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Block Objectives
Diagram the cardiac cycle, relating electrical and mechanical events to chamber and great artery pressures and ventricular volume; identify systole and diastole Describe the mechanism of production of normal and abnormal heart sounds: relate them to the electrical and pressure events of the cardiac cycle
Objectives
Describe the use of the stethoscope
Auscultatory areas Use of bell and diaphagm Interpret the meaning of the auscultatory findings
Resources
Lilly 5th edition Chapter 2 pp. 28-36, Table 2.2 P. 42 Blaufuss.org. heart sound tutorial quiz There will be auscultation practice sessions in week 7 The Blaufuss Sound Builder program used in the practice sessions is available in the App store for a fee
Always Keep In Mind-- The history and physical examination of the cardiovascular system make the vast majority of the diagnoses!!!!!!!
Auscultation Requires.
A quiet environment A stethoscope with a bell for low pitched sounds and diaphragm for high pitched sounds, well fitting ear pieces and short tubing A thinking auscultator who knows where to place the stethoscope to hear specific sounds-based on anatomic relationships of the heart and great vessels to chest wall landmarks
Auscultatory Areas
Auscultatory Areas
Aortic
Pulmonic
Aortic 2nd ICS RSB Pulmonic 2nd ICS LSB Tricuspid LLSB Mitral Apex 5th ICS MCL
Tricuspid
Mitral
A Thinking Auscultor
Can describe the events in the cardiac cycle responsible for the production of the normal and abnormal heart sounds. can reproduce the diagram below relating electrical events, mechanical events and heart sounds. This cardiac cycle diagram will be the reference for the discussion of all of the heart sounds.
Cardiac Cycle
ECG Electrical activation causes chamber contraction which raises pressure. Chamber relaxation and emptying cause pressure to fall
Cardiac Cycle
LV 140/12 Ao 140/90 RV 30/8 PA 30/10 LA (12) RA (8) Chamber and Arterial Pressures
ECG Left heart pressures and velocities are higher than in the right heart, so the contributions to S1 and S2 are louder, higher in frequency
Mitral valve closes Tricuspid valve closes Ventricular depolarization causes ventricular contraction. S1 occurs when ventricular pressure exceeds atrial pressure. Mitral closes before tricuspid
LV and RV pressures drop below aortic and PA pressures after ejection and relaxation. A2 is louder, best at 2RSB; normal P2 only heard at 2LSB
Increased A2, P2
Arterial hypertension Mobile leaflets with increased mass Increased pulmonary blood flow (P2)
Decreased A2,P2
Decrease mobility in severe valve stenosis Calcification of aortic valve may restrict mobility so severely that A2 is absent
Normal Splitting of S2
A2 and P2 normally superimposed in expiration Inspiration delays P2 due to..
Negative intrathoracic pressure increases right heart filling. The RV takes longer to eject the increased volume Negative intrathoracic pressure increases pulmonary capacitance, delaying the back flow to close the pulmonic valve
Abnormal Splitting of S2
Widened splitting electrical or mechanical delay of the RV delays P2. Split S2 in expiration widens with inspiration Fixed splitting atrial septal defect continuously increases right heart filling, delaying P2, unaffected by respiration Paradoxical splitting electrical or mechanical delay in LV delays A2 so it follows P2. P2 moves out to meet A2 with inspiration
Ejection Clicks
Aortic valve opens
Audible opening of mobile congenitally stenotic aortic or pulmonic valves, also dilated aorta or PA
S1
EC
S2
Nonejection Clicks
S1
SC
A2
Prolapsing mitral or tricuspid valve leaflets reach limit of motion and tense later in systole
Opening Snaps
S2 S1 OS
Audible opening of mobile stenotic mitral or tricuspid valves; high pitched at LLSB or apex
S2 S1 S3
Dilated ventricle with poor function and/or increased early diastolic filling
LV early diastolic filling S3 RV early diastolic filling S3
In late diastole before S1. Low pitched with the bell LLSB, apex
S4 S1
S2
Atrial contraction S4
EC
P2 S3
S4
S1
SC
A2 OS