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Chief complaint: Chest pain the pain was felt a day ago after coming from the garden, before admitted to the hospital. During the attack, patient feel sweating, nausea, vomit (-), palpitations (-), shortness of breath (-)
Chief complaint: Chest pain the pain was felt a day ago after coming from the garden, before admitted to the hospital. During the attack, patient feel sweating, nausea, vomit (-), palpitations (-), shortness of breath (-)
Chief complaint: Chest pain the pain was felt a day ago after coming from the garden, before admitted to the hospital. During the attack, patient feel sweating, nausea, vomit (-), palpitations (-), shortness of breath (-)
BAGIAN ILMU ANESTESI, PERAWATAN INTENSIF, DAN MANAJEMEN NYERI Supervisor : dr. Khalid Saleh, Sp. PD,KKV,FINASIM
MR number : 579492 Name : Mr. J Age : 58 years old Date administered : November 20 th 2012 Chief complaint: Chest pain The pain was felt a day ago after coming from the garden, before admitted to the hospital. The pain felt pressed by heavy things, radiated to left arm, but no penetrated to the back body. The pain was felt for more than 30 minutes and didnt relieved by rest. During the attack, patient feel sweating, nausea, vomit (-), palpitations (-), shortness of breath (-). Cough (-), history of cough(-) Dizziness (-), Headache (-) , Fever (-) PND (-), DOE (-) Defecation and urination : normal
History of heart disease ( - ) History of hypertension is (-) History of diabetes melitus (-) History of dyslipidemia is unknown History of smoking (+) +25years
General status Moderate illness/well nourished/conscious Vital sign BP : 100/70 mmHg HR : 60 x/min RR : 24 x/min T : 36.5 0 C Head : Anemia (-) , Icterus () Neck : JVP R-2cm H20
Lung : Vesicular, Rhonchi -/- , Wheezing -/- Cor : I : Ictus cordis not visible P : Ictus cordis not palpable P : Dull, normal heart size -Upper border : left 2 nd ICS -Right border : right parasternalis line -Left border : left medioclavicular line A : Heart Sound I/II pure regular, murmur(-) Abdomen : Inspection : flat and following breath movement Auscultation : peristaltic sound (+) , normal Palpation : liver and spleen unpalpable Percussion : tympani, ascites (-) Extremities : Edema -/-
Right ECG Posterior ECG
Rhythm : Sinus rhythm P wave : 0,08 s Heart Rate : 50 x/min, reguler PR interval : 0,24 s Duration QRS : 0,12 s Axis : +10 ST Segment : ST elevation II,III, AvF
Conclusion:
Cardiomegaly with dilatatio, elongatio et atherosclerosis aorta Date of lab test Types of test Result November 20 th 2012 WBC: 13,78 x10 3 mm 3
Myocardial infarction (MI) rapid development of myocardial necrosis caused by a critical imbalance between the oxygen supply and demand of the myocardium.
This usually results from plaque rupture with thrombus formation in a coronary vessels, resulting in an acute reduction of blood supply to a portion of the myocardium.
Occurs when coronary blood flow decreases abruptly after a thrombotic occlusion of a coronary artery previously affected by atherosclerosis.
In most cases, infarction occurs when an atherosclerotic plaque fissures, ruptures, or ulcerates.
ACS describe a group of conditions resulting from acute myocardial ischemia (insufficient blood flow to heart muscle) ranging from unstable angina to myocardial infarction.
Non- Modifiable Modifiable Gender and Age Men, increased risk after age 45 Women, increased risk after age 55 Family History Heart disease diagnosed before age 55 in father or brother Heart disease diagnosed before age 65 in mother or sister Smoking Hypertension Diabetes Mellitus Dyslipidemia Obesity Lack of physical activity 1. Clinical history of ischaemic type chest pain lasting >20 minutes 2. Changes in serial ECG tracings 3. Rise and fall of serum cardiac biomarkers such as creatinine kinase-MB fraction and troponin 1. Chest pain, >30 minutes 2. Usually tight, crushing, and band like 3. Location in retrosternal 4. May radiate to left arm, throat, and jaw 5. Associated features including palpitation, sweating, breathlessness, and nausea.
ST segment elevation over area of damage ST depression in leads opposite infarction Pathological Q waves Reduced R waves Inverted T waves No Yes Yes No
Acute Myocardial Infarction
(STEMI)
NSTEMI
( Non ST-Elevation
Myocardial Infarction ) Unstable Angina Signs of myocardial ischemia Biochemical cardiac markers ? ECG Lab ST segmen elevation ? Fixing the chest pain and fearness o Bed rest o Diet o O 2 2-4 lpm o Nitrat sublingual/oral/IV o Antiplatelet : aspirin and clopidogrel o Morfin/petidine o Diazepam 2-5mg/8 hour Stabilizing the hemodynamic ( blood pressure and pheripheral pulse control) o -blocker o Calcium chanel blocker (CCB) o ACE-Inhibitor Reperfusion of the myocard o Thrombolitik
Class Description Mortality Rate (%) I No clinical signs of heart failure 6 II Rales or crackles in the lungs, an S 3 , and elevated jugular venous pressure 17 III Acute pulmonary edema 30 - 40 IV Cardiogenic shock or hypotension (systolic BP < 90 mmHg), and evidence of peripheral vasoconstriction 60 80 KILLIP CLASSIFICATION Thank you for your attention