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LEVELS OF PREVENTION

(LEAVELL & CLARK)


THE NATURAL HISTORY OF ANY DISEASE OF MAN
Interrelation of Agent, Host, and Environmental Reaction of the HOST to the STIMULUS
factors
Early Discernible Advanced
pathogenesis earlyl esions disease Convalescence
Production of STIMULUS
PREPATHOGENESIS PERIOD PERIOD OF PATHOGENESIS

EARLY DIAGNOSIS and REHABILITATION


HEALTH PROMOTION
SPECIFIC PROTECTION PROMPT TREATMENT
Provision of hospital
Use of specific Case-finding measures, and community facilities
Immunizations individual and mass DISABILITY LIMITATION for retraining and
Health education
Attention to Screening survey education for
Good standard
of nutrition personal hygiene maximum use
Selective examinations Adequate treatment of remaining capacities
Use of environmental Objectives : to arrest
Provision of adequate
sanitation To cure and prevent the disease process Education of the public
Housing, recreation,
disease processes and to prevent and industry to utilized
and agreeable
work conditions Protection against further complication the rehabilitated
occupational hazards To prevent the spread of and sequelae as full employment
communicable diseases
Marriage counseling
and sex education Protection from accidents Provision of Selective placement
To prevent complications facilities to limit
Use of specific nutrients and sequele disability Work therapy in hospitals
Genetics
and to prevent death
Protection from carsinogens To shorten period of disability Use of sheltered colony
Avoidance of allergens

PRIMARY PREVENTION SECONDARY PREVENTION TERTIARY PREVENTION


LEVELS OF APPLICATION of PREVENTIVE MEASURES
THE NATURAL HISTORY OF OMSK
The course of the disease in man DEATH

Interrelations of the various Chronic


state
AGENT: PYOGENIC BACTERIA
Disability
DEAFNESS
HOST: HUMAN illness
(IMMUNOLOGIC, MASTOIDITIS
AGE, NUTRITION Defect
OMSK
Signs and
ENVIRONMENT: CLINICAL HORIZON symptom :
(HYGIENE, URTI / OMA
SANIATION ssand
Tissue
physiologic URTI
Bring AGENT and Immunity and
changes resistance
HOST together
STIMULUS and AGENT
STIMULUS or BECOMES established and Eustachian tube
AGENT becomes increases by multiplication or resistance RECOVERY
Or Produce a increment
disease-provoking

STIMULUS Interactions of HOST HOST REACTION


in and SIMULUS
huma Discernible
Early Advanced
n pathogenesis early lesions Convalescence
HOST disease

PREPATHOGENESIS
PERIOD PERIOD OF PAT H O G E N E S I S
THE NATURAL HISTORY OF OMSK
Interrelation of Agent, Host, and Environmental Reaction of the HOST to the STIMULUS
factors
Early URTI/OMA OMSK/MAS
Production of STIMULUS
pathogenesis TOIDITIS Convalescence

PREPATHOGENESIS PERIOD PERIOD OF PATHOGENESIS

EARLY DIAGNOSIS and REHABILITATION


HEALTH PROMOTION PROMPT TREATMENT
Provision of hospital
Early/ adequate and community facilities
treatment of URTI DISABILITY LIMITATION for retraining and
Health education
Adequate Treatment of education for
Good standard OMA maximum use
of nutrition Adequate treatment of remaining capacities
SPECIFIC PROTECTION to arrest
Provision of adequate Objectives : the disease process Education of the public
Housing Avoid recurrent URTI To cure and prevent and to prevent and industry to utilized
disease processes further complication the rehabilitated
Hygiene, sanitation and sequelae as full employment

To prevent complications Provision of Selective placement


and sequele facilities to limit
disability Work therapy in hospitals
To shorten period of disability and to prevent death
Use of sheltered colony

PRIMARY PREVENTION SECONDARY PREVENTION TERTIARY PREVENTION


LEVELS OF APPLICATION of PREVENTIVE MEASURES
Levels of prevention
(diabetic retinopathy)

Primary prevention:
Health promotion
Balance diet, exercise (healthy life)
Specific protection:
For high risk : low GI diet, exercise
Secondary Prevention
Early diagnosis & prompt treatment:
- DM screening
- Adequate and continuing treatment
- Avoid complication:
(microvascular, macrovascular) through
maintenance normal blood sugar
to avoid hyperglycemia or hypoglycemia
DM diet
Tertiary prevention
- Disability limitation
Prevent from blindness caused by
diabetic retinopathy through DM control

- Rehabilitation: to save and rehabilitated


vision as much as possible.
Health promotion

Stroke Clinical
Heart/CV care
disease

Time Smoking Quit

Community Tobacco
environment control

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