Periodontal defects but this is not so. This defect is caused by dental plaque with accentuation due to the open contact region and poor subgingival margin of Restoration.
Histology of Intrabony defect due
to plaque induced Periodontitis. Arrows show sub gingival plaque on root surface
Irritation factors are plaque that
induces Gingivitis which progresses to Periodontitis. Traumatizing factors from occlusion cause tissue changes in periodontal ligament space.
Zone of co-destruction occurs when
plaque induced Periodontitis occurs in a tooth that also has Traumatic Occlusion resulting in more severe bone loss than that seen with Periodontitis alone.
Host parasite reaction between
bacterial plaque and host inflammatory response is the cause of pocket depth and attachment loss. The presence of Traumatic occlusion can accentuate the damage when Periodontitis proceeds apically into the Periodontal Ligament Space.
Tissue Changes Due to
Traumatic Occlusion
The first reaction to increased
occlusal loading is increased vascularity in the Periodontal ligament space. No changes are seen in gingival tissues.
Normal Periodontal ligament
with normal occlusal forces showing dense collagen fibers attached to bone and cementum with minimal vascularity.
With excessive occlusal
loading the collagen fibers lose their connections between cementum and bone ,and blood vessels proliferate.
This initial increased
vascularity results in a more compressible periodontal ligament and increased clinical mobility.
Changes in the apical
periodontal ligament vascular patterns can also result in increased vasodilation of the pulp with increased sensitivity and pain to Hot and Cold stimuli secondary to Traumatic Occlusion.
In Traumatic Occlusion after
the initial change of increased vascularity, there is a stimulation of osteoclasts which cause bone loss and a widened periodontal ligament space. This also causes increased tooth mobility.
Further effects of Traumatic
Occlusion are seen with loss of density of collagen and absence of a functional fiber arrangement.
Loss of Density of Collagen
High power view. No collagen
fibers adjacent to bone and loss of functional support of Periodontium.
Advanced Traumatic Occlusion
with minimal Periodontal ligament tissue. An advancing plaque induced Periodontitis can rapidly spread apically in this situation.
Normal Periodontium
Result of Traumatic Occlusion
Periodontal ligament tissues
can respond with Traumatic Occlusion changes when a normal periodontium is affected by increased occlusal loading due to bruxing clenching or a high restoration
These changes are called
Primary Occlusal Trauma or Primary Trauma from occlusion.
In teeth with bone loss due to
periodontal disease previously well tolerated occlusal loading can become traumatic and cause changes in the periodontal ligament tissues.
These changes are called
secondary occlusal trauma or secondary trauma from occlusion.
Coronal portion of plaque
induced Periodontitis with pocket formulation
Region of crestal bone showing
intrabony pocket due to plaque this is blending with Traumatic Occlusion induced Periodontal ligament changes of loss of collagen and increased vascularity.
More Apical region with
Traumatic Occlusion changes seen deep in Periodontal tissues apical to Periodontitis.
Apical part of plaque induced Periodontitis
Traumtic occlusion changes deep in periodontal ligament
Radiograph of lower Molar with
Traumatic Occlusion. Widened Periodontal ligament space on Mesial all the way around the apex with beginning bone loss in furcation (arrows).
There is also thickened lamina
dura and this tooth has increased mobility.
First molar has traumatic occlusion
causing the bone loss in the furca. Clinically there is no pocket depth nor Periodontitis in the furcation and so the diagnosis is Traumatic Occlusion and the treatment is occlusal adjustment to reduce occlusal loading.
Both premolars have traumatic
occlusion and there is an addition Periodontitis related bone loss and pockets on the mesial of the first premolar.
Gingival recession is not
caused by Traumatic Occlusion but is related to inadequate Keratinized Gingiva and excessive tooth brushing.
Wedge shaped defect in root
of lower first premolar is due to traumatic toothbrushing and is not related to Traumatic Occlusion
Abfraction type of root loss
like this has not been shown to occur clinically in association with heavy occlusal forces.
At time of Periodontal surgery
large hyperplastic bone response to heavy occlusal load called Buttressing Bone
Buttressing Bone removed
during periodontal surgery to facilitate normal contour of gingival tissues.