Académique Documents
Professionnel Documents
Culture Documents
Cardiovascular
Examination
2
Anatomy
3
4
nspection
1 Precardial projection
and excavation
2 Apical impulse
3 Abnormal pulsations
of precardium
5
1 Precardial projection and
excavation
1) Precardial projection
congenital heart disease: tetralogy of
Fallot
Valvular heart disease--
MS,PS
pericardial effusion (large , childhood)
nspection
2 Apical impulse
`Normal:
position-the fifth left intercostal
space 0.5-1.0cm medial to the
midclavicular line
range-2.0-2.5cm in diameter
nspection
`Abnormal
1) Location
#diaphragm:
~transverse position upper,outward
obesity ,child, pregnacy;
ascites; tumor of abdominal cavity
10
one side pleural effusion or
pneumothorax-to the healthy
side
one side atelectesis or pleural
adhesion-to the affected
#mediastinum:
11
#enlargement of the heart
right ventricular dilatation -left or
slightly upper
left ventricular dilatation-left
inferior
LV &RV dilatation -left inferior
(both side dilatation)
12
13
#Posture:
recumbent position-upper
left lateral position-to the left 2-
3cm
right lateral position-to the right
1.0-2.5cm
Dextrocardia: 5-ICS-RS
14
Decrease Increase
Physiological Chest wall pachynsis
Narrow intercostol space
Thin chest wall
Broaden intercostol space
exercise,euphoric mood
Pathological . myopathy(AMI,DCM)
. pericardial eIIusion
. emphysema
.constrictive pericarditis
. leIt side massive pleural
eIIusion or
pneumothroax
.LV hypertrophy
.hyperthyroidism
. Iever
.anemia
2)Intensity and extent changes
nspection-
apical impulse - abnormal
15
3)Inward impulse:
apex excavation in the systole
seen: adhensive pericarditis
prominent RV hypertrophy
nspection
-apical impulse - abnormal
1
nspection
1)left third-forth intercostal space
lateral to the sternum(3,4ICS-LS)
seen: RV hypertrophy
3 Abnomal pulsations of
percardium
1
2)hypoxiphoid process
seen:
difference deep inspiration
RV hypertrophy
abdominal aorta (aneurysm)
1
3)basal part of the heart
2 ICS-LS: dilatation of the
pulmonary artery or pulmonary
hypertensin, occasionally healthy
young man
2 ICS-RS: aneurysm of aortic arch
or dilatation of ascending aorta
1
Palpation
1 Apical impulse and
pulsation of precardium
2 %hrill
3 Pericardial friction rub
20
1 Apical impulse and
pulsation of precardium
Exact position of apex
%he beginning of systole of
ventricle first sound
Heaving apex impulse: reliable
of LV hypertrophy
Palpation
21
2 %hrill
One of characteristic signs of organic heart disease.
Mechanism : the flow of bloodnarrowed
orificevortices
vibrationchest wall
thrill-high frequency
murmurs-low frequency
Method:position,phase of cardiac cycle,clinical
significance
seen: CHD or valvular stenosis ,
occasionally insurficiency
22
Clinical signiIicance oI thrill
Location phase Disease
2 ICS-RS Systole AS (RHD,CHD,senile)
2 ICS-LS Systole PS (CHD)
3,4 ICS-LS Systole VSD (CHD)
Apex Systole MI (severe)
Apex Diastole MS (RHD)
2 ICS-LS Continous PDA
CHD:congenital heart disease
23
1)Precardium-4
th
ICS-LS
2) both phases of the cardiac cycle
3) systolic period, sitting erect and leaning
forward, the end of expiration
4)mechanism: rub of the visceral and
parietal layers of pleura
5)seen:acute pericarditis
3 Pericardil friction rub
24
Percussion
Aim:to determine the size and
shape of the heart .
Absolute dullness: contain no gas
Relative dullness : real size
25
1 murneuver of percussion
patient in erect position -the
pleximeter is vertical with the
intercostal space
patient in the recumbent position
-the pleximeter is parallel with the
intercostal space
2
2 order :
left-right ; upwards ; inward
left margin : from 2-3 cm lateral to
the apex beat up to the 2
nd
ICS
right margin : one intercostal space
higher than the border of liver
dullness up to the 2
nd
ICS
size: vertical distance from margin
to the anterior midline
2
2
3 Normal heart borders
(area oI relative dullness)
Right(cm) ICS LeIt(cm)
2~3
2~3
2~3 III 3.5~4.5
3~4
5~
~
(LMCLML:~10cm)
Percussion
2
4 The composition oI various parts oI
the border oI the heart
(1)
Right ICS LeIt
SVC,SA II PA
RA III LA
RA
LV
LV
Percussion
30
(2)%he upper border -the lower
border of the anterior end of the
third rib
(3)%he basal part -the second
intercostal space upward
left: aortic node and PA
(4)Concave part -between the aorta
and the left ventricle
31
5 Changes in the area of cardiac
dullness and its significance
Cardiac factors :
1)LV enlargement: ~boot shape
Seen:aortic valvular disease ,
hypertension heart disease
Percussion
32
2)RV enlargement :
slightly--absolute dullness
Prominent--relative dullness
to the left side prominently
Seen:PHD, MS
3)%wo ventricle :
~generally enlarged heart
seen:DCM , Kashan cardiomyopathy
33
4)LA and/or pulmonary artery:
LA:concave part disappear
LA+PA:2,3 ICS-LS outwards
~pear shape
Seen: MS--- ~mitrial type
34
5)pericardial effusion: enlargement of
both sides of the border
bodys position.
recumbent positionwidening of base of
the heart
erect position:~triangular shape
35
)dilatation of the aorta /ascending
aortic aneurysm:
widening if the dull area of first and second
intercostal space (with systolic pulsation)
3
Extacardial factors :
1)large pleural effusions and
pneumothorax to the healthy side
2)atelectasis /pleural pachynsis to the
affected
3)a large amount of ascites or big
abdominal tumor:
diaphragm elevatedtransverse
position left side enlargement
3
Ausclutation
3
1 Ausclutatoty valve areas
1)ausclutatory mitral area: apical area
2)ausclutatory pulmonary area:2 ICS-
LS
3)ausclutatory aortic area: 2 ICS-RS
4)second ausclutatory aortic area: 3
rd
ICS-LS-Erb area
5)tricuspid area :4,5 ICS-LS
3
40
2 Order:
MV---PV---AV1---AV2---%V
3 Contents : 1) rate 2)rhythm
3)heart sound 4)extra heart sound
5)murmurs )pericardial friction
sound
41
1)heart rate:
0~100bpm FM
child (3 years) 100bpm
tachycardia: normal adult 100bpm
child(3 years) 150bpm
bradycardia: HR 0 bpm
42
Ausclutation
heart rate:0-100bmp
43
2)cardiac rhythm:
`sinus arrythmia-affected by breath
`premature beat:
classification:atrial~ ventricular ~
junctional ~
frequently: bpm
occasionally: bpm
bigeminy trigeminy
44
`atrial fibrillation:
absolute irregular rhythm
S1 intensity inequality
Pulse deficit
seen:MS,CHD,hyperthyroidism,
PHD,DCM
45
Ausclutation
atrial Iibrillation
4
Cycle Nature Duration Site Mechanism
S1 Isovolumetric
contraction phase
Blunt
0.1
Apical
area
Closure oI the
MV and TV
S2 Isovolumetric
relaxation phase
Distinct
0.0
Basal
part
Closure oI the
AV and PV
S3 The end oI
ventricular rapid
Iilling phase
Weak
Blunt
0.04
aIter S2
0.12~0.1
Apex
(inner-
upper)
Ventricular
vibration
S4 The end oI
ventricular
diastolic phase
Weak
0.1
Iorward S1
Apex Atrium
contraction
3) cardiac sound
4
Ausclutation
content
cardiac sound
S1
S2
4
4
4)Abnormal cardiac sound
`Intensity:
position of the atrioventricular
valve
Ventricular contractility and
output
Valvular integrity and activity
50
S1: Accentuation:
MS
HRcontractility
fever,anemia,hyperthyroidism
complete AVB cannon sound
51
52
S1 attenuation :
MI
P-R interval enlong
AI
myocarditis,myopathy,MI,HF
inequality: af, IIIgAVB
53
54
S2---A2,P2
S2 ---pressure and flow of
blood
A2 : hypertensin, arterisclerosis
P2 : PHD,CoHD(L--R),LVF
S2 ---pressure flow
Seen:hypotension,AS/AL,PS/PI
55
5
`Quality
mono rhythm
pendular rhythm---embryocardia
`Splitting of heart sound
S1 splitting:
seen-RBBB, right heart failure
Ebetein malformation ,MS
LA myxoma
5
5
S2 splitting:
(1)physiological splitting :end of
inspiration
(2)general splitting : most commonly
seen: CRBBB, PS, MS,MI ,VSD
(3)fixed splitting :ASD
(4)paradoxical splitting(reversed
splitting) :pathological
seen: CLBBB ,AS, hypertension
5
0
5)extra cardiac sound
Diastolic period
1)gallop rhythm:
--protodiastolic gallop: S1+S2+S3 the third
sound gallop (sign of organic heart disease)
seen : HFAMI, severe myocarditis ,
myopathy etc.
-- late diastolic gallop: atrial gallop S1+S2+S4
seen : HBP ,HCM ,AS ,CHD
-- summation gallop: quadruple rhythm
seen:HF,cardiomyopathy
1
2
3
5) extra cardiac sound
Diastolic period
2)opening snap:MS
3)pericardial knock: constrictive
pericarditis
4)tumor plop: LA myxoma
4
5
Ausclutation
CONTENT
Tumor plop
Systolic period
(1)early systolic ejection sound(click)
pulmonary :pulmonary hypertension;
pulmonary artery dilatation
PS, ASD, VSD
Aortic: hypertension, aneurysm ,
AS, AI ,aorta constriction
(2)mid and late systolic click:
S1----mid0.08" late0.08"
seen: mitral prolapse
iatrogenic
(1)prosthetic valvular sound
(2)pacemaker
0
)cardiac murmurs
*Mechanism:
acceleration oI blood Ilow
stenosis oI valvular oriIice
or great vessles turbulent Ilow
valvular insuIIiciency vortices
abnormal passage
Ioreign body
dilatation oI vessles(aneurysm)
1
2
`characterization of murmur and
ausclutatory key points
(1)location:L3,4 -VSD L2,3-PDA
(2)transmission:
MI ---left axilla AS---neck
(3)phase: systolic murmurs
diastolic ~
continuous ~
biphasic ~
early,mid,late,whole
murmurs
3
(4)quality: blowing-MI
rumbling-MS
sighing--AI
machinery--PDA
(5)intensity :Levine grade classification
shape: crescendo---MS
decrescendo---AI
crescendo-decrescendo---AS
continuous---PDA
regular---MI
murmurs
4
() others:
body position:
MS--left lateral position
AI--sitting erected and forward
MI,%I,PVS--lie on one` back
Lie stand: HCM
breath:expiration--LV murmurs
inspiration --RV murmurs
valsalva--HCM
exercise: HR--murmurs
murmurs
5
clinical significance murmurs:
functional and organic
7)pericardial friction sound:
both phases , unaffected by
respiration .
seen: pericarditis ,
RHD ,AMI ,renal failure, SLE
0
` clinical significance of cardiac
murmurs
systolic murmurs
pulmonary physiology
relativeMSASD
organicPS
TVrelative RV enlarged
organic rare
1
` clinical significance of cardiac
murmurs
Diastolic murmurs
MV:organic:RHD(MS)
relative:AI(severe)
Austin Flint murmur
AV:AI
2
3
4
` clinical signiIicance oI cardiac
murmurs
Diastolic murmurs
PV:organic murmur is rare
PI(dilatation oI pulmonary artery)
MSP2 ---- Graham Steell murmur
TV:rare
5
` clinical signiIicance oI cardiac
murmurs
continuous murmurs
PDA
innocent murmur
'ascular examination
The second clinical hospital of CNU
pulse
pulse rate
pulse rhythm
tensions and state oI arterial
wall
intensity
pulse wave
pulse
pulse rate
Atrial Iibrillation and Irequent premature
beat stroke volume peripheral artery
no pulse pulse rate less than HR(pulse
deIicit)
pulse
pulse rhythm
pulse deIicit
bigeminal pulse,trigeminal pulse
dropped pulse
0
pulse
tensions and state oI arterial wall
Artery tension depending on blood
pressure (mainly SBP).
Judge state oI artery wall
1
pulse
intensity
Bownding pulse
seen:high Iever, hyperthyroidism, AI
Microsphygmia
seen:HF,AS and shock
2
pulse
pulse wave
normal pulse wave
composed oI upstrokeknocking wave
peak tide waveand downstroke
dicrotic wave
3
pulse
pulse wave
water hammer pulse seen:AI,hyperthyroidism,PDA,
severe anemia
pulse tardus seen:AS
dicrotic pulse seen:HCM
pulsus alternans seen:HBP,AMI,AI
paradoxical pulse
seen:cardiac tamponade,constrictive pericarditis
Pulseless
seen:serious shock, arteritis
4
blood pressure
method oI measurement
direct measurement method
indirect measurement method
5
blood pressure
standard
deIinition oI Bp level and classiIication(older than 1 years old)
classiIication SBP(mmHg) DBP(mmHg)
Ideal BP 120 0
Normal BP 130 5
High limit oI BP 130-13 5-
Grade 1mild 140-15 0-
subgroup
boundline hypertension 140-14 0-4
Grade 2moderate 10-1 100-10
Grade 3severe _ 10 _110
Simple systolic hypertension 140 0
subgroup
boundline systolic hypertension 140-14 0
blood pressure
clinical signiIicance oI BP changes
hypertensionhigher than 140/0mmHg Ior 3 times not in
the same day
hypotensionlower than 0/0-50mmHg
Shock,,MI,acute cardiac tamponade
obvious diIIerence between bilateral upper limbsmore than
10mmHg---arteritis,congenital artery malIormation
diIIerence between upper and lower limbslower limb BP is
20-40mmHg higher than upper one normally
pathological:constrictive aorta ,arteritis(chest-abdominal aorta)
change oI pulse BP
40mmHgwide pulse BP---hyperthyroidism,AI
30mmHgnarrow pulse BP---AS,pericardial eIIusion
blood pressure
dynamic BP monitoring
Average BP Ior 24h 130/0mmHg;
bright day 135/5mmHg;
night: 125/5mmHg
Peak:am10am,4pmpm