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RootCare Cream

The novel natural pathway


for the treatment of most
eczema types

Eczema

Diagnostique eczema

Histoire
Caractre de la lsion
Territoire
Pathologie
Tests cutans

Characteristiques des
lesions

Erytheme
oedeme
Vsicule
Papule
suintement Srum
caille
Fissure
lichenification

Classifications

Clinical staging
Causes of eczema

Clinical classification

eczema aigue
eczema subaigue
eczema chronique

Aigue

Subaigue

Chronique

Chronique

Causes of eczema

Endogenous cause
Exogenous cause

Cause Exogene

Dermatite de contact irritante


Dermatite de contact allergique
Dermatite de photosensibilite
dermatite eczmatiformes
infectieuse

cause Endogene

Dermatite atopique
Dermatite seborrheique
Eczema nummulaire
Dyshidrosis
Lichen simplex chronicus
Asteatotic dermatitis
Pityriasis alba
Eczema de stase
Juvenile plantar dermatitis

Dermatite atopique

Dermatite chronique de lenfant la


plus frquente
Historique familial
Autosomal dominant
Bas age ou enfant
Relie une rhinite allergique ou
asthme

Formes Cliniques

Prurit est commun


3 age groups : infantile, childhood,
adolescent or adult AD

Infantile AD

Rgion faciale et perioral puis


extension surface des bras et des
jambes, zone priauriculaire, pargn
zone de la couche
Intensment prurigineuses, papules
rythmateuses, excoriation, vsicules
sur la peau rythmateuse, et exsudat
sreux
2 mois-2 ans

Infantile AD

Infantile AD

Infantile AD

Infantile AD

Atopic dermatitis

Childhood AD

Age >2-3 ans


Habituellement dermatite subaigue
plis des extrmits, le cou et les
paupires
paississement et fissures des
paumes et des plantes

Childhood AD

Childhood AD

Childhood AD

Adolescent AD

As in childhood AD but prominent in


hand eczema, nipple eczema and
cheilitis

Etiology

Genetics : autosomal dominant


Environments : food, irritants,
infections, emotional stress
Immunologic factors : abnormal T
cell activation

Treatment

La premire tape consiste briser


le cycle dmangeaisons-grattage
Prvention des dclencheurs
Contrle des facteurs aggravants
dans l'environnement
La restauration de la fonction
barrire de la peau et l'hydratation
de la peau avec des mollients

Treatment

Medication topique ou orale pour


traiter les dmangeaisons et
l'inflammation
Les corticostrodes topiques ont
dmontr la fois rduction de la
colonisation de S. aureus de la peau
et ont des effets anti-inflammatoires
dans le traitement de la DA

Treatment

Les inhibiteurs topiques de la


calcineurine (TCI) ont des effets
anti-inflammatoires sans le profil
d'effets secondaires des
corticostrodes topiques

Treatment

La peau est souvent colonise fortement


avec S. aureus.
antiseptique topique a t montr pour
rduire les taux de colonisation et de
rduire les symptmes de la peau.
Crusting, pleurant ou pustules indiquent
souvent l'infection Staphylococcus
aureus et le traitement avec des
antibiotiques par voie orale est indiqu.

Treatment

Oral antihistamine
But !
Treatment of AD with topical antihistamines
is generally not recommended because it mig
ht cause cutaneous sensitization

Treatment

Nummular
eczema

Nummular eczema

Common in male
Age 55-65 yrs. Of both sexes or 1825 yrs in female
Rare in children, esp. under 1 yr

Nummular eczema

Clinical features

Etiology

Unknown
May associated with atopy, allergic
sensitivity to infections or irritant
(nickle, chromium), dry skin

Treatments

Emollients, avoid drying of skin


Moderately potent topical steroids
UV light
Tacrolimus
Antihistamine
Systemic steroids in refractory case

Lichen simplex
chronicus and
prurigo nodularis

Etiology

Chronic repetitive rubbing,


scratching and piking skin

Lichen simplex chronicus

Lichen simplex chronicus

Prurigo nodularis

Treatments

Avoid scratching, rubbing


Highly potent topical steroid
Keratolytics
Intralesional steroid injection
Capsaisin, PUVA, UVB
Antihistamine
Medical conditions

Pityriasis alba

Pityriasis alba

Unknown etiology
Not ass. Infection
May be the variant of AD but also
found in non allergic patients

Pityriasis alba

Pityriasis alba

Treatments

Mild potent topical steroid


Emollient cream

Seborrheic
dermatitis

Seborrheic dermatitis

3 age group : infancy, puberty, old


Unknown etiology :
Pityrosporum ovale (Malassezia furfur)
Sebaceous gland activity
Neurological disorder : Parkinsonism,
paralysis
HIV
Obesity or malabsorption

Seborrheic dermatitis

Emotional changes
Drugs : Arsenic, gold, Methyldopa,
cimetidine, neuroleptics

Seborrheic dermatitis

Seborrheic dermatitis

Seborrheic dermatitis

Treatments

Mild potent topical steroids


Shampoo : tar, 2% selenium sulfide,
zinc pyrithione, pyroctone olamine
or antifungal
Cotrimazole or nicodazole cream

Stasis eczema

Stasis eczema

Stasis eczema

Stasis eczema

High pressure in venous system


Varicose vein at legs, DVT
Obesed, pregnant, genetics
Middle age to old, common in female

Treatments

Mild potent topical steroid


Correct the cause of venous stasis

Exogenous
eczema

Contact dermatitis

Irritant contact dermatitis


Allergic contact dermatits
Phototoxic contact dermatits
Photoallergic contact dermatitis

Irritant CD

Nonimmune irritation to chemical


agents, detergent, soap
Chronic wet work, detergent, soap

Irritant contact
dermatitis

Irritant contact
dermatitis

Irritant contact
dermatits

Allergic contact
dermatitis

Allergic contact
dermatitis

Allergic contact
dermatitis

Treatments

Avoidance
Treatment is according to stage of
eczema

Vesicular
Palmoplantar
Eczema

Acute and/or chronic dermatitis


Small to large blisters on the palms
and soles
Endogenous dermatitis
Divided 4 categories : pompholyx,
chronic vesiculobullous hand
dermatitis, hyperkeratotic hand
dermatitis, id reactions

RootCare Cream
Pharmacology Overview

Active Constituent

Aloe Vera Gel

Main active
ingredient

Lupeol (natural
sterol) and beta
sitosterol as antiinflammatory.
Mannose 6phosphate and
acemannan as
healing agents. Ale
Vera contains
vitamins, amino
.acids, and minerals

Action

Anti-inflammatory
and strong healing
.agent

Active Constituent

.Licorice Ext

Main active
ingredient

Glabridin and
glycyrrhizin

Action

Prevent cell overproliferation and


considered as strong
anti-inflammatory by
inhibiting
.prostaglandin E2

Active Constituent

Evening Primrose
Oil

Main active
ingredient

Action

Essential fatty acids


Considered as one are important as
cellular structural
of the most
important source of elements and as
GLA (essential fatty precursors of
acids)
.prostaglandins

Active Constituent

Lavender Oil

Main active
ingredient

Terpene ketone,,
alpha pinene and
. limonene

Action

Lavender Oil is
antiseptic,
antibacterial, antifungal and it is also
an immune booster
and powerful antiinflammatory for
. the skin

Active Constituent

Main active
ingredient

Action

Potent antiChamomile Oil

Bisabolol,
chamazulene, and
the flavonoids
apigenin and
luteolin

.inflammatory agent

Active Constituent

.Calendula Ext

Main active
ingredient

Calendulin,
calendic acid, as
natural sterols and
flavonol and
triterpene
glycosides

Action

Anti-inflammatory
and anti-microbial
.effect

Active Constituent

.Bergamot fruit Ext

Main active
ingredient

Furocoumarins as
Bergapten

Action

Immunomodulator
agent

Active Constituent

Emu Oil

Main active
ingredient

Linolinic acid and


other essential fatty
acids, Terpenines,
Saponines and
. Vitamin A and E

Action

Anti-inflammatory,
reepithelialization,
healing,
moisturizing and
.soothing

Active Constituent

Vitamin E
(Tocopherol)

Main active
ingredient

Tocopherol
(Vitamin E)

Action

Powerful anti.oxidant agent

Active Constituent

Zinc Oxide

Main active
ingredient

Zinc oxide

Action

Skin healing and


astringent (Zinc
provides the body
with the extra zinc
it needs to repair
.skin cells)