borne viruses, and
pathogen biology studies on novel swine coronaviruses.
In just the past five years alone, Prof. Zhengli Shi has almost US$10 million in grants to study
coronaviruses.

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We can quite safely conclude that when it comes to interspecies
coronaviruses, Professor Zhengli Shi is a bona fide Jedi master. In fact,
her Wikipedia page credits her and her colleague, Cui Jie, with the
actual discovery that the SARS virus originated in bats. Her noted
“Research Interests” on her C.V. include “Discovery of unknown
viruses in wild animals especially bats, molecular epidemiology of
emerging zoonotic viruses, and interspecies infection mechanism of
zoonotic viruses.” Prof. Zhengli appears to be one of the world’s
leading bat virologists — and most definitely the leading bat virologist
in China. Indeed, her C.V. explicitly states,
“Prof. Zhengli Shi ’s researches focus on the molecular

epidemiology and interspecies infection discovery and
characterization of novel viruses in bats and other wildlife. She has
gain [sic] rich expertise on pathogen biology of coronaviruses and
other emerging viruses of bat origin, virus discovery, virus
evolution, and development of diagnostic technologies for
emerging viruses. Prof Shi has identified ultimately the animal
origin of SARS, by discovering genetically diverse bat SARS related
coronaviruses (SARSr CoV), isolating bat SARSr CoVs highly
homologous to SARS CoV that are able to the same receptor [sic]
as SARS CoV, and revealing the potential recombination origin of
SARS CoV. She has discovered a large number of novel viruses
from Chinese bat populations, including viruses with potential
public health significance.”
Unsurprisingly, Prof. Zhengli has been featured as a key presenter at

over two dozen international virology conferences, the latest being
From SARS to SADS: predict of emerging infectious diseases, held at
UC Berkeley in the summer of 2018. Her presentations at the next five

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most recent conferences all relate specifically to the genetic evolution
and interspecies infection of bat coronaviruses. A complete list of Prof.
Zhengli’s conference presentations may be found in Appendix B.
Nearly all of Prof. Zhengli’s recent conference presentations relate to bat coronaviruses. Do you believe in
coincidences?
Prof. Zhengli has been or is currently a professional member of the

Chinese Society for Biochemistry and Molecular Biology (2000–2016),
the Chinese Society for Microbiology (2002-present), the American
Society for Microbiology (2007-present), and the Scientific Committee
of the DIVERSITAS ecoHEALTH Core Project (2014–2016). She has
served on the Editorial Board of Virologica Sinica (2016–2016), on the
Editorial Board of Journal of Medical Virology (2015–2017), and on the
Editorial Board of Virology (2017–2019). She was Associate Editor of
Virology Journal (2016–2018), and Editor-in-Chief of Virologica Sinica
(2017–2019). Prof. Zhengli is also the recipient of numerous,
prestigious awards and honors, including the Natural Science Award of

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Hubei Province, China (First Prize and Second Prize), Outstanding
Scientist of the Chinese Academy of Sciences, and Outstanding
Research Article on Natural Science (Grand Prize and Second Prize).
OK, but how is Prof. Zhengli relevant to the current new outbreak
of Coronavirus 2019-nCoV?
Coronavirus 2019-nCoV outbreak in Wuhan, China — where the National Biosafety Laboratory is located —
causes a massive quarantine of 11 million citizens.
Chinese scientists, researchers, and doctors examining the emergent

2019-nCoV Coronavirus report that the new viral menace appears to
be “a recombinant virus between the bat coronavirus and an origin-
unknown coronavirus. The recombination occurred within the viral
spike glycoprotein, which recognizes cell surface receptor.” But Prof.
Zhengli appears to have worked with recombinant Coronavirus
derivations involving viral spike proteins for over a decade at Wuhan
Institute of Virology, all the way back to 2006 and up to as recently as
December, 2019 — the very month that 2019-nCoV Coronavirus was

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first reported as having infected visitors at Huanan Seafood Wholesale
Market just down the road from her laboratory!
The day before the Coronavirus 2019-nCoV outbreak, this report was published. Do you believe in
coincidences?
In fact, on the day before the new coronavirus would find its first
victims just 8.6 miles away at the market on December 12, 2019, Prof.
Zhengli and her team published the study entitled Molecular
mechanism for antibody-dependent enhancement of coronavirus
entry on December 11, 2019. The abstract reads,
“Coronavirus spike protein mediates viral entry into cells by first

binding to a receptor on host cell surface and then fusing viral and

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host membranes. Our study reveals a novel molecular mechanism
for antibody-enhanced viral entry and can guide future vaccination
and antiviral strategies. This study reveals complex roles of
antibodies in viral entry and can guide future vaccine design and
antibody-based drug therapy.”
And immediately after this study was published — literally the

following day — the first victims became infected with what would
soon be named Coronavirus 2019-nCoV began to get infected…just a
few miles away from Prof. Zhengli’s laboratory. And as The Sun
reports, victims of the new coronavirus are infected via a strong
binding affinity to a human protein called ACE2,” in precisely the
identical manner as Prof. Zhengli’s just-discovered “novel molecular
mechanism” identified (or engineered) literally weeks if not days
before. Do you believe in coincidences?
Let’s say that’s just a coincidence Prof. Zhengli published a study

or two specifically on bat coronaviruses. Have there been others?
How much time you got? The above study, specifically relating to
human host cell binding and entry of coronavirus infection, and
published the day before the first viral infections were reported at a
location adjacent Prof. Zhengli’s laboratory, is far from the only study
in which she has directed on the subject. The scientist’s entire virology
history is rife with hands-on experience with coronaviruses, with
especial attention devoted to understanding their spike protein
properties, as related to potentiality of human cell entry and infection.
In June 2016’s study, Bat Severe Acute Respiratory Syndrome-Like
Coronavirus WIV1 Encodes an Extra Accessory Protein, ORFX, Involved
in Modulation of the Host Immune Response she writes that what was

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important was that bats “harbor genetically diverse SARS-like
coronaviruses (SL-CoVs), and some of them have the potential for
interspecies transmission.” She further states that her team created a
“reverse genetics system” that would be helpful for “study of the
pathogenesis of this group of viruses and to develop therapeutics for
future control of emerging SARS-like infections.”
In a letter to the editor of SCIENCE CHINA Life Sciences published in

November, 2017, entitled Cross-neutralization of SARS coronavirus-
specific antibodies against bat SARS-like coronaviruses, Prof. Zhengli
warns that severe acute respiratory syndrome coronavirus (SARS-
CoV) is considered to be an emerging zoonotic pathogen crossing
species barriers to infect humans, and that the spike protein of the
virus’ RNA genome plays a key role in human cellular entry.
In that same month, the results of a study Prof. Zhengli conducted,

Serological evidence of bat SARS-related coronavirus infection in
humans, China indicated that some SARSr-CoVs may have high
potential to infect human cells, without the necessity for an
intermediate host.
In 2016, one of the Directors at Wuhan Institute of Virology posted the

annual Director’s Message, of which the following finding was the top
announcement: “The live SARS-like coronavirus SL-CoV-WIV1 has
been isolated for the first time from the bat droppings; and such virus
has been confirmed to invade the host cells through the ACE2 of
human beings, civets and Rhinolophus sinicus. The research result has
so far provided the most convincing evidence to the view that
Rhinolophus sinicus is the natural host of SARS-CoV (Nature, 2013).”
Does this not sound precisely like Coronavirus 2019-nCoV, which

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invades the host cells through the ACE2 protein? At any rate, since
Prof. Zhengli is Senior Scientist and Principal Investigator of both the
Emerging Viruses Group and the National Biosafety Laboratory, this is
squarely her turf; the current outbreak seems amazingly similar.
In a study conducted in September of 2015, Two Mutations Were

Critical for Bat-to-Human Transmission of Middle East Respiratory
Syndrome Coronavirus, Prof. Zhengli and team successfully achieved
viral entry (bat-to-human transmission)of bat coronavirus HKU4 via its
spike protein by performing two small mutations. Doing so also helped
explain how MERS coronavirus was able to infect humans as well.
It was in 2015’s study, Isolation and Characterization of a Novel Bat

Coronavirus Closely Related to the Direct Progenitor of Severe Acute
Respiratory Syndrome Coronavirus that Prof. Zhengli and team
highlighted “the likelihood of future bat coronavirus emergence in
humans” by isolating a new bat coronavirus closer to SARS-CoV in
genomic sequence, particularly in its spike gene. “Cell entry and
susceptibility studies indicated that this virus can…infect animal and
human cell lines,” they concluded.
And in 2010’s Angiotensin-converting enzyme 2 (ACE2) proteins of

different bat species confer variable susceptibility to SARS-CoV entry
Prof Zhengli and her team of scientists “extended [their] previous
study to ACE2 molecules from seven additional bat species and tested
their interactions with human SARS-CoV spike protein using both HIV-
based pseudotype and live SARS-CoV infection assays.”
Even earlier in 2010, Prof. Zhengli published, Bat and virus, a keystone
study identifying bats “as a natural reservoir of emerging and

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reemerging infectious pathogens,” emphasizing that an astonishing
amount (more than 70, at the time) and genetic diversity of viruses
isolated from the bat have been identified in different populations
throughout the world. She stresses that many viruses were found in
apparently healthy bats, suggesting that bats may have a particularly
robust immune system or “antiviral activity against virus infections.”
In 2009’s Immunogenicity difference between the SARS coronavirus

and the bat SARS-like coronavirus spike (S) proteins, Prof. Zhengli and
her team concluded “SARS-like coronavirus (SL-CoV) in bats have a
similar genomic organization to the human SARS-CoV.” And notably,
that this work “provides useful information for future development of
differential serologic diagnosis and vaccines for coronaviruses with
different S [spike] protein sequences.”
Prof. Zhengli’s research in 2009’s Differential stepwise evolution of

SARS coronavirus functional proteins in different host species
produced results that supported the hypothesis that “SARS-CoV
originated from bats and that the spill over into civets and humans
were more recent events.”
Moving even further back in time to 2007, Prof. Zhengli worked on

Determination and application of immunodominant regions of SARS
coronavirus spike and nucleocapsid proteins recognized by sera from
different animal species, producing assays that would be a “useful tool
to trace the origin and transmission of SARS-CoV and to minimise the
risk of animal-to-human transmission.”
It appears that 2006 was the year Prof. Zhengli first researched
recombinant spike proteins along with other distinctive genome

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sequences resulting from the interaction of bat, palm civet, and human
isolates. “Full-length genome sequences of two SARS-like
coronaviruses in horseshoe bats and genetic variation analysis.”
Basically, she is tremendously versatile and adept in her research
whenever she encounters these recombinant spikes proteins in viral
interactions.
Moreover, it’s not just coronaviruses from bats that she and her team
have discovered and explored, but also diverse novel viruses/virus
antibodies in bats, including adenoviruses, adeno-associated viruses,
circoviruses, paramyxoviruses, and filoviruses. In fact, Prof. Zhengli
has coauthored over an astounding 130 publications on viral pathogen
identification, diagnosis and epidemiology — nearly all of which
commandeered at Wuhan Institute of Virology where the National
Biosafety Laboratory is located and where she reigns as Head of the
Department. In fact, on the World Society for Virology website, Prof.
Zhengli’s profile confirms that one of her great contributions was to
“uncover genetically diverse SARS-like coronaviruses in bats with her
international collaborators and provide unequivocal evidence that bats
are natural reservoirs of SARS-CoV.” Thus, her adeptness in the
specialized field of bat virology — especially where transmission to
humans is concerned — is inarguable.
Such an expansive personal history of expertise into coronaviruses is

not only impressive, but unique, and the bulk of her 30-year career at
Wuhan Institute Virology seems to have been dedicated primarily to
the examination and exploration of all facets of interspecies (though
primarily bat) pathogenic infection of coronaviruses into human host
cells. For reference, you can check Appendix A for the sum total of all
her published (or otherwise unclassified or declassified) studies at the

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end of this essay. Prof. Zhengli’s absolute mastery of bat-to-human
transmission of viruses via their spike protein binding with human cell
receptors is virtually conclusive and unrivalled.
Unanswered Questions About the Coronavirus 2019-nCoV

Outbreak in Wuhan
In Prof. Zhengli’s March 2019 study, Bat Coronaviruses in China, she

proves seemingly prophetic, writing that it was “highly likely that
future SARS- or MERS-like coronavirus outbreaks will originate from
bats, and there is an increased probability that this will occur in China.
Therefore, the investigation of bat coronaviruses becomes an urgent
issue for the detection of early warning signs, which in turn minimizes
the impact of such future outbreaks in China.” Just nine months later,
2019-nCoV rears its viral head, less than 10 miles from her labatory:
how did Prof. Zhengli know?
The Sun cited a Nature.com report voicing warnings given back in

2017 “that a deadly SARS-like virus could escape from lab [sic] in
Wuhan set up to study some of the world’s deadliest diseases.” The
worries surrounding Wuhan’s laboratory surfaced almost an entire
year before the Chinese government announced its official
commencement of operation in January, 2018. And likely with good
cause, as the “SARS virus [had] escaped from high-level containment
facilities in Beijing multiple times, notes Richard Ebright, a molecular
biologist at Rutgers University in Piscataway, New Jersey.” However,
the article in The Sun exaggerates the distance from Wuhan’s National
Biosafety Laboratory to Huanan Market, erroneously claiming that it’s
20 miles away, instead of 8.6 miles, and also states that Dr. Ebright
reportedly said “at this point there’s no reason to harbor suspicious

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that the facility had anything to do with the outbreak.” Seriously? Does
Dr. Ebright believe in coincidences?
Another new article from The Sun published January 23, 2020, reports
a “new study was carried out jointly by the Chinese Academy of
Sciences, the People’s Liberation Army and Institut Pasteur of
Shanghai, revealing that the coronavirus has a strong binding affinity
to a human protein called ACE2.” But Zhengli and her team mates have
been aware of the susceptibility of ACE2 to SARS and coronavirus
infection for at least the last ten years, publishing their studies with the
US National Library of Medicine and with other prominent industry
repositories.
So we are left with the following pressing, unanswered questions

about Prof. Zhengli, the Wuhan National Biosafety Laboratory, and the
Coronavirus 2019-nCoV outbreak in Wuhan:
1. Why are the Chinese authorities seemingly ignoring the Wuhan

Institute Virology’s contemporaneous coronavirus study
(culminating in a Dec. 11, 2019 report, published the day before
the outbreak) conducted at the Wuhan National Biosafety
Laboratory, located just 8.6 miles distant from the claimed
epicenter of pandemic origin, Huanan Seafood Wholesale Market?
Why is the media not reporting this?
2. Why are most media reports covering the coronavirus still
misreporting the source of the virus’ genome sequence as snakes
instead of bats?
3. Since the Wuhan Institute of Virology has already isolated live,
novel SARS-like Coronavirus SL-CoV-WIV1 from bat droppings in
2016, and such virus has been confirmed to invade the host cells

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through the ACE2 of human beings just like the new, emergent
Coronavirus 2019-nCoV — have the two coronaviruses been
compared with each other? Was there a vaccine developed from
Coronavirus SL-CoV-WIV1 that can be tested on victims of the
latest outbreak? After all, it’s been about four years now.
4. Has any formal investigation been launched into any role the
Wuhan Institute of Virology (and specifically, its Classification P4
Biosafety Laboratory) may have played in the pandemic outbreak?
5. Did the new coronavirus penetrate the biosecurity measures of
Wuhan National Biosafety Laboratory? Did some bats mount a
successful escape?
6. Did any scientists, researchers, professors, observers, students,
or other staff persons working at or visiting the Wuhan National
Biosafety Laboratory visit the Huanan Seafood Market in the first
twelve days of December, 2019?
7. Since the original technology for viral confinement at the Wuhan
National Biosafety Laboratory was developed in France, and since
most of its actual, functional equipment was imported from
France — has the laboratory received ongoing certification
inspections from French officials, given its lengthy, ongoing
activities using Class 4 pathogens (P4) — the most virulent
viruses that pose the highest risk of aerosol-transmitted person-
to-person infections? If so, where are the certification test
results?
8. Has the Wuhan National Biosafety Laboratory been regularly
inspected and audited by Chinese government health officials,
especially by Li Bin, minister of the National Health and Family
Planning Commission? If so, where are the inspection and audit
results?

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9. Could there have been either a staff person or visitor who
smuggled out the coronavirus from the laboratory? (After all, a
Chinese national was just arrested at Harvard University for
attempting to smuggle research vials back to China at the same
time when the Coronavirus 2019-nCoV outbreak started.)
10. At any time did Prof. Zhengli Shi — who simultaneously currently
holds the multiple titles of Senior Scientist and Principal
Investigator, Director of the Center for Emerging Infectious
Diseases, Director of BSL-3 Labatory, Director of the Committee
of Biosafety, Director of Chinese Academy Sciences (CAS) Key
Laboratory of Special Pathogens and Biosafety, and Vice Director
of BSL-4 Laboratory at Wuhan Institute of Virology, CAS — ever
work directly or indirectly for the CCP military services or military
intelligence community?
11. Did Prof. Zhengli previously or does she currently co-conduct,
coparticipate, collaborate, or collude with CCP military service
members or military intelligence members?
12. Do members of the CCP military services or military intelligence
contribute or participate in any manner or conduct viral research
at the Wuhan National Biosafety Laboratory?
13. Did Prof. Zhengli or any other faculty member at the Wuhan
Institute of Virology take possession, either illicitly or officially, of
any biological substance, whether pathogen, vaccine, or other
biomatter, originating from the United States or Canada?
14. Why did the US National Institute of Health (NIH) grant Prof.
Zhengli $665,000 in 2014 to fund her study, The ecology of bat
coronaviruses and the risk of future coronavirus emergence?
What did the US receive in return?
15. Why did the United States Agency of International Development

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grant Prof. Zhengli $559,500 to fund her study, Emerging
Pandemic Threats PREDICT 2_China? What did the US receive in
return?
16. Why did Prof. Zhengli receive funding from U.S. Department of
Defense, the U.S. Defense Threat Reduction Agency (the agency
which deals specifically with Weapons of Mass Destruction), the
U.S. Biological Defense Research Directorate of the Naval Medical
Research Center, and the Department of Atomic of the
Government of India?
17. What other professional relationships with U.S. defense agencies
does Prof. Zhengli have currently, or previously, in any capacity?
18. When Prof. Zhengli received a visa to the United States to present
at the Cell Symposium: Emerging and Re-emerging Viruses 2017
conference in Arlington, Virginia, did she visit the Pentagon or
meet with Pentagon officials, since it was less than a mile away?
19. When Prof. Zhengli received a visa to the United States to present
at the US-China Workshop on Frontiers in Ecology and Evolution
of Infectious Diseases conference at UC Berkeley in 2018, did she
visit Federal research facility, Lawrence-Berkeley-Livermore
Laboratory — in particular, the Department of Energy’s Joint
Genome Institute — or meet with government officials, since it
was only a mile and a half away?
20. Prof. Zhengli’s C.V. indicates she received a visa to the United
States to present at the U.S.-China Dialogue on the Challenges of
Emerging Infections, Laboratory Safety and Global Health
Security conference on January 17, 2018, in Galveston, Texas.
However, such a conference appears to not have existed. For
what purpose did she really come to Galveston — perhaps to visit
the Galveston National Laboratory, a high security National

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Biocontainment Laboratory housing several Biosafety Level 4
research laboratories, one of the only 15 biosecurity Level 4
facilities in the United States and the largest one in the world
located on an academic campus? Was this apparently imaginary
conference merely a ruse for a surreptitious rendezvous?
21. Of Prof. Zhengli’s 130 published scientific studies, 5 of them are
not to be found anywhere. Why are they not public? Are they
classified?
22. Has Prof. Zhengli (or any other staff, resident or guest scientists,
researchers, students, visitors, or others) at the Wuhan National
Biosafety Laboratory, or at Wuhan Institute of Virology in general,
collaborated, participated with, colluded with, or in any way
professionally acted in concert or collusion with, or in any way
worked with or for, the World Economic Forum, the U.S. Center for
Disease Control, the Bill and Melinda Gates Foundation, the
Pilbright Institute, the European Commission, the World Health
Organization, the Biotechnology and Biological Sciences
Research Council, or the John Hopkins Center for Health
Security?
23. Prof. Zhengli recently (January 23, 2020) claimed to know very
little about the latest epidemic outbreak, including basic biology,
animal source, or any specific treatment, and indicated she
doesn’t know if ACE2 targeting drugs could treat Coronavirus
2019-nCoV infected victims. How can this be the case, given that
she has studied human ACE2/coronavirus interaction for many
years — even most recently in her study immediately preceding
the outbreak — as reported in Prof. Zhengli’s study published the
day immediately preceding the outbreak? “The full-length genes
of MERS-CoV spike (GenBank accession number 415

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AFS88936.1), SARS-CoV spike (GenBank accession number
AFR58742), human DPP4 416 (GenBank accession number
NM_001935.3) and human ACE2 (GenBank accession 417 number
NM_021804) were synthesized (GenScript Biotech).”
24. Considering Prof. Zhengli is the recipient of millions of dollars in
grants and salaries, commands one of the world’s leading, most
advanced biosafety laboratories, has performed innumerable
research studies into coronaviruses for three decades and
counting — what vaccines, to date, has she successfully
produced? Has she produced any successful coronavirus
vaccines at all? If so, where are they and how have they been
publicly administered?
Summary, conclusion, and just a wee bit of speculation
The facts presented herein compel an alternative theory as to the

origin of the Coronavirus 2019-nCoV outbreak. The truth remains to be
formally investigated whether infected viral bio-matter from the
National Biosafety Laboratory at Wuhan Institute of Virology — the
only lab of its kind in all of China and under expressed safety concerns
for almost a year — somehow escaped. And, if so, it also remains to be
seen whether such a viral release and subsequent viral infection was
accidental or intentional. In any event, the following observations and
concerns seem to place considerable suspicion on the laboratory —
and its Senior Scientist and Principal Investigator, Prof. Zhengli Shi —
and its contemporaneous coronavirus research activity at the exact
time of the Coronavirus 2019-nCoV outbreak officially reported at a
location conveniently just 8.6 miles distant at Huanan Seafood
Wholesale Market, just across the Yangtze River:

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1. The National Biosafety Laboratory at Wuhan Institute of Virology
is the only high-level P4 facility of its kind in all of China, literally
the only place where high contagious and infectious pathogens
and diseases such as Ebola, SARS, MERS, and assorted
coronaviruses can be “safely” studied, mutated, and engineered.
2. The professional background, experience, and qualifications of
the Wuhan National Biosafety Laboratory’s Senior Scientist and
Principal Investigator — Professor Zhengli Shi — is nonpareil. She
has commandeered, produced and/or co-authored over 130
scientific studies, including dozens of reports specifically on
coronaviruses. So specialized and talented is she that the even
the United States has granted her over $1 million for her research
conducted in China.
3. It cannot be overstated the importance and implication of the
short distance between the Wuhan National Biosafety Laboratory
and the reported epicenter of Coronavirus 2019-nCoV outbreak
— the Huanan Seafood Wholesale Market — of only 8.6 miles.
With a total area of 3.8 million square miles, and a breadth of
about 3,000 miles, these two locations are relatively-speaking
right next to each other. Even before the lab’s government-
announced formal operational opening, American scientists and
biosafety experts had expressed their concerns for the laboratory,
especially its proximity to the relatively large population of Wuhan,
capital city of Hubei province.
4. At the time of the new coronavirus outbreak, or immediately
preceding it, Prof. Zhengli was actively conducting coronavirus
experiments and research at the Wuhan National Biosafety
Laboratory. Notably, the very next day following the publishing of
her coronavirus study on December 11, 2019, the first victims of

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Coronavirus 2019-nCoV were reported, as confessed by Prof.
Zhengli herself in her most recent, latest report, posted online on
January 23, 2020: “The epidemic, started from December 12th,
2019, has caused 198 laboratory confirmed infections with three
fatal cases by January 20th, 2020.”
5. Most alarming is the apparent, glaring disingenuousness of Prof.
Zhengli’s latest report, which is the only public statement since
the official Chinese acknowledgement of Coronavirus 2019-nCoV
outbreak in Wuhan. On January 23, 2020, she published the
report with the allegedly misleading statements:
“Finally, based on our results, it should be expected and worth to

test if ACE2 targeting or SARS-CoV targeting drugs can be used for
nCoV-2019 patients. At this stage, we know very little about the
virus, including basic biology, animal source or any specific
treatment. The almost identical sequences of this virus in different
patients imply a probably recent introduction in humans, thus
future surveillance on viral mutation and transmission ability and
further global research attention are urgently needed.”
However, other Chinese scientists reported on January 22, 2020,

“Results obtained from our analyses suggest that the 2019-nCoV
appears to be a recombinant virus between the bat coronavirus and an
origin-unknown coronavirus. The recombination occurred within the
viral spike glycoprotein, which recognizes cell surface receptor.” Our
findings suggest “that homologous recombination within the spike
glycoprotein may contribute to cross-species transmission.” Although
this other scientific team incorrectly attributes the originating species
as reptilian (snake) instead of bats, they at least rapidly identified the
coronavirus as a recombinant virus with one of the contributors being

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a bat coronavirus, and also discerned in what manner the genetic
recombination occurred to allow for human infection: in a viral spike
protein which recognized the cell surface receptor. But as shown
previously, this precise area of coronavirus study involving spike
protein and cell surface receptor was the focus of Prof. Zhengli’s
contemporaneous December 2019 study published the day before the
epidemic started. “Coronavirus spike protein mediates viral entry into
cells by first binding to a receptor on host cell surface and then fusing
viral and host membranes,” she wrote. Why would she feign ignorance
about this?
Even more concerning, on October 31, 2019, Prof. Zhengli had

published a report entitled Filovirus-reactive antibodies in humans and
bats in Northeast India imply zoonotic spillover, curiously funded by
the U.S. Department of Defense, the U.S. Defense Threat
Reduction Agency, the U.S. Biological Defense Research
Directorate of the Naval Medical Research Center, and the
Department of Atomic Energy of the Government of India, and
edited by a microbiologist employed by the U.S. Center for Disease
Control.

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U.S. Defense Threat Reduction Agency? Can viruses from bats be used as weapons of mass destruction?
Of note is the fact that the Defense Threat Reduction Agency is an

agency within the U.S. Department of Defense and is the official
Combat Support Agency for countering weapons of mass
destruction. Why would they be funding this project? Could it be that
these coronaviruses with filovirus reactive antibodies are being
weaponised? Are they really that dangerous? Could they actually be

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employed as a weapon of mass destruction? Well, let’s a take a look at
what Prof. Zhengli was studying, filovirus surface glycoproteins:
Bats are reservoirs for several zoonotic pathogens, including

filoviruses. High risk activities at the bat-human interface pose the
threat of zoonotic virus transmission. We present evidence for prior
exposure of bat harvesters and two resident fruit bat species to
filovirus surface glycoproteins. Our results indicate circulation of
several filoviruses in bats and the possibility for filovirus
transmission from bats to humans. Filoviruses, including
ebolaviruses and marburgviruses, are pathogens with epidemic
potential. They were previously detected in bats and have caused
disease outbreaks in humans with a high case fatality rate. Our
findings suggest bats in South Asia act as a reservoir host of a
diverse range of filoviruses and filovirus spillover occurs through
human exposure to these bats.
Thus, it’s readily apparent that just from this single project that Prof.
Zhengli was quite aware that pathogenic viruses from bats could
transmit from bats to humans via filovirus surface glycoproteins, with
potentially epidemic consequences. Could our brilliant, pioneering,
decorated Senior Scientist and Principal Investigator of the only Level
P4 Biosafety Laboratory in China be feigning ignorance presently to
deflect discovery of her connections to four major defense agencies
and her possible stewardship of a brand-new bioactive weapon of
mass destruction? At this point, only speculation is possible…but if
we’re going to speculate, let’s take one step more, shall we?
Could there be a another study previously spearheaded by Prof.

Zhengli whose findings may have attracted multiple American defense

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departments for such a project with epidemic potential? Perhaps we
can find the answer in the study, Discovery of Novel Bat Coronaviruses
in South China That Use the Same Receptor as Middle East
Respiratory Syndrome Coronavirus, a seemingly important and
relevant 2018 project where Prof. Zhengli provided evidence of a
Middle East respiratory syndrome coronavirus (MERS-CoV) “derived
from the great evening bat that uses the same host receptor as human
MERS-CoV. This virus also provides evidence for a natural
recombination event between the bat MERS-related CoV and another
bat coronavirus, HKU4” (emphasis added). The purpose of this study
was “the prevention and control of the spread of MERS-CoV to
humans.” It pertains precisely to the implications presented by the
current Coronavirus 2019-nCoV, which were identified by the other
group of Chinese scientists as a bat-involved, recombinant virus with a
viral spike protein, recognizing cell surface receptor and so able to
infect human cells.
And yet another highly relevant study with the potential to capture the
attention of biowarfare officials in United States defense departments
is Discovery of a Rich Gene Pool of Bat SARS-related Coronaviruses
Provides New Insights Into the Origin of SARS Coronavirus, published
in November 2017, where Prof. Zhengli and her colleagues conducted
cell entry studies which “demonstrated that three newly identified
SARSr-CoVs [SARS-related coronaviruses] with different [spike]
protein sequences are all able to use human ACE2 as the receptor,
further exhibiting the close relationship between strains in this cave
and SARS-CoV. This work provides new insights into the origin and
evolution of SARS-CoV and highlights the necessity of preparedness
for future emergence of SARS-like diseases” (emphasis added).

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All of the studies cited here appear related and interconnected, and
considering the involvement of American defense agencies — in
particular, the U.S. Defense Threat Reduction Agency which deals
exclusively with matters pertaining to weapons of mass destruction
and threat networks — there seems ample reason to be gravely
concerned. And that concern remains whether there’s reason to
suspect coronaviruses could be used by others as bioweapons of
mass destruction, or that rogue, Deep State operatives within our own
defense departments — colluding with Communists — are developing
or have already developed a bioweapon of mass destruction.
In conclusion, though admittedly much investigation remains to be

performed (especially into the numerous unanswered questions posed
in this essay), it seems the likeliest source of origin for Coronavirus
2019-nCoV is the Wuhan National Biosafety Laboratory at the Wuhan
Institute of Virology. Further, it appears to me that, at best, there may
be concerted efforts to conceal the precise nature of the virus, its
source, and the parties responsible, or that, at worst, the
dissemination of the epidemic coronavirus is intentional. Could the
actual RNA genome source, sequencing and recombination of the
coronavirus already be known, and could its vaccine have already
been developed? Could it already be patented? Essentially, is this
latest global pandemic threat a Communist cover-up, or a pandemic
bioweapon of mass destruction developed by the global Deep State?
Appendix A: Professor Zhengli Shi’s published scientific papers
1. Zhou, P., # Fan, H., # Lan, T., # Yang, X-L, Shi, W-F, Zhang, W., Zhu.
Y., Zhang, Y-W., Xie, Q-M., Mani, S., Zheng, X-S., Li, B., Li, J-M., Guo,
H., Pei, G-Q., An, X-P., Chen J-W., Zhou, L., Mai, K-J., Wu, Z-X., Li, D.,

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Anderson, D.E., Zhang, L-B., Li, S-Y., Mi, Z-Q., He, T-T., Cong, F., Guo,
P-J., Huang, R., Luo, Y., Liu, X-L., Chen, J., Huang, Y., Sun, Q., Zhang,
X-L-L., Wang, Y-Y., Xing, S-Z., Chen, Y-S., Sun, Y., Li, J., Daszak, P.*,
Wang, L-F.*, Shi, Z-L.*, Tong, Y-G.*, Ma, J-Y.* (2018). Fatal swine acute
diarrhoea syndrome caused by an HKU2-related coronavirus of bat
origin. Nature, 556 (7700): 255–258.
2. Xie, J.Z., Li, Y., Shen, X., Goh, G., Zhu, Y., Wang, L-F., Cui, J., Shi, Z-
L.,* Zhou, P.* (2018). Dampened STING-dependent interferon
activation in bats. Cell Host Microbe, 23(3): 297–301 e4.
3. Li, W., Wang, B., Li, B., Zhang, W., Zhu, Y., Shi, Z. L. & Yang, X. L*.
(2018). Genomic Characterization of a novel hepatovirus from great
roundleaf bats in China. Virol Sin 33 (1), 108–110.
4. Luo, C. M., Wang, N., Yang, X. L., Liu, H. Z., Zhang, W., Li, B., Hu, B.,
Peng, C., Geng, Q. B., Zhu, G. J., Li, F*. & Shi, Z. L*. (2018). Discovery
of novel bat coronaviruses in South China that use the same receptor
as Middle East respiratory syndrome coronavirus. J Virol 92 (13).
10.1128/JVI.00116–18.
5. Luo, Y., Li, B., Jiang, R. D., Hu, B. J., Luo, D. S., Zhu, G. J., Hu, B., Liu,
H. Z., Zhang, Y. Z., Yang, X. L. & Shi, Z. L*. (2018). Longitudinal
surveillance of betacoronaviruses in fruit bats in Yunnan province,
China during 2009–2016. Virol Sin 33 (1), 87–95.
6. Wang, B., Li, W., Zhou, J. H., Li, B., Zhang, W., Yang, W. H., Pan, H.,
Wang, L. X., Bock, C. T., Shi, Z. L., Zhang, Y. Z*. & Yang, X. L*. (2018).
Chevrier’s field mouse (Apodemus chevrieri) and Pere David’s vole
(Eothenomys melanogaster) in China carry orthohepeviruses that form
two putative novel genotypes within the species orthohepevirus C.

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Virol Sin 33 (1), 44–58.
7. Wang, N., Li, S. Y., Yang, X. L., Huang, H. M., Zhang, Y. J., Guo, H.,
Luo, C. M., Miller, M., Zhu, G., Chmura, A. A., Hagan, E., Zhou, J. H.,
Zhang, Y. Z., Wang, L. F., Daszak, P. & Shi, Z. L*. (2018). Serological
evidence of bat SARS-related coronavirus infection in humans, China.
Virol Sin 33 (1), 104–107.
8. Hu, B., Zeng, L.P., Yang, X.L., Ge, X.Y., Zhang, W., Li, B., Xie, J.Z.,
Shen, X.R., Zhang, Y.Z., Wang, N., Luo, D.S., Zheng, X.S., Wang, M.N.,
Daszak, P., Wang, L.F., Cui, J.*, Shi, Z.L*. (2017). Discovery of a rich
gene pool of bat SARS-related coronaviruses provides new insights
into the origin of SARS coronavirus. PloS Pathogens 13(11): e1006698.
9. Waruhiu, C#., Ommeh, S#., Obanda, V., Agwanda, B., Gakuya, F.,
Ge, X. Y., Yang, X. L., Wu, L. J., Zohaib, A., Hu, B. & Shi, Z. L*. (2017).
Molecular detection of viruses in Kenyan bats and discovery of novel
astroviruses, caliciviruses and rotaviruses. Virol Sin. 32 (2), 101–114.
10. Zhang, Q., Zeng, L.P., Zhou, P., Irving, A.T., Li, S., Shi, Z.L.*, Wang,
L.F. (2017). IFNAR2-dependent gene expression profile induced by
IFN-α in Pteropus alecto bat cells and impact of IFNAR2 knockout on
virus infection. PloS One. 12(8):e0182866.
11. Wang, B., Cai, C.L, Li, B., Zhang, W., Zhu, Y., Chen, W.H., Zhuo, F.,
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in wild rodents and shrews from Shenzhen city, China. Virol Sin.
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12. Zeng, L.P., Ge, X.Y., Peng, C., Tai, W.B., Jiang, S.B., Du, L.Y.*, Shi,
Z.L.* (2017). Cross-neutralization of SARS coronavirus-specific
antibodies against bat SARS-like coronaviruses. Sci China Life Sci.
60(12):1399–1402.
13. Wang, B., Yang, X. L., Li, W., Zhu, Y., Ge, X. Y., Zhang, L. B., Zhang,
Y. Z., Bock, C. T. & Shi, Z. L.* (2017). Detection and genome
characterization of four novel bat hepadnaviruses and a hepevirus in
China. Virol J. 14:40.
14. Liang, J., Yang, X.L., Li, B., Liu, Q., Zhang, Q., Liu, H., Kan, H.P.,
Wong, K.C., Chek, S.N., He, X., Peng, X., Shi, Z.L., Wu, Y.* & Zhang, L.*
(2017). Detection of diverse viruses in alimentary specimens of bats in
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15. Ge, X.Y., Yang, W.H., Zhou, J.H., Li, B., Zhang, W., Shi, Z.L.* &
Zhang, Y.Z.* (2017). Detection of alpha- and betacoronaviruses in
rodents from Yunnan, China. Virol J. 14:98.
16. Waruhiu, C., Ommeh, S., Obanda, V., Agwanda, B., Gakuya, F., Ge,
X.Y., Yang, X.L., Wu, L.J., Zohaib, A., Hu. B., Shi, Z.L.* (2017).
Molecular detection of viruses in Kenyan bats and discovery of novel
astroviruses, caliciviruses and rotaviruses. Virol Sin. 32(2):101–114.
17. Tan, B., Yang, X. L., Ge, X. Y., Peng, C., Liu, H. Z., Zhang, Y. Z.,
Zhang, L. B. & Shi, Z. L.* (2017). Novel bat adenoviruses with low G+C
content shed new light on the evolution of adenoviruses. J Gen Virol.
98(4):739–748.
18. Yang, X. L., Zhang, Y. Z., Jiang, R. D., Guo, H., Zhang, W., Li, B.,
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Wang, L. F. & Shi, Z. L.* (2017). Genetically Diverse Filoviruses in
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19. Tan, B., Wu, L.J., Yang, X.L., Li, B., Zhang, W., Lei, Y.S., Yang, G.X.,
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characterization of adenoviruses infecting endangered golden snub-
nosed monkeys (Rhinopithecus roxellana). Virol J. 13:190
20. Zeng, L. P., Gao, Y. T., Ge, X. Y., Zhang, Q., Peng, C., Yang, X. L.,
Tan, B., Chen, J., Chmura, A. A., Daszak, P. & Shi, Z. L*. (2016). Bat
Severe Acute Respiratory Syndrome-Like Coronavirus WIV1 Encodes
an Extra Accessory Protein, ORFX, Involved in Modulation of the Host
Immune Response. J Virol 90 (6), 6573–6582.
21. Tan, B., Yang, X. L., Ge, X. Y., Peng, C., Zhang, Y. Z., Zhang, L. B. &
Shi, Z. L*. (2016). Novel bat adenoviruses with an extremely large E3
gene. J Gen Virol., 97, 1625–1635.
22. Ge, X. Y., Yang, W. H., Pan, H., Zhou, J. H., Han, X., Zhu, G. J.,
Desmond, J. S., Daszak, P., Shi, Z. L*. & Zhang, Y. Z*. (2016). Fugong
virus, a novel hantavirus harbored by the small oriental vole
(Eothenomys eleusis) in China. Virol J., 13, 27.
23. Pan, X., Cao, Z., Yuan, J., Shi, Z., Yuan, X., Lin, L., Xu, Y., Yao, J.,
Hao, G. & Shen, J. (2016). Isolation and Characterization of a Novel
Dicistrovirus Associated with Moralities of the Great Freshwater Prawn,
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24. Yang, X.-L., Hu, B., Wang, B., Wang, M.-N., Zhang, Q., Zhang, W.,
Wu, L.-J., Ge, X.-Y., Zhang, Y.-Z., Daszak, P., Wang, L.-F. & Shi, Z.-L*.

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(2016). Isolation and
Characterization of a Novel Bat Coronavirus Closely Related to the

Direct Progenitor of Severe Acute Respiratory Syndrome Coronavirus.
J Virol., 90, 3253–3256.
25. Wang, M. N., Zhang, W., Gao, Y. T., Hu, B., Ge, X. Y., Yang, X. L.,
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like coronaviruses in bats by quantitative real-time PCR. Virol Sin., 31,
78–80.
26. Ge, X. Y., Wang, N., Zhang, W., Hu, B., Li, B., Zhang, Y. Z., Zhou, J.
H., Luo, C. M., Yang, X. L., Wu, L. J., Wang, B., Zhang, Y., Li, Z. X. &
Shi, Z. L*. (2016). Coexistence of multiple coronaviruses in several bat
colonies in an abandoned mineshaft. Virol Sin., 31, 31–40.
27. Hu, B., Ge X., Wang, L. F., Shi, Z*. (2015). Bat origin of human
coronaviruses. Virol J., 12(1): 221.
28. Liang, Y. Z., Wu, L. J., Zhang, Q., Zhou, P., Wang, M. N, Yang, X. L,
Ge, X. Y, Wang, L. F, Shi, Z. L*. (2015). Cloning, expression, and
antiviral activity of interferon beta from the Chinese microbat, Myotis
davidii. Virol Sin., 30(6):425–432.
29. Yang, X. L., Tan, B., Wang, B., Li, W., Wang, N., Luo, C. M., Wang,
M. N., Zhang, W., Li, B., Peng, C., Ge, X. Y., Zhang, L. B.,Shi, Z*.(2015).
Isolation and identification of bat viruses closely related to human,
porcine, and mink orthoreoviruses. J Gen Virol. 96(12):3525–3531.
30. Wang MN, Ge XY, Wu YQ, Yang XL, Tan B, Zhang YJ,Shi ZL*. 2015.
Genetic diversity and temporal dynamics of phytoplankton viruses in

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East Lake, china. Virol Sin, 30: 290–300.
31. Wang Y, Sun Y, Wu A, Xu S, Pan R, Zeng C, Jin X, Ge X, Shi Z,

Ahola T, Chen Y, Guo D*. 2015. Coronavirus nsp10/nsp16
methyltransferase can be targeted by nsp10-derived peptide in vitro
and in vivo to reduce replication and pathogenesis. J Virol, 89: 8416–
8427.
32. Yang Y, Liu C, Du L, Jiang S, Shi Z, Baric RS, Li F*. 2015. Two
mutations were critical for bat-to-human transmission of Middle East
respiratory syndrome coronavirus. J Virol, 89: 9119–9123.
33. Menachery VD, Yount Jr BL, Debbink K, Agnihothram S, Gralinski

LE, Plante JA, Graham RL, Scobey T, Ge X-Y, Donaldson EF, Randell
SH, Lanzavecchia A, Marasco WA,Shi Z-L, Baric RS*. 2015. A SARS-
like cluster of circulating bat coronaviruses shows potential for human
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34. Mazet JK., Wei Q, Zhao GP, Cummings DT, Desmond JS, Rosenthal
J，King CH., Cao WC, Chmura AA, Hagan EA, Zhang SY, Xiao XM, Xu
JG, Shi Z, Feng F, Liu XP, Pan WQ, Zhu GJ, Zuo LY & Daszak P. (2015).
Joint China-US Call for Employing a Transdisciplinary Approach to
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1060–1.
35. Hu, B., Chmura, A. A., Li, J., Zhu, G., Desmond, J. S., Zhang, Y.,
Zhang, W., Epstein, J. H., Daszak, P. & Shi, Z*.(2014). Detection of
diverse novel astroviruses from small mammals in China. J Gen Virol
95, 2442–2449.
36. Ge, X-Y., Li, J-L., Yang, X-L., Chmura, A.A., Zhu, G., Epstein, J.H.,

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Mazet, J.K., Hu, B., Zhang, W., Peng, C., Zhang, Y.J., Luo, C.M., Tan,
B., Wang, N., Zhu, Y., Crameri, G., Zhang, S.Y., Wang, L.F., Daszak, P.*,
Shi, Z-L*.(2013). Isolation and characterizationof a bat SARS-like
coronavirus that uses the ACE2 receptor. Nature, 503(7477):535–538.
37. Zhang, G#., Cowled, C#., Shi, Z#., Huang, Z#., Bishop-Lilly, K. A#.,
Fang, X., Wynne, J. W., Xiong, Z., Baker, M. L., Zhao, W., Tachedjian,
M., Zhu, Y., Zhou, P., Jiang, X., Ng, J., Yang, L., Wu, L., Xiao, J., Feng,
Y., Chen, Y., Sun, X., Zhang, Y., Marsh, G. A., Crameri, G., Broder, C. C.,
Frey, K. G*., Wang, L. F*. & Wang, J*. (2013). Comparative Analysis of
Bat Genomes Provides Insight into the Evolution of Flight and
Immunity. Science 339 (6118):456–460.
38. Wu, L., Zhou, P., Ge, X., Wang, L. F., Baker, M. L. & Shi, Z*. (2013).
Deep RNA sequencing reveals complex transcriptional landscape of a
bat adenovirus. J Virol 87, 503–511.
39. Shi, Z. Emerging infectious diseases associated with bat viruses.

(2013). Sci China Life Sci. 56: 678–682.
40. Zhou, P., Han, Z., Wang, L. and Shi, Z*. (2013). Identification of
Immunogenic Determinants of the Spike Protein of SARS-like
Coronavirus. Virol Sin 28, (2):92–96.
41. Xia, H., Wang, M., Ge, X., Wu, Y., Yang, X., Zhang, Y., Li, T. and Shi,
Z*. (2013). Study of the Dynamics of Microcystis aeruginosa and its
Cyanophage in East Lake using quantitative PCR. Virol Sin 28 (5): 309–
311.
42. Ge, X., Wu, Y., Wang, M., Wang, J., Wu, L., Yang, X., Zhang, Y. and
Shi, Z*. (2013). Viral Metagenomics Analysis of Planktonic Viruses in

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East Lake, Wuhan, China. Virol Sin 28 (5): 280–290.
43. Yuan, J., Zhang, Y., Li, J., Zhang, Y., Wang, L. F. & Shi, Z*. (2012).
Serological evidence of ebolavirus infection in bats, China. Virology
Journal 9, 236.
44. Ge, X., Li, Y., Yang, X., Zhang, H., Zhou, P., Zhang, Y. & Shi, Z*.
(2012). Metagenomic analysis of viruses from bat fecal samples
reveals many novel viruses in insectivorous bats in China. J Virol
86(8):4620–4630.
45. Yang, X., Zhang, Y., Ge, X., Yuan, J. & Shi, Z*. (2012). A novel
totivirus-like virus isolated from bat guano. Arch Virol, 157:1093–1099.
46. Yuan, J., Su, N., Wang, M., Xie, P., Shi, Z. & Li, L. (2012). Down-
regulation of heme oxygenase-1 by SVCV infection. Fish & shellfish
immunology 32, 301–306.
47. Zhou, P., Li, H., Wang, H., Wang, L. F. & Shi, Z*. (2012). Bat severe
acute respiratory syndrome-like coronavirus ORF3b homologues
display different interferon antagonist activities. J Gen Virol 93, 275–
281.
48. Zhou, P., Cowled, C., Marsh, G. A., Shi, Z., Wang, L. F. and Baker,
M. L. (2011). Type III IFN Receptor Expression and Functional
Characterisation in the Pteropid Bat, Pteropus alecto. PloS one 6,
e25385.
49. Zhou, P., Cowled, C., Todd, S., Crameri, G., Virtue, E. R., Marsh, G.
A., Klein, R., Shi, Z., Wang, L. F. and Baker, M. L. (2011). Type III IFNs in
pteropid bats: differential expression patterns provide evidence for

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distinct roles in antiviral immunity. J Immunol 186:3138–3147.
50. Ge, X., Li, J., Peng, C., Wu, L., Yang, X., Wu, Y., Zhang, Y. and Shi,
Z*. (2011). Genetic diversity of novel circular ssDNA viruses in bats in
China. J Gen Virol., 92:2646–2653.
51. Bai, H., Wang, Y., Li, X., Mao, H., Li, Y., Han, S., Shi, Z. and Chen, X.
(2011). Isolation and characterization of a novel alphanodavirus. Virol J
8:311.
52. Tan, Y. W., and Shi, Z*. (2011). Genotyping of white spot syndrome
virus in Chinese cultured shrimp during 1998–1999. Virol Sin 26:123–
130.
53. Xing, Y., and Shi, Z*. (2011). Nucleocapsid protein VP15 of White
spot syndrome virus colocalizes with the nucleolar proteins nucleolin
and fibrillarin. Can J Microbiol., 57:759–764.
54. Yuan, J., Marsh, G., Khetawat, D., Broder, C. C., Wang, L. F. and
Shi, Z*. (2011). Mutations in the G-H loop region of ephrin-B2 can
enhance Nipah virus binding and infection. J Gen Virol 92:2142–2152.
55. Zhang, Y., Yuan, J., Yang, X., Zhou, J., Yang, W., Peng, C., Zhang,
H. L. and Shi, Z*. (2011). A novel hantavirus detected in Yunnan red-
backed vole (Eothenomys miletus) in China. J Gen Virol 92:1454–1457.
56. Zhou B., Y. Li, J. Belser, M. Pearce, M. Schmolke, A. Subba, Z. Shi,

S. Zaki, D. Blau, A. Sastre, T. Tumpey, D. Wentworth*. (2011). NS
deletions convert the 2009-H1N1 pandemic virus into a live attenuated
vaccine. Influenza and Other Respiratory Viruses. 5:388–391.

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57. Yu, M., Tachedjian, M., Crameri, G., Shi, Z., and Wang, L. F. (2010).
Identification of key amino acid residues required for horseshoe bat
angiotensin-I converting enzyme 2 to function as a receptor for severe
acute respiratory syndrome coronavirus. J Gen Virol 91(Pt 7), 1708–
1712.
58. Hou, Y., Peng, C., Yu, M., Li,Y., Han, Z., Wang, L-F., Li, F., Shi, Z.*
(2010). Bat Angiotensin Converting Enzyme-2 Displays Different
Receptor Activity to Severe Acute Respiratory Syndrome Coronavirus
Entry. Arch Virol., 155, (10 ): 1563–1569.
59. Li, Y., Ge X., Hon C. C., Zhang H., Zhou P., Zhang Y., Wang L. F.
and Shi Z*. (2010). Prevalence and Genetic Diversity of Adeno-
Associated Viruses in Bats, China. J Gen Virol. 91(10): 2601–2609.
60. Zhang Y., Zhang H., Dong X., Yuan J., Zhang H., Yang X., Zhou
Peng., Ge X., Li Y., Wang L-F, and Shi Z* (2010). Hantavirus Outbreak
Associated with Laboratory Rats in Yunnan, China. Infection, Genetics
and Evolution. 10(5): 638–644.
61. Li, Y., Ge X., Zhang H., Zhou P., Zhu Y., Zhang Y., Yuan J., Wang L-
F., Shi Z.* (2010). Host Range, Prevalence and Genetic Diversity of
Adenoviruses in Bats. J. Virol. 84 (8):3889–3897.
62. Yuan, J., Hon,C. C., Li, Y., Wang, D., Xu, G., Zhang, H., Zhou, P.,
Poon, L. M., Lam, T. T. Leung, F. C. and Shi, Z*. (2010). Intra-species
Diversity of SARS-Like Coronaviruses (CoVs) in Rhinolophus sinicus
and Its Implications on the Origin of SARS-CoVs in human. J Gen Virol.
91(4):1058–1062.
63. Liao, M., Cheng, K., Yang, J., Zhao, Y., Shi, Z*. (2010). Assessment

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of UV-B damage in cyanophage PP. Aquat Microb Ecol 58: 323–328.
64. Shi,Z. (2010) Bat and virus. Protein Cell 2010, 1(2): 109–114
65. Hou, Y., P., Han, Z., Zhou, P., Chen, J. and Shi, Z*. (2010).
Immunogenicity of the Spike Glycoprotein of Bat SARS-like
Coronavirus. Virol Sinica, 25 (1):36–44.
66. Li, H., Zheng, Z., Zhou, P., Zhang, B., Shi, Z., Hu, Q. and Wang, H.
(2010). The cysteine protease domain of porcine reproductive and
respiratory syndrome virus non-structural
protein 2 antagonizes interferon regulatory factor 3 activation. J Gen

Virol. 91(12), 2947–2958.
67. Zhou, B., Li, Y., Belser, J. A., Pearce, M. B., Schmolke, M., Subba, A.
X., Shi, Z., Zaki, S. R., Blau, D. M., Garcia-Sastre, A., Tumpey, T. M. &
Wentworth, D. E. (2010). NS-based live attenuated H1N1 pandemic
vaccines protect mice and ferrets. Vaccine 28, 8015–8025.
68. Tang, X. C.‚ Li, G.‚ Vasilakis, N.‚ Zhang, Y.‚ Shi, Z.L‚ Zhong, Y.‚
Wang, L.F.‚ Zhang, S. Y. (2009) Differential stepwise evolution of SARS
Coronavirus functional proteins in different host species. BMC Evol
Biol 9: 52.
69. Zhou, P., Han, Z., Wang, L.F. and Shi, Z*. (2009) Immunogenicity
difference between the SARS coronavirus and the bat SARS-like
coronavirus spike (S) proteins. Biochem Biophys Res Commun 387(2),
326–329.
70. Tan, Y., Xing, Y., Zhang, H., Feng, Y., Zhou, Y. and Shi, Z*. (2009)

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Molecular detection of three shrimp viruses and genetic variation of
white spot syndrome virus in Hainan province, China, in 2007. J Fish
Dis, 32: 777–784.
71. Yuan, J., Li, Y., Zhang, H., Zhou, P., Ke, Z., Zhang, Y. and Shi, Z*.
(2009) Indirect enzyme-linked immunosorbent assay based on the
nucleocapsid protein of SARS-like coronaviruses. Virol Sinica 24 (2):
146–151.
72. Li, L., Zhang, H., Zhang C., Shi Z*. (2009) Identification and
characterization of nuclear localization signals within the nucleocapsid
protein VP15 of White Spot Syndrome Virus. Virol Sinica 24 (1):71–76
73. Wang, J., Zhang, H. and Shi, Z*. (2008) Expression and assembly
mechanism of the capsid proteins of a satellite virus (XSV) associated
with Macrobrachium rosenbergii nodavirus. Virol Sinica 23 (1):73–77.
74. Tang, Y., Shi, Z*. (2008) Proteomic analyses of the shrimp white
spot syndrome virus. Virol Sinica 23 (3):157–166.
75. Bai, B., Hu, Q., Hu, H., Zhou, P., Shi, Z., Meng, J., Huang, Y., Lu, B.,
Mao, P., Wang, H. (2008) Virus-like particles of SARS-like coronavirus
formed by membrane proteins from different origins demonstrate
stimulating activity in human dendritic cells. 3(7), e2685.
76. Li,Y., Wang, J., Hickey, A. C., Zhang, Y., Li, Y., Wu, Y., Zhang, H.,
Yuan, J., Han, Z., McEachern, J., Broder, C. C., Wang, L. F. and Shi, Z*.
(2008) Antibodies to Nipah or Nipah-like viruses in bats, China. Emerg
Infect Dis 14(12):1974–1976.
77. Wang, J., Wang, L-F. and Shi, Z*. (2008) Construction of a non-

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infectious SARS coronavirus replicon for application in drug screening
and analysis of viral protein function. Biochem Biophys Res Commun
374(1):138–142.
78. Yu, M., Stevens, V., Berry, J. D., Crameri, G., McEachern, J., Tu, C.,
Shi, Z., Liang, G., Weingart, H., Cardosa, J., Eaton, B. T., Wang, L. F.
(2008) Determination and application of immunodominant regions of
SARS coronavirus spike and nucleocapsid proteins recognized by sera
from different animal species. J Immunol Methods 331(1–2):1–12.
79. Hon, C. C., Lam, T. Y., Shi, Z., Drummond, A. J., Yip, C. W., Zeng,
F., Lam, P. Y. and Leung, F. C.. (2008) Evidence of the recombinant
origin of a bat severe acute respiratory syndrome (SARS)-like
coronavirus and its implications on the direct ancestor of SARS
coronavirus. J Virol 82(4): 1819–1826.
80. Ren, W., Qu, X., Li, W., Han, Z., Yu, M., Zhang, S., Wang, L. F.,
Deng, H., Shi, Z*. (2008) Difference in receptor usage between SARS
coronavirus and SARS-like coronavirus of bat origin. J Virol 82(4):
1899–1907.
81. Shi, Z. and Hu, Z. (2008) A review of studies on animal reservoirs

of the SARS coronavirus. Virus research 133:74–87.
82. Cheng, K., Zhao, Y., Du, X., Zhang, Y., Lan, S., Shi, Z*. (2007) Solar
radiation-driven decay of cyanophage infectivity, and
photoreactivation of the cyanophage by host cyanobacteria. Aquatic
Microbial Ecology 48(1): 13–18.
83. Cui, J., Han, N., Streicker, D., Li, G.., Tang, X., Shi, Z., Hu, Z., Zhao,
G., Fontanet, A., Guan, Y., Wang, L., Jones, G., Field, H. E., Daszak, P.

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and Zhang, S. (2007) Evolutionary relationships among bat
coronaviruses and their hosts. Emerg Infect Dis 13(10):1526–1532.
84. Zhang, C, Yuan, J, Shi, Z*. (2007) Molecular epidemiological

investigation of infectious hypodermal and hematopoietic necrosis
virus and taura syndrome virus in Penaeus vannamei cultured in China.
Virol Sinica 22(5): 380–388.
85. Gu, W., Yuan, J., Xu, G., Li, L., Liu, N., Zhang, C., Zhang, J. and Shi,
Z*. (2007) Production and characterization of monoclonal antibody of
shrimp white syndrome virus envelope protein VP28. Virol Sinica 22(1):
21–25.
86. Wang, L. F., Shi, Z., Zhang, S., Field, H., Daszak, P. and Eaton B. T.
(2006) A review of bats and SARS: virus origin and genetic diversity.
Emerg Infect Dis 12(12): 1834–1840.
87. Ren, W., Li, W., Yu, M., Hao, P., Zhang, Y., Zhou, P., Zhang, S., Zhao,
G., Zhong, Y., Wang, S., Wang, L. F. and Shi, Z*. (2006) Full genome
sequences of two SARS-like coronaviruses in horseshoe bats and
genetic variation analysis. J Gen Virol 87(11): 3355–3359.
88. Zhang, H., Wang, J., Yuan, J., Li, L., Zhang, J., Bonami, J. R. and
Shi, Z*. (2006) Quantitative relationship of two viruses (MrNV and
XSV) in white tail disease of Macrobrachium rosenbergii de Man. Dis
Aquat Org 71(1): 11–17.
89. Li, L., Yuan, J., Cai, C., Gu, W. and Shi, Z*. Multiple envelope
proteins are involved in white spot syndrome virus (WSSV) infection in
crayfish. Arch Virol, 2006, 151(7): 1309–1317.

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90. Li, W., Shi Z*., Yu M., Ren W., Smith C., Epstein H. J., Wang H.,
Crameri G., Hu Z., Zhang H., Zhang J., Mceachern J., Field H., Daszak
P., Eaton T.B., Zhang S*., and Wang L. F*. (2005) Bats are natural
reservoirs of SARS-like coronaviruses. Science 310(5748): 676–679.
91. Huang, R., Xie, Y., Zhang, J. and Shi, Z*. (2005) A novel envelope
protein involved in white spot syndrome virus infection. J Gen Virol 86
(5): 1357–1361.
92. Shi, Z., Wang, H., Zhang, J., Xie, Y., Li, L., Chen, X., Edgerton, B. F.
and Bonami, J. R. (2005) Response of crayfish, Procambarus clarkii,
haemocytes infected by white spot syndrome virus. J Fish Dis 28(3):
151–156.
93. Bonami, J. R, Shi, Z., Qian, D. and Sri Widada, J. (2005) White tail
disease of the giant freshwater prawn, Macrobrachium rosenbergii:
separation of the associated virions and characterization of MrNV as a
new type of nodavirus. J Fish Dis 28(1): 23–31.
94. Zhang, S., Shi, Z. and Bonami, J. R. (2004) Purification,

characterization and morphology of a freshwater crab reovirus. J Fish
Dis 27(12): 687–692.
95. Sri Widada, J., Richard, V., Shi, Z., Qian, D. and Bonami, J. R.
(2004) Dot-blot hybridization and RT-PCR detection of extra small
virus (XSV) associated with white tail disease of prawn Macrobrachium
rosenbergii. Dis Aquat Org 58(1): 83–87.
96. Sri Widada, J., Durand, S., Cambournac, I., Qian, D., Shi, Z.,
Dejonghe, E., Richard, V. and Bonami J. R. (2003) Genome-based
detection methods of Macrobrachium rosenbergii nodavirus, a

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pathogen of the giant freshwater prawn, Macrobrachium rosenbergii
dot-blot, in situ hybridization and RT-PCR. J Fish Dis 26(10): 583–590.
97. Shi, Z., Qian, D., Zhang, J., Cao, Z. and Bonami, J. R. (2004)
Isolation, purification and nucleic acid characterization of two viral
particles from freshwater prawn Macrobrachium rosenbergii. Chin J
Virol 20 (1): 58–61. (English abstract).
98. Shi, Z., Xie, Y., Tang, X., Sri Widada, J. and Bonami J.R. (2004)
Nucleic acid detection and partial sequence analysis of
Macrobrachium rosenbergii nodavirus. Chin J Virol 20 (1): 62–66.
(English abstract).
99. Qian, D#., Shi, Z#., Zhang, S., Li, L., Xie, Y. and Bonami, J. R.
(2003) Extra small particles (XSP) and nodavirus associated with
whitish muscle disease in the giant fresh water prawn Macrobrachium
rosenbergii. J Fish Dis, 26 (9): 521–527.
100.Zhang, S., Bonami, Jean-Robert, Shi, Z*. (2003) cDNA library

construction of a Chinese mitten crab reovirus RNA1 and partial
sequence analysis of its RNA polymerase gene. Virol Sinica 18(1): 72–
75. (English abstract).
101.Wang, C., Guo, Y., Cheng, K., Zhao, Y. and Shi, Z*. (2003) The
correlation of host’s growth stage with enlargement of plaque and
absorption rate of cyanophage. Acta Hydrobiol Sinica 27(6): 660–663.
(English abstract).
102.Luo, W., Ju, C., Cheng, K., Zhao, Y. and Shi, Z*. (2003) A
backflushing ultrafiltration technique for concentrating cyanophage.
Virol Sinica 18(4): 397–400. (English abstract).

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103.Xie, Y., Huang, R. and Shi Z*. (2003) Sequence analysis, cloning
and expression of a putative cytokine receptor gene of white spot
syndrome virus. Virol Sinica, 18(4): 362–366. (English abstract).
104.Xie Y., Zhang S., Huang R. and Shi Z*. (2003) A modified
technique for purifying white spot syndrome virus. Virol Sinica 18(4):
391–393. (English abstract).
105.Guo, Y., Cheng, K., Zhao, Y., Wang, J., Wang, C., Shi, Z. and Liu, Y.
(2003) The distribution and infectivity of cyanophage and other algae-
lysin factor in fresh water. China Environ Science 23(2): 167–170.
(English abstract).
106.Cheng, K., Wang, C., Guo, Y., Shi, Z. and Zhao, Y. (2002)
Measurement of lysing cycle and burst size of cyanophage infecting
filamentous cyanobacteria (blue-green algae). Virol Sinica 17(4): 374–
376. (English abstract).
107.Shi, Z. and Zhu, H. (2002) Aquatic crustacean viruses —

Bacilliform viruses. Virol Sinica 17(3): 282–288. (English abstract).
108.Zhang, S., Zhang, J., Huang, C., Bonami, J.R. and Shi Z. (2002)
Preliminary studies on two types of reo-like viruses from crab Eriocheir
sinensis. Virol Sinica 17(3): 264–267. (English abstract).
109.Zhu H., Shi, Z. and Zhao, Y. (2002) Analysis of one gene from
white spot syndrome virus of shrimp. Acta Hydrobiol Sinica 26(5):
560–563. (English abstract).
110.Corbel, V., Zuprizal, Shi, Z., Huang, C, Arcier, J.M and Bonami, J.R.
(2001) Experimental infection of European crustaceans with white spot

Page 62 of 68
syndrome virus (WSSV). J Fish Dis 224: 377–382.
111.Huang, C., Shi, Z., Zhang, L., Xie, Y., Zhang, L., Chen, D. and Wu,
Q. (2001). Homology comparison of white spot syndrome baculovirus
(WSSV) from Penaeid shrimp. Virol Sinica 16: 81–84. (English
abstract).
112.Shi, Z., Huang C., Zhang J., Chen D. and Bonami J.R. (2000).
White spot syndrome virus (WSSV) experimental infection of the
freshwater crayfish Cherax quadricarinatus. J. Fish Dis 23: 285–288.
113.Shi, Z., Durand S. and Bonami J.R. (2000). Screening of DNA

polymerase gene from white spot syndrome virus (WSSV) by using
degenerated oligonucleotides. Virol Sinica 15: 302–307. (English
abstract).
114.Shi, Z., Huang, C., Chen, D., Durand, S. and Bonami J.R. (1998).
Partial cloning of the genome of non-occluded baculovirus from
Penaeus chinensis and preparing the probe for detection. Virol Sinica
13: 263–267. (English abstract).
115.Huang, C., Shi, Z. Zhang, L., Xie, L., Zhang, L., Chen, D. and Wu,
Q. (2000). Study of white spot syndrome baculovirus infection process
in Penaeus monodon by in situ Hybridization. Chin J Virol 16: 242–246.
(English abstract).
116.Zhao, Y., Shi, Z., Huang, G. and Wang, X. (1999). Blue green algae
viruses (cynoaphages). Virol Sinica, 14(2):100–105. (English abstract).
117.Huang, C., Shi, Z. Zhang, J., Zhang, L., Chen, D. and Bonami, J. R.
(1999). Establishment of a model for proliferating white spot syndrome

Page 63 of 68
virus in vivo. Virol Sinica 14: 358–363. (English abstract).
118.Huang, C., Shi, Z., Zhang, L., Wang, B. and Li, H. (1997)
Cytopathic changes of Penaeus chinensis infected by two kinds of
viruses and immunogold labelling. Virol Sinica 12: 171–177. (English
abstract).
119.Huang, C., Zhang, J., Gao, W. and Shi, Z. (1997) Observation and
analysis of histo-and cyto-pathological changes of diseased shrimp
with light and electron Microscopy. Virol Sinica 12 (4): 364–370.
(English abstract).
120.Zhao, Y. and Shi, Z. (1996). Virus and virus-like particles of

eukaryotic algae. Virol Sinica 11(2): 93–102. (English abstract).
121.Shi, Z., Xiao, L. and Chen, D. (1996). Immulogical detection of two

shrimp viruses. Virol Sinica 11: 365–368. (English abstract).
122.Shi, Z., Xiao, L., Gao, W. Zhang, L. and Chen d. (1996).

Immunological detection of two kinds of viruses from Penaeus
chinensis. Virol Sinica 11(4): 368–371. (English abstract).
123.Xiao, L., Shi, Z., Gao, W., Zhang, L., Chen, D. (1995) Isolation,
purification of Penaeus chinensis parvovirus and analysis of its nucleic
acid and protein. Virol Sinica 10: 356–361. (English abstract).
124.Li, Y., Shi, Z. and Chen, D. (1994). A Study on some biochemical

characteristics of Nuclear Polyhedrosis virus of Ectropis grisescens
Warren. Virol Sinica 9(3): 266–271. (English abstract).
125.Shi, Z. Zhang, L. and Chen, D. (1992) Immunity studies on the

Page 64 of 68
Euproctis pseudoconspersa nuclear polyhedrosis virus. Virol Sinica
7(3): 276–282. (English abstract).
Appendix B: Professor Zhengli Shi’s Conference Presentations
Shi, Z. (2018) From SARS to SADS: predict of emerging infectious

diseases. US-China Workshop on Frontiers in Ecology and Evolution of
Infectious Diseases. University of California, Berkeley, June 27–29,
2018.
Shi, Z. (2018) Risk assessment of bat coronavirus spillover and

prevention strategy. Sino-Germany symposium “Globalization-
Challenge and Response for Infectious Diseases” September 5, 2018,
Hamburg, Germany.
Shi, Z. (2018) Coronaviruses associated with human and animal

diseases in China-From SARS to SADS. U.S. China Dialogue on the
Challenges of Emerging Infections, Laboratory Safety and Global
Health Security. January 17, 2018, Galveston, USA.
Shi, Z. (2017) SARS coronavirus may have originated from frequent

recombination events between SARS-related coronaviruses in a single
horseshoe bat habitat. Cell Symposia: Emerging and Re-emerging
Viruses 2017. October 1–3, Arlington, USA.
Shi, Z. (2017) Genetic evolution and interspecies infection of bat

SARS-like coronavirus. International Advisory Board Meeting and
Coronavirus Mini-Symposium for the Theme-based Research Scheme
Project on MERS Coronavirus. September 11–12, Hong Kong.
Shi, Z. (2017) SARS coronavirus may have originated from frequent

Page 65 of 68
recombination events between SARS-related coronaviruses in a single
horseshoe bat habitat. 27th Annual Meeting of the Society for Virology
(Germany). March 22–25, 2017, Marburg, Germany.
Shi, Z. (2016) Prevalence, animal origins and diagnosis of MERS-CoV.

Devising Strategies to Control Emerging Viral Hemorrhagic Fever in
Pakistan. November 14–16, 2016, Lahore, Pakistan.
Shi, Z. (2015) Emerging viral zoonosis in China. Annual meeting of

Sino-Germany Society for Medicine. October 2–3, Berlin, Germany.
Shi, Z. (2015) Bat coronaviruses associated with human diseases.

CAS-NAS Workshop on the Challenges of Emerging Infections,
Laboratory Safety, and Global Health Security. September 29–30,
Beijing, China.
Shi, Z. (2015) The animal origin of SARS coronavirus; from genome to

receptor usage. Annual meeting of Hubei Society for Microbilogy.
August 22–23, Enshi, China.
Shi. Z. (2015) New evidence in support of bat origin of SARS

coronavirus. In “workshop on Coronavirus and Arterivirus, Special
lecture”, ASV2015, July 5–12, London, Canada.
Shi, Z. (2015) The animal origin of SARS coronavirus; from genome to

receptor usage. The 3rd annual “host pathogen interaction in
biodefense and emerging infectious diseases” conference. Feb. 12,
Manassas, Virginia.
Shi, Z et al. (2014) Isolation and identification of bat mammalian

orthoreovirus from Chinese bats. The 6th International Symposium on

Page 66 of 68
Emerging Viral Diseases. October 29–30, Wuhan, China.
Shi, Z, et al., (2013) New evidence further supports bats as natural

reservoirs of SARS coronavirus. The 5th Wuhan International
Symposium on Modern Virology. Oct. 30–31, Wuhan, China.
Shi, Z. (2013) Bat borne viruses. CSIRO-CAS Biosecurity Workshop.

13–15 June 2013, Cairns, Australia.
Shi, Z.(2012) Bat viruses detected in China, 31th annual ASV meeting.
Jul 21–25, Madison, USA.
Shi, Z.(2011) Virome in Bat Intestinal Tract, Implication of Important

Roles Played by Bats in Ecosystem, XVIth International Union of
Microbiological Societies 2. Sep 12–16, Sapporo, Japan.
Shi, Z. (2010) Novel hantavirus detected in Yunnan Red-backed Vole,

Eothenomys miletus. Infectious Disease Genomics and Global Health.
Sep 11–15, Hinxton, UK.
Shi, Z. (2008) Antibodies to Nipah or Nipah-like viruses among bats in

mainland China. The 3rd International Symposium on Emerging Viral
Diseases. Oct. 26–28, Wuhan, China.
Shi Z. (2008) Genetic Evolution of SARS coronavirus. The 179th forum

of Young Scientists of China Association of Science and Technology.
Nov 1–2, Lijiang, China.
Shi, Z, et al. (2008) The angiotension converting enzymes-2 of bats

display different susceptibility to severe acute respiratory syndrome
coronavirus. Annual meeting of Hubei Society for Microbiology. June

Page 67 of 68
26–29, Hohhot, China.
Shi, Z. (2007) Macrobrachium rosenbergii nodavirus (MrNV) and its

associated satellite virus. Aquaculture 2007, Feb. 28- Mar. 2, San
Antonio, USA.
Shi, Z. (2007) Functional analysis of structural envelope proteins of

white spot syndrome virus (WSSV) and prevalence of WSSV and other
shrimp viruses in china — a review. Aquaculture 2007, Feb. 28- Mar. 2,
San Antonio, USA.
Shi, Z. (2007) Evolution on SARS Coronavirus. The First Mexico-China

Scientific Cooperation Conference. Aug. 27–29, Mexico City, Mexico.
Shi, Z. (2006) Bats are natural reservoirs of SARS-like coronaviruses.

France- China Medical Symposium. Oct. 23–24, Paris, France.
Shi, Z. et al. (2006) Genetic diversity of bat SARS-like coronavirus and

its interaction with ACE2. The 8th Session of the International
Congress « Molecular Epidemiology and Evolutionary Genetics of
Infectious Diseases » (MEEGID VIII). Nov. 30 — Dec. 2, Bangkok,
Thailand.
Shi Z. (2006) Biology and molecular genetics of white spot syndrome

virus. Society for Invertebrate Pathology 39th Annual Meeting. Aug. 27
to Sept. 1, Wuhan, China.

Page 68 of 68

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Coronavirus Exposed, Part 1:

Communist Coverup, or Pandemic

The official story about Coronavirus 2019 nCoV is that it “appears to

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Typically not included in most mainstream news stories, however, is

The article refers to statements provided by Dany Shoham, a former

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Thus, it seems rather astute to examine the details of government-

Claims of surprise by Chinese scientists and State officials are

Let’s begin by examining the glaring discrepancies in the official story

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

A concurrent article published in the South China Morning Post on

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

“During the past two decades, three zoonotic coronaviruses have

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

“Angiotensin-I converting enzyme 2 (ACE2) is the receptor for

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

So what happened — did the virology and surrounding scientific

1. Severe acute respiratory syndrome (SARS) is caused by the

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

What is the Wuhan Institute of Virology’s National Biosafety

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

At any rate, the forgoing storyline is the official word on Coronavirus

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

What’s odd is that despite completing the decade-long construction

UPDATE — JANUARY 29, 2020: What’s also odd, and outright

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Whether Wuhan Institute of Virology actually remains at its original

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Wuhan Institute of Virology is also connected with the recent, major

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Similarly, and perhaps connected, is the recent indictment of Charles

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

In short, although there are actually two laboratories in Wuhan linked

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Who is Professor Zhengli Shi and what is her relevance to Wuhan

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Notably, starting in 2014, Prof. Zhengli began to win particularly large

On top of these lucrative American grants she concurrently received

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

“Prof. Zhengli Shi ’s researches focus on the molecular

Unsurprisingly, Prof. Zhengli has been featured as a key presenter at

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Prof. Zhengli has been or is currently a professional member of the

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

Chinese scientists, researchers, and doctors examining the emergent

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

“Coronavirus spike protein mediates viral entry into cells by first

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

And immediately after this study was published — literally the

Let’s say that’s just a coincidence Prof. Zhengli published a study

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

In a letter to the editor of SCIENCE CHINA Life Sciences published in

In that same month, the results of a study Prof. Zhengli conducted,

In 2016, one of the Directors at Wuhan Institute of Virology posted the

https://medium.com/@siradrianbond/coronavirus-2019-ncov-part-1-d6a338eed7c5 1/31/20, 8:51 PM

In a study conducted in September of 2015, Two Mutations Were