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Definition
The term birth injury is used to denote: avoidable and unavoidable mechanical, hypoxic and ischemic injury affecting the infant during labor and delivery.
Causes
Birth injuries may result from : 1.Inappropriate or deficient medical skill or attention. 2.They may occur, despite skilled and competent obstetric care.
Incidence
Has been estimated at 2-7/1,000 live births.
Predisposing factors:
1. Macrosomia, 2. Prematurity, 3. Cephalopelvic disproportion, 4. Dystocia, 5. Prolonged labor, and 6. Breech presentation.
Incidence
5-8/100,000 infants die of birth trauma, and 25/100,000 die of anoxic injuries; Such injuries represent 2-3% of infant deaths.
Cranial Injuries
Of facial or scalp soft tissues may be seen after forceps or vacuumassisted deliveries. Their location depends on the area of application of the forceps.
Subconjunctival ,retinal hemorrhages and petechiae of the skin of the head and neck
All are common. All are probably secondary to a sudden increase in intrathoracic pressure during passage of the chest through the birth canal. Parents should be assured that they are temporary and the result of normal hazards of delivery.
Moulding
Moulding of the head and overriding of the parietal bones are frequently associated with caput succedaneum and become more evident after the caput has receded but disappear during the first weeks of life. Rarely, a hemorrhagic caput may result in shock and require blood transfusion.
Caput succedaneum
Diffuse, sometimes ecchymotic, edematous swelling of the soft tissues of the scalp involving the portion presenting during vertex delivery. It may extend across the midline and across suture lines. The edema disappears within the first few days of life.
Caput succedaneum
Analogous swelling, discoloration, and distortion of the face are seen in face presentations. No specific treatment is needed, but if there are extensive ecchymoses, phototherapy for hyperbilirubinemia may be indicated.
Cephalhaematoma
It is a subperiosteal haematoma most commonly lies over one parietal bone. It may result from difficult vacuum or forceps extraction .
Cephalhaematoma
Management: - It usually resolves spontaneously.
Cephalohematoma
Is a subperiosteal hemorrhage, so it is always limited to the surface of one cranial bone. There is no discoloration of the overlying scalp, and swelling is usually not visible until several hours after birth, because subperiosteal bleeding is a slow process. An underlying skull fracture, usually linear and not depressed, is occasionally associated with cephalohematoma.
Subgaleal hemorrhage
Subgaleal hemorrhage is bleeding into the subgaleal compartment . This injury occurs as a result of pressure through the head ( of the infant) into the pelvic outlet. It is commonly occurred after vacuum delivery. The early detection is so vital .
Serial head circumferences may detect any increase due to hemorrhage . The bleeding may extend to the posterior aspect of the ear and neck. Monitoring of the bleeding times and coagulation is important. Assessment to the level of consciousness. Assessment to the level of Hb and Hct. Increase in billirubin is expected due to blood lyses.
Fracture Skull:
Usually occurs due to difficult forceps delivery. It may be: (1) Vault fracture: affecting the frontal or parietal bone. may be linear or depressed fracture. no treatment reqd unless there is intracranial haemorrhage. (2) Fracture base of skull: Usually associated with intracranial haemorrhage.
Fractures of the skull Fracture of the Occipital bone almost causes fatal hemorrhage due to disruption of the underlying vascular sinuses. It may result during breech deliveries from traction on the hyperextended spine of the infant with the head fixed in the maternal pelvis.
Intracranial Haemorrhage:
Causes:
1. Sudden compression and decompression of the head as in breech and precipitate labour. 2. Marked compression by forceps or in cephalopelvic disproportion. 3. Fracture skull.
Intracranial Haemorrhage:
Predisposing factors:
1. Prematurity due to physiological hypoprothrombinaemia, fragile blood vessels and liability to trauma. 2. Asphyxia due to anoxia of the vascular wall . 3. Blood diseases.
Intracranial Haemorrhage:
Clinical manifestation
1- Altered level of consciousness. 2- Flaccidity muscle tone 3- Breathing -irregular and periodic, gasping or apnoeic 4- Pupils may react sluggishly to light, fixed and dilated no eyes movement, 5- Opisthotonus, rigidity, twitches and convulsions. 6- Vomiting -projectile 7- High pitched cry. 8- Anterior fontanelle is tense and bulging.
Intracranial Haemorrhage
Investigations:
1. 2. 3. 4. Ultrasound is of value. CT scan is the most reliable. MRI Lumbar puncture-blood stained CSF
CLINICAL MANIFESTATIONS
Incidence of IVH increases with decreasing BWT:
CLINICAL MANIFESTATIONS
CLINICAL MANIFESTATIONS
1. 2. 3. 4. 5. 6. Periods of apnea, Pallor, or cyanosis; Failure to suck well; Abnormal eye signs; A high-pitched cry; Muscular twitches, convulsions, decreased muscle tone, or paralyses; 7. Metabolic acidosis; shock, and a 8. Decreased hematocrit or its failure to increase after transfusion may be the first indications. 9. The fontanel may be tense and bulging.
DIAGNOSIS
Intracranial hemorrhage is diagnosed; 1. History, 2. Clinical manifestations, 3. Transfontanel cranial ultrasonography 4. Computed tomography (CT) 5. Lumbar puncture
PROGNOSIS Neonates with: ( massive hemorrhage associated with tears of the tentorium or falx cerebri) rapidly deteriorate and may die after birth.
PREVENTION The incidence of traumatic intracranial hemorrhage may be reduced by: judicious management of cephalopelvic disproportion and operative delivery.
PREVENTION
Fetal or neonatal hemorrhage due to: 1. Maternal idiopathic thrombocytopenic purpura (ITP) or 2. Alloimmune thrombocytopenia may be prevented by maternal treatment with: Steroids, Intravenous immunoglobulin Fetal platelet transfusion.
PREVENTION The incidence of IVH may be reduced by antenatal steroids and by postnatal administration of low-dose indomethacin. Vitamin K should be given before delivery to all women receiving phenobarbital or phenytoin during the pregnancy.
TREATMENT
Seizures are treated with anticonvulsant drugs. Anemia-shock, requires transfusion with packed red blood cells or fresh frozen plasma. Acidosis is treated with slow administration of sodium bicarbonate.
TREATMENT
Symptomatic subdural hemorrhage in large term infants should be treated by removing the subdural fluid collection by means of a spinal needle placed through the lateral margin of the anterior fontanel.
1. Areflexia, 2. Loss of sensation, and 3. Complete paralysis of voluntary motion Occur below the level of injury
(1)Erb's palsy:
1. Injury to C5 and C6 roots. 2. The upper limb drops beside the trunk, internally rotated with flexed wrist (policemans or waiters tip hand).
- less common, - Injury to C7 and C8 and 1st thoracic roots. - It leads to paralysis of the muscles of the hand and weakness of the wrist and fingers' flexors.
BRACHIAL PALSY
Injury to the brachial plexus may cause paralysis of the upper arm with or without paralysis of the forearm or hand or, more commonly, paralysis of the entire arm. Approximately 45% are associated with shoulder dystocia.
BRACHIAL PALSY
occur in : 1.Macrosomic infants and when lateral traction is exerted on the head and neck during delivery of the shoulder in a vertex presentation, 2. When the arms are extended over the head in a breech presentation, 3.When excessive traction is placed on the shoulders.
1 2 3 4 5 6 9 8 7
Roots Trunks
Cords Nerves
1 2 3
4 5 6
7 8 9
In Erb-Duchenne paralysis
The injury is limited to the 5th and 6th cervical nerves. The characteristic position consists of:
( Adduction and internal rotation of the arm with pronation of the forearm).
Moro reflex is absent on the affected side
In Erb-Duchenne paralysis
There may be some sensory impairment on the outer aspect of the arm. The power in the forearm and the hand grasp are preserved unless the lower part of the plexus is also injured;
Klumpke's paralysis
Is a rarer form of brachial palsy; Injury to the 7th and 8th cervical nerves and the 1st thoracic nerve produces a paralyzed hand, (Horner syndrome)
If the sympathetic fibers of the 1st thoracic root are also injured : paralyzed hand and ipsilateral ptosis and miosis.
Klumpke's paralysis
The mild cases may not be detected immediately after birth. Differentiation must be made from : 1. Cerebral injury; 2. Fracture, dislocation, or epiphyseal separation of the humerus; 3. Fracture of the clavicle. MRI demonstrates nerve root rupture or avulsion
common
edema and hemorrhage
uncommon
Laceration
The prognosis
Depends on whether the nerve was merely injured or was lacerated. If the paralysis was due to edema and hemorrhage about the nerve fibers, function should return within a few months; If due to laceration, permanent damage may result.
The prognosis
Involvement of the deltoid is usually the most serious problem and may result in a shoulder drop secondary to muscle atrophy. In general, paralysis of the upper arm has a better prognosis than paralysis of the lower arm.
Treatment
Partial immobilization and appropriate positioning to prevent development of contractures. In upper arm paralysis: the arm should be abducted, with external rotation at the shoulder and with full supination of the forearm and slight extension at the wrist with the palm turned toward the face.
Treatment In lower arm or hand paralysis: the wrist should be splinted in a neutral position and padding placed in the fist.
Gentle massage and range of motion exercises may be started by 7-10 days of age.
Treatment
If the paralysis persists without improvement for 3-6 months:
neuroplasty, neurolysis, end-toend anastomosis, or nerve grafting offers hope for partial recovery.
PHRENIC NERVE PARALYSIS Phrenic nerve injury (3rd, 4th, 5th cervical nerves) with diaphragmatic paralysis must be considered when cyanosis and irregular and labored respirations develop.
Such injuries, usually unilateral, are associated with ipsilateral upper brachial palsy.
Recovery usually occurs spontaneously by 1-3 months; rarely, surgical plication of the diaphragm may be indicated.
Manifestations: There is paresis of the facial muscles on the affected side with: Partially opened eye and: Flattening of the nasolabial fold. The mouth angle is deviated towards the healthy side.
The prognosis depends on whether the nerve was injured by pressure or whether the nerve fibers were torn. Care of the exposed eye is essential.
Improvement occurs within few weeks. Neuroplasty may be indicated when the paralysis is persistent.
Other peripheral nerves are seldom injured in utero or at birth except when they are involved in fractures or hemorrhages.
V) VISCERAL INJURIES
(Liver, spleen and kidney) may be injured in breech delivery which should be avoided by holding the fetus from its hips.
Adrenal hemorrhage
Occurs with some frequency, especially after breech delivery in LGA infants or infants of diabetic mothers. 90% are unilateral; 75% are right sided. The symptoms are profound shock and cyanosis If suspected, abdominal ultrasonography may be helpful, and treatment for acute adrenal failure may be indicated
Fractures
BONE INJURIES
usually occur during difficult breech delivery.
(A) Vertebral Column Injuries:
These are fatal if associated with spinal cord transection above C4 ,due to diaphragmatic paralysis.
CLAVICLE
-fractured during labor and delivery
CLAVICLE
The infant characteristically does not move the arm freely on the affected side; Crepitus and bony irregularity may be palpated, and Discoloration is occasionally visible over the fracture site.
CLAVICLE
Treatment, consists of immobilization of the arm and shoulder on the affected side. A remarkable degree of callus develops at the site within a week and may be the first evidence of the fracture. The prognosis is excellent.
EXTREMITIES
In fractures of the long bones, spontaneous movement of the extremity is usually absent. The Moro reflex is also absent from the involved extremity. There may be associated nerve involvement.
EXTREMITIES (Humerus)
Satisfactory results of treatment for a fractured humerus are obtained with 2-4 wk of immobilization (during which the arm is strapped to the chest). A triangular splint and a bandage are applied, or a cast is applied.
EXTREMITIES
Healing is usually accompanied by excess callus formation. The prognosis is excellent for fractures of the extremities. Fractures in preterm infants may be related to osteopenia
MUSCLE INJURIES
Strenomastoid injury Exaggerated lateral flexion of the neck leading to torticollis and swelling in the muscle. It is usually improved within 2 weeks but permanent torticollis may continue.