Académique Documents
Professionnel Documents
Culture Documents
INTRODUCTION
DETERMINANTS OF BURN
Causative agent Depth Extent/luasnya Location Age Concomitant injury Preexisting diseases
CAUSATIVE AGENT
Thermal
Flame Flash Scald Contact
DEPTH
First-degree (Superficial)
Erythema Blanches on pressure Pain
Second-degree (Partial-thickness)
Red, shiny, wet Vesicles Swelling Very painful
DEPTH
Third-degree (Full-thickness)
Color : waxy white, gray Charred/terbakar hangus Leathery/strukturnya kasar Dry Brownish streaks Do not blanch on pressure Pain free Sunken appearance
DEPTH
DEPTH
First-degree (Superficial)
First-degree (Superficial)
Second-degree (Partial-thickness)
Second-degree (Partial-thickness)
Third-degree (Full-thickness)
Third-degree (Full-thickness)
EXTENT
Rule of nines The Lund and Browder chart The palmar surface of the adults hand 1% of the TBSA
Rule of Nines
LOCATION
Face Neck Hands Feet Joints Perineum Circumferential burn
Full thickness : <2% TBSA Does not involve special care areas (eyes, ears, face, hands, feet, perineum) Excludes electrical injury, inhalation injury, complicated injury (fracture), all poor-risk patients (extremes of age, intercurrent disease)
Full thickness : <10% TBSA Does not involve special care areas Excludes electrical injury, inhalation injury, complicated injury, all poor-risk patients
Full thickness : >10% TBSA Involve special care areas Includes electrical injury, inhalation injury, complicated injury, all poor-risk patients
Smaller burns in patients not able to care for the burn, which if left unattended, pose a significant potential for development of burn infection :
Drug abusers Mentally ill Homeless Patients hospitalized at other institutions who experience a serious burn Unreliable home environment for small children
Acute massive skin loss syndromes requiring burn center quality of care (e.g. Steven-Johnson syndrome / toxic epidermal necrolysis, large traumatic degloving injuries)
PATHOPHYSIOLOGY
Zone of Stasis
Area surrounding zone of coagulation Inflammation, decreased blood flow
Zone of Hyperemia
Peripheral area of burn Limited inflammation, increased blood flow
PATHOPHYSIOLOGY
PATHOPHYSIOLOGY
PATHOPHYSIOLOGY
PATHOPHYSIOLOGY
THERAPY
Initial : ABCs Special priority : stop burning process Airway control & ventilation
THERAPY
Measurement of arterial blood gases & carbon monoxide level Indications for endotracheal intubation:
Supraglottic edema & inflammation on bronchoscopy Progressive hoarseness or air hunger Coma or respiratory depression Acute respiratory distress Full-thickness burns of face or perioral region Circumferential neck burns
THERAPY
PARKLAND FORMULA
PARKLAND FORMULA
THERAPY
Assessment parameters
Urine output : 30 50 cc / hr Cardiopulmonary factors
Blood pressure (systolic 90 100 mmHg) Pulse rate ( 100 / minute) Respiration (16 20 / minute)
THERAPY
THERAPY
Other considerations
Nasogastric intubation Analgesia Systemic antibiotics Tetanus toxoid +/- hyperimmune human globulin
COMPLICATIONS
Organ system Respiratory Cardiovascular Major complications Inhalation injury, pulmonary edema, pneumonia, ARDS, embolism, sinusitis Myocardial infarction, endocarditis, pericarditis, arrhythmias, hypertension, septic thrombophlebitis, embolism, thrombosis, ruptured vessel wall Renal failure, hematuria,myoglobinuria DM, adrenal insufficiency, adrenal hemorrhage Encephalopathy, CO poisoning
KOMPLIKASI
Gastrointestinal Curling ulcer, hepatic dysfunction, paralytic ileus, acalculous cholecystitis, pancreatitis, mesenteric occlusion, superior mesenteric artery syndrome, peritonitis Hematologic Anemia, coagulopathies Immunologic Sepsis, immunodeficiency Metabolic-Nutritional Prolonged catabolism, starvation Musculoskeletal Contracture, loss of limb Skin Infection, hypertrophic scar, contracture