System Upper Respiratory Tract -consists of nose, sinuses, nasal passages, pharynx, tonsils, adenoids, larynx and trachea. Nose- composed of an internal and external portion external portion- protrudes from the face supported by the nasal bones and cartilages anterior nares- external openings of the nasal cavities
Review of Anatomy and Physiology of the Respiratory System Paranasal sinuses- include four pairs of bony cavitites. -connected by a series of ducts that drain into the nasal cavity named by their location: frontal, ethmoidal, sphenoidal, and maxillary - fxn: resonating chambers in speech - common site of infection Review of Anatomy and Physiology of the Respiratory System Turbinates(Bones)- also called conchae -increase the surface area of the mucous membrane in the nasal passages - As air enters the nostrils, it comes in contact with the mucous membrane to trap all the dust and microorganisms; air is moistened and warmed to body temperature Review of Anatomy and Physiology of the Respiratory System Internal portion- hollow cavity separated into right and left nasal cavities by the nasal septum Each nasal cavity divided into 3 passageways by the projection of the turbinates(conchae) from the lateral walls -lined with highly vascular ciliated mucous membranes caleed the nasal mucosa Review of Anatomy and Physiology of the Respiratory System Pharynx- tubelike structure that connects the nasal and oral cavities to the larynx 3 regions: nasopharynx- posterior to the nose and above the soft palate oropharynx- houses the palatine tonsils laryngopharynx- extends from the hyoid bone to the cricoid cartilage Epiglottis- forms the entrance to the larynx Review of Anatomy and Physiology of the Respiratory System Larynx- cartilaginous, epithelium lined structure that connects the pharynx and the trachea - major function: vocalization -consists of the following: *Epiglottis- flap of cartilage that covers the opening to the larynx during swallowing *Glottis- the opening between the vocal cords in the larynx *Thyroid cartilage- the largest of the cartilage structures; part of it forms the Adams apple *Cricoid cartilage- the only complete cartilaginous ring in the larynx(located below the Thyroid cartilage) *Arytenoid cartilages- used in vocal cord movement with the Thyroid cartilage *Vocal cords- ligaments controlled by muscular movements that produce sounds; located in the lumen of the larynx Review of Anatomy and Physiology of the Respiratory System Trachea- a.k.a. windpipe composed of smooth muscle with C-shaped rings of cartilage at regular intervals The cartilaginous rings are incomplete on the posterior surface and give firmness to the wall of the trachea to prevent it from collapsing Serves as passage between the larynx and the bronchi Review of Anatomy and Physiology of the Respiratory System Lungs- paired elastic structures enclosed in the thoracic cage(airtight chamber with distensible walls) Ventilation- requires movement of the thoracic cage and diaphragm(Floor) Inspiration- air enters because of the negative pressure in the thoracic cage Expiration- air is forced out during lung recoil Passive process requiring little energy COPD- expiration requires energy Review of Anatomy and Physiology of the Respiratory System Pleura- serous membrane lining the lungs(Visceral pleura) and the wall of the thorax(parietal pleura) -small amount of pleural fluid between the two membranes serve to lubricate the thorax and the lungs to permit smooth motion of the lungs within the thoracic cavity with each breath Pleural friction rub- Review of Anatomy and Physiology of the Respiratory System Mediastinum- middle of the thorax between the pleural sacs that contain the two lungs -extends from the sternum to the vertebral column and contains all the thoracic tissue outside the lungs. Lobes -left lobe- consists of an upper and lower lobe -right lobe- consists of an upper, middle, and lower lobe *Each lobe further subdivided into two to five segments separated by fissures Review of Anatomy and Physiology of the Respiratory System Bronchi and bronchioles -lobar bronchi- 3 in the right and 2 in the left Lobar bronchi divide into: Segmental bronchi- 10 on the right and 8 on the left -structures identified when choosing the the most effective postural drainage position for a given patient. Segmental bronchi divide into: Subsegmental bronchi- surrounded by by connective tissue that contains arteries, lymphatics, and nerves Subsegmental bronchi then divide into: Bronchioles- no cartilage in their walls -patency depends on the elastic recoil of the surrounding smooth muscle and on the alveolar pressure *The bronchi and bronchioles are lined with cells that have cilia(propels mucus and foreign substances away from the lungstowards the larynx Review of Anatomy and Physiology of the Respiratory System Alveoli- the lung is made of 300 million alveoli arranged in clusters of 15 to 20 =70 sq. meters(tennis court) 3 Types of alveolar cells Type1- epithelial cells that form the alveolar walls Type2- metabolically active(secrete surfactant which is a phospholipid that lines the inner surface to prevent alveolar collapse Type3- large phagocytic cells that ingest foreign matter( bacteria, mucus) - acts a defense mechanism Review of Anatomy and Physiology of the Respiratory System Functions of Respiration Oxygen transport- capillary-cell exchange Respiration- capillary-alveoli exchange Ventilation- inspiration from the trachea to the alveoli expiration- alveolar gas travels in reverse Review of Anatomy and Physiology of the Respiratory System Air Pressure Variances- Inspiration 1.movement of the diaphragm and thoracic cavity 2. enlarges the thoracic cavity 3. lowers the pressure inside the thorax to a level below atmospheric pressure 4. air is drawn into the alveoli
Review of Anatomy and Physiology of the Respiratory System Air pressure Variances Expiration 1. The diaphragm relaxes and the lungs recoil 2. Decrease in the size of the thoracic cavity 3. Alveolar pressure exceeds atmospheric pressure 4. Air flows from the lungs into the atmosphere Airway Resistance determined by the radius or size of the airway through which the air is flowing Eg. Changes in bronchial diameter- affects airway resistance and alters the rate of air flow Eg. Bronchial asthma Review of Anatomy and Physiology of the Respiratory System Causes of Increased airway Resistance: *contraction of bronchial smooth muscle- asthma *obstruction of the airway- mucus, tumor or foreign body *loss of lung elasticity- emphysema, characterized by connective tissue encircling the airways, keeping them open during inspiration and expiration Review of Anatomy and Physiology of the Respiratory System Compliance- measure of the elasticity, expandability and distensibility of the lungs Volume-pressure relationship in the lungs High compliance- overdistended lungs Low or decreased- stiff lungs eg. Pnemothorax, hemothorax, pleural effusion, pulmonary edema, pulmonary fibrosis, ARDS Tidal Volume -VT or TV - the volume of air inhaled and ehaled with each breath -normal value: 500 ml - Significance : the tidal volume may not vary even with severe disease Inspiratory Reserve Volume -IRV - the maximum volume of air that can be inhaled after a normal inspiration -normal value: 3000 ml
Expiratory reserve Volume ERV The maximum volume of air that can be exhaled forcibly after a normal exhalation Normal value: 1100 ml. Decreased with restrictive conditions such as obesity, ascites, pregnancy
Residual Volume RV The volume of air remaining in the lungs after a maximum exhalation Normal value: 1200 ml Increased with obstructive disease Vital Capacity VC The maximum volume of air exhaled from the point of maximum inspiration VC= TV+IRV+ERV Normal value: 4600 ml. Decreased VC may be found in neuromuscular disease, generalized fatigue, atelectasis, pulmonary edema, and COPD Inspiratory Capacity IC Maximum volume of air inhaled after normal expiration IC=TV+IRV Normal value: 3500 ml. Decreased IC in restrictive disease Functional Residual Capacity FRC Volume of air remaining in the lungs after a normal expiration FRV= ERV+RV Normal value: 2300 ml. May be increased with COPD And decreased in ARDS
Total Lung capacity TLC The volume of air in the lungs after a maximum inspiration Normal value: 5800 ml. May be decreased with restrictive disease ie. Atelectasis, pneumonia Increased in COPD
Diffusion- process by which oxygen and carbon dioxide are exchanged at the air- blood interface(alveolar- capillary membrane) Pulmonary perfusion- actual blood flow to the pulmonary circulation Ventilation- actual flow of gas in and out of the lungs Perfusion- filling of the pulmonary capillaries with blood Adequate gas exchange depends on an adequate ventilation-perfusion ratio(V/Q) Imbalance occurs as a result of inadequate perfusion, inadequate ventilation or both Four possible V/Q states Normal ratio -healthy lung -equal amount of blood passes an alveolus matched by an equal amount of gas - ratio is 1:1 -ventilation matches perfusion
Four possible V/Q states Low ventilaton- perfusion ratio -also called shunt producing disorders -perfusion exceeds ventilation, a shunt exists -blood bypasses the alveoli but without gas exchange occuring - seen in distal airway obstruction ie. Pneumothorax, atelectasis, tumor, mucus plug
Four possible V/Q states High Ventilation-perfusion ratio (Dead Space) Ventilation exceeds perfusion Alveoli do not have adequate blood supply for gas exchange to occur Pulmonary emboli, pulmonary infarction, cardiogenic, shock.
Four possible V/Q states Silent unit Absence or limited ventilation and perfusion Seen in pneumothorax, severe ARDS
Health History- focuses on the physical and functional aspects of the patient and the effects of these problems on the patient, including activities of daily living. Reason the patient is seeking health care often related to DYSPNEA, HEMOPTYSIS Dyspnea- difficult or labored breathing is a common symptom in pulmonary and cardiac patients May be associated in neuromuscular disorders ie. Myasthenia gravis, Guillain-Barre syndrome, muscular dystrophy May also occur after physical exercise in people without disease. Common at the end of life. Acute diseases of the lungs produce a more severe grade of dyspnea Sudden dyspnea in a healthy person- pneumothorax, acute respiratory obstruction, or ARDS Sudden dyspnea in immobilized patients- pulmonary embolism May occur with other disorders: cardiac disease, anaphylactic reactions, severe anemia. Orthopnea- inability to breath easily except in the upright position May be found in patients with heart disease or COPD
How much exertion triggers shortness of breath Cough? Is dyspnea related to other symptoms? Was the onset sudden or gradual At what time of the day or night does SOB occur Worse when flat in bed Does it occur at rest? Exercise? Running? Climbing stairs? Is the SOB worse while walking? If so, when walking how far? How fast Relief measures: identify and correct cause, rest and high Fowlers position, Oxygen Cough- reflex that protects the lungs from the accumulation of secretions or the inhalation of foreign bodies Results from the irritation of the mucous membrane anywhere in the resp. tract Symptom of a disorder Causes: airborne irritant, infection, cardiac disease, medications(ACE inhibitors), smoking, GERD(gastro-esophageal reflux disease) Sputum production- reaction of the lungs to any constantly recurring irritant May also be associated with nasal discharge. Bacterial infection- profuse, thick yellow, green or rusty colored sputum Viral bronchitis- thin, mucoid sputum Chronic bronchitis- gradual increase of sputum over time Lung abscess- foul smelling sputum Relief measures: hydration, aerosolized solutions Chest Pain- in pulmonary conditions, pain is sharp, stabbing, intermittent, dull, aching or persistent May occur with pneumonia, pulmonary embolism, lung infarction Wheezing- may be heard with or without a stethoscope. High pitched, musical sound heard mainly on expiration Bronchodilators( oral or inhalants) Clubbing of the fingers- sign of lung disease in patients with chronic hypoxic conditions Initially manifested as sponginess of the nail bed and loss of nail bed angle. Hemoptysis- expectoration of blood from the respiratory tract May be sudden, intermittent or continuous Most common causes: pulmonary infection, carcinoma, pulmonary artery or vein abnormalities, pulmonary embolus and infarction, abnormalities of the heart and blood vessels. Cyanosis- bluish coloring of the skin Assessmnet of cyanosis is affected by room lighting, skin color, and distance of the blood vessels from from the surface of the skin
Eupnea- normal breathing at 12-18 breaths /minute Bradypnea- slower than normal(<10 breaths/min) with normal depth and regular rhythm Tachypnea- rapid,shallow breathing >24 breaths/min Hypoventilation- shallow,irregular breathing Hyperventilation-increased rate and depth of breathing( Kussmauls respiration if caused by diabetic ketoacidosis) Apnea- period of cessation of breathing.Time duration varies -may occur with other breathing disorders ie. Sleep apnea Cheyne- Stokes- regular cycle where the rate and depth of breathing increase, then decrease until apnea. Biots Respiration- periods of normal breathing(3-4 breaths) followed by a varying period of apnea( usually 10-60 seconds) Tactile Fremitus- detection of vibration on the chest wall by touch -sound generated by the larynx travels distally along the bronchial tree to set the chest wall in resonant motion. Factors affecting fremitus: 1. thickness of the chest wall 2. obesity with increased subcutaneous tissue *lower pitched sounds travel better through the normal lung and produce greater vibration on the chest wall *more pronounced in men than in women because of deeper male voice *most palpable in the upper thorax anteriorly and posteriorly *patient is asked to repeat ninety nine, one-two-three, or eee, eee, or tres-tres. Air does not conduct sound well Solid substances such as tissue conduct sound well Emphysema patients- decreased tactile fremitus Pneumonia with lung consolidation- increased fremitus Determines whether the underlying tissues are filled with air, fluid or solid material Estimate the size and location of certain structures within the thorax( eg. Diaphragm, heart and liver) Flatness----- Eg. Large pleural effusion location example: thigh Dullnes------Eg. Lobar pneumonia location example: liver Resonance-----eg. Simple chronic bronchitis location example: normal lung Hyperresonance--- Eg. Emphysema, pneumothorax location example: none Tympany----- eg. Large pneumothorax location example: gastric air bubble; puffed out cheek Vesicular- inspiratory sounds lasts longer than expiratory sounds location- entire lung field over the upper sternum and between the scapula Bronchovesicular- inspiratory nd expiratory sounds are about equal location: 1 st and 2 nd interspacesanteriorly and between the scapula( over the main bronchus) Bronchial- expiratory sounds lasts longer the inspiratory sounds location: over the manubrium Tracheal- inspiratory and expiratory sounds are about equal location: over the trachea in the neck Crackles in general- discontinuous popping sounds that occur during inspiration etiol: fluid in the airways or alveoli or to opening of collapsed alveoli. coarse crackles- discontinuous popping sounds heard in early inspiration; harsh moist sounds originating in the large bronchi etiol: obstructive pulmonary disease Fine crackles- discontinuos poppping sounds heard in late inspiration; like hair rubbing together; originates in the alveoli etiol: instertitial pneumonia, restrictive pulmonary disease(fibrosis), bronchitis, pneumonia Wheezes sonorous wheezes(rhonchi)- deep low pitched rumbling sonds heard primarily during expiration;caused by air moving through narrowed tracheobronchial passages etiol: secretions or tumor Wheezes sibilant wheezes- continuous, musical, high pitched, whistle like sounds heard during inspiration and expiration etiol: air passing through narrowed or partially obstructed airways eg. Bronchospasm, asthma Friction rubs Pleural friction rubs- harsh crackling sound, like 2 pieces of leather rubbing together -heard during inspiration alone or during inspiration and expiration -may subside when patient holds breath. Coughing will not clear sound etiol: inflammation or loss of lubricating pleural fluid Assessment Findings in Common Respiratory Disorders Disorder Tactile fremitus Percussion Auscultation Consolidation eg pneumonia Increased Dull Bronchial breath sounds Bronchitis Normal Resonant Normal to dec breath sounds, wheezes Emphysema Normal to decreased Resonant to hyperresonant Dec. intensity of breath sounds, usually with prolonged expiration Asthma Normal to decreased Resonant to hyperresonant Wheezes Assessment Findings in Common Respiratory Disorders Disorder Tactile fremitus Percussion Auscultation Pulmonary edema Normal Resonant Crackles at lung bases,possibly wheezes Pleural effusion Absent Dull to flat Decreased to absent breath sounds Pnemothorax Decreased Hyperresonant Absent breath sounds Atelectasis Absent Flat Decreased to absent breath sounds Diagnostic Studies Pulmonary function tests(PFTs) -routinely used in patients with chronic respiratory disorders -assesses respiratory function and determines the extent of destruction -monitors the course of the patient with an established respiratory disease and assesses response to therapy Pulmonary Function Tests(PFTs) Forced Vital capacity -FVC -reduced in COPD because of air trapping -performed with a maximally forced expiratory effort Forced Expiratory volume -FEV1 -volume of air expired in the specified time during the performance of forced vital capacity; FEV1 is volume exhaled in 1 second -clue to the severity of airway obstruction Arterial Blood Gas Studies Measurement of the blood pH and arterial oxygen and CO2 tensions Blood obtained from arterial punctures Nsg. Responsibility: !!!!! Adequate hemostasis PaO2- indicates the degree of oxygenation of blood PaCO2- indicates the adequacy of alveolar ventilation Aid in assessing the ability of the lungs to provide adequate oxygen and remove CO2 and the ability of the kidneys to reabsorb or excrete bicarbonate ions to maintain normal body pH Serial ABGs- explain to the patient the procedure Arterial Blood Gas Studies Normal ABG values pH- 7.35- 7.45 PaCO2- 35 -45 mm Hg PaO2- 80- 100 mm Hg HCO3- 22-26 meq/L Pulse oximeter Non invasive method of continuously monitoring the oxygen saturation of hemoglobin(SaO2) Referred to as spO2 when oxygen saturation is measured by pulse oximetry Effective tool for monitoring for subtle or sudden changes in oxygen saturation. Probe may be attached to: finger tip, ear lobe, bridge of the nose, forehead Normal spO2- 95-100%? Factors that affect SpO2 Vales dyes, vasoconstrictor medications, cardiac arrest and shock Cultures Cultures from the throat or nose- useful in identifying organisms responsible for causing the disease
Sputum Studies Obtained for analysis to identify the pathogenic organisms and to determine whether malignant cells are present. May also be used for hypersensitivity states to guide treatment. Expectoration is the usual method of collection Sputum Studies Other methods of collecting sputum 1.Endotracheal aspiration 2. bronchoscopic removal 3. transtracheal aspiration Nsg responsibility: 1. specimen should be delivered to the lab within 2 hours of collection Imaging Studies Chest x-ray normal pulmonary tissue- radiolucent Routine chest x-ray- 2 views PA view; lateral projection usually taken with a full inspiration
X-ray: shows major contrasts between body densities such as bone, soft tissues, and air Imaging Studies Computed Tomography -imaging method where the lungs are scanned in successive layers by a narrow beam x-ray -provides a cross sectional view of the chest - may use contrast agents Nsg Responsibility: allergy to iodine or seafoods BUN/Creatinine levels
Imaging Studies Magnetic Resonance Imaging (MRI) -similar to CT scan except magnetic fields and radiofrequency are used instead of a narrow beam x-ray -yields more detailed images than a CT. Imaging Studies Pulmonary Angiography - commonly used to investigate thromboembolic diseases of the lungs. eg. Pulmonary embolism,congenital abnormalities of the pulmonary vascular tree Procedure: rapid injection of a radiopaque agent into the vasculature of the lungs for radiographic study of the pulmonary vessels Endoscopic Procedures Bronchoscopy- direct inspection and examination of the larynx, trachea, and bronchi through a flexible fiberoptic bronchoscope Purposes: Diagnostic 1. determine the location and extent of the pathologic process 2. examine tissues or collect secretions 3. determine whether a tumor can be resected surgically 4. obtain tissue sample for diagnosis 5. diagnose bleeding sites Endoscopic Procedures Bronchoscopy Purposes: Therapeutic 1. remove foreign bodies from the tracheobronchial tree 2. remove secretions when the patient cannot clear them 3. treat post op atelectasis 4. destroy and excise lesions Complications of Bronchoscopy 1. reaction to local anesthetic 2. infection 3. aspiration 4. bronchospasm 5. hypoxemia 6. pnemothorax 7. bleeding 8. perforation Endoscopic Procedures Bronchoscopy- Nursing Interventions 1. consent 2. withhold food and fluids 6 hours 3. Preop meds- atropine- vagal stimulation and bradycardia opioids- 4. remove dentures 5. post op- nothing by mouth until cough reflex returns, then ice chips---fluids Imaging Studies Thoracentesis -accumulation of pleural fluid may occur with some disorders of which a sample of this fluid can be obtained thoracentesis - aspiration of pleural fluid for diagnostic or therapeutic purposes -may be ultrasound guided- less complications -Studies include: Gram staining, C/S, acid fast staining and culture, differential cell count, cytology, pH, sp. Gravity, total protein, and lactic dehydrogenase Thoracentesis Nursing Activities Ascertain in advance- chest x-ray has been ordered Assess for allergy to the local anesthetic Administer sedation if prescribed Inform the patient: 1. remain immobile 2. pressure sensations expected 3. minimal discomfort after the procedure Position: Upright- facilitates removal of fluid that localizes at the base of the thorax *sitting on the edge of the bed with the feet supported and arms and head on a padded over the bed table. Imaging Studies Biopsy- excision of a small amount of tissue for examination of cells Pleural Biopsy -accomplished by needle biopsy of the pleura or pleuroscopy(visual explorationthrough a fiberoptic bronchoscope inserted into the pleural space. -performed when there is pleural exudateof undetermined origin or when there is a need to stain or culture the tissue to identify tuberculosis or fungi. Pulmonoray Angiography Used to investigate thromboembolic disease of the lungs eg. Pulmonary embolism, congenital abnormalities of the pulmonary system Procedure- rapid injection of a radiopaque substance into a vein while simultaneously doing a radiographic study (CT Scan). Upper Airway Infections Rhinitis- inflammation and irritation of the mucous membranes of the nose -acute, chronic, non allergic, allergic -Allergic rhinitis- may be classified as seasonal (pollen seasons) Perennial(occurs throughout the year) Pathopyhysiology: environmental factors changes in temp., odors, food, drugs such as cocaine and antihypertensive meds., infection. Rhinitis Management 1. H and P - ask for possible exposure to allergens - swollen and congested nasal mucosa Meds: decongestants/paracetamol corticosteroids desensitizing immunizations antibiotics antihistamines saline sprays Viral Rhinitis/common cold: rhinorrhea, nasal congestion, sneezing, headache, muscle pains - highly contagious Cause: rhinovirus other causes: adenovirus, Coronavirus, RSV, influenza virus ***Each virus has many strains---vacccine is impossible
Acute Sinusitis Inflammation of the mucous membranes of the paranasal sinuses Often follows URIobstruction of the nasal cavities--- bacterial growth Most often caused by bacteria (60%) Other causes: abnormal structures of the nose, diving and swimming, trauma S/Sx: facial pain fatigue purulent nasal discharge fever, headache ear pain nasal obstruction sense of fullness decrease sense of smell sore throat snoring periorbital edema Acute Sinusitis Assessment 1.tenderness to palpation of the sinus area 2. tenderness on tapping of the sinus area 3. transillumination with light- decrease transmission Complications: meningitis, abscess, osteomyelitis, Meds: same as in rhinitis Acute Sinusitis Nsg. Management 1. Steam inhalation 2. Warm compresses 3. Avoid- swimming, diving, air travel 4. Nasal decongestant- rebound congestion 5. Follow recommended antibiotic regimen 6. Advise for signs of complications: nuchal rigidity, severe headache, fever
Chronic Sinusitis Symptoms of sinusitis lasting more than 3 months Pathopysiology: same as in Acute sinusitis S/sx: same as in acute Medical mgt: same as in acute *surgery to correct to correct structural deformities eg. Excising nasal polyps, correcting deviated nasal septum Acute Pharyngitis Sudden inflammation of the pharynx Sore throat- primary symptom Causes: bacterial and viral(self limiting) Complications: 1. sinusitis 2. otitis media 3. peritonsillar abscess 4. pneumonia 5. meningitis 6. rheumatic fever (Group A B-hemolytic Streptococcus) 8. nephritis S/Sx: swollen pharyngeal membrane and tonsils(exudate), lymphadenopathy
Chronic Sinusitis Persistent inflammation Causes: -Common in adults, over use of voice, alcohol, tobacco S/Sx: soreness /fulness of the throat dysphagia postnasal drip Management: antibiotics, antihistamines, decongestants
Tonsillitis/Adenoiditis Acute tonsillitis- can be confused with pharyngitis Group A Beta hemolytic Sreptococcus- most common organism S/sx: fever, sore throat, snoring, mouth breathing, earache, May spread to the middle ears via Eustachian tubes otitis media Management: increased fluid intake, salt water gargles, pain mends, PCN, Surgery: tonsillectomy/ adenoidectomy for repeated episodes Tonsillitis/Adenoiditis Post op care in tonsillectomy 1. immediate post op care- patient in prone position with head turned to the side 2. ice collar to the neck 3. frequent swallowing- warrants investigation- may suggest hemorrhage 4. Vital signs- increasing heart rate (^Temp or decreasing BP) 5. check for swallowing reflex- ice chips 6. Advise: refrain from too much talking 7. control coughing
Peritonsillar abscess Collection of purulent exudate between the tonsillar capsule and the surrounding tissues Common cause : Beta hemolytic streptococcus Edema can cause airway obstruction-life threatening S/Sx: fever, trismus, drooling, odynophagia swelling of the soft palate, unilateral tonsillar hypertrophy Med Management: antibiotics(PCN)- effective -fine needle aspiration of the of pus or incision and drainage Laryngitis Result from voice abuse, dust, chemicals, smoke, or as part of URI, allergies S/Sx: hoarseness aphonia severe cough Management: voice rest, antibiotics and steroids, dysphagia, hemoptysis Obstruction and Trauma of the Upper Respiratory Airway Sleep Apnea 1. excessive daytime sleepiness 2. frequent nocturnal awakening 3. insomnia 4. loud snoring 5. morning headaches 6. intellectual deterioration 7. irritabilty 8. impotence 9. dysrhythmias 10. severe hypertension 11. pulmonary hypertension 12. polycythemia 13. enuresis Sleep Apnea
Managementt 1.avoid sleeping on the back 2.avoid alcohol and meds that depress the upper airway 3. weight loss 4. CPAP or biPAP(bilevel positive airway pressure) 5. surgery- uvulopalatopharyngoplasty 6. Meds: Protryptiline increases the respiratory drive Medroxyprogesterone ? Acetazolamide(Diamox) ?