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PATHOPHYSIOLOGY OF BURN

ENTRY OF MICROORGANISM

SEPTICEMIA

LOSS OF FLUID SKIN LOSS/ DESTRUCTION LOSS OF HEAT

HYPOVOLEMIA

HYPOTHERMIA

IMMUNOSUPPRES ION

SUSCEPTIBILITY TO INFECTION RELEASE OF NEUROPEPTIDES AND ACTIVATION OF COMPLEMENT DIRECT FLUID LOSS THIRD SAPCE COLLECTION (NON-BURN TISSUE OEDEMA) CIRCULATORY SHOCK NEUTROPHILS ATTRACTION AND GRANULATION

INFLAMMATION

PAIN AND ALTERATION OF PROTEIN

DEGRANULATION OF MAST CELL

RELEASE OF FREE RADICALS AND PROTEASE

FURTHER DAMAGE TO TISSUE

BURN CIRCULATORY CHANGES

LOSS OF SKIN

INCREASE VESSEL PERMIABILITY

KIDNEY

LUNGS OTHER CHANGES STOACH

INTESTINE

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