TRANSIENT ISCHEMIC

ATTACK
Batcagan, Glenda
Bernard Maverick
Labi, John Alfred
Lacdao, Dona Dee
 INTRODUCTION
 Transient ischemic attack is also known as a
mini-stroke, a hemorrhagic stroke, or an
ischemic stroke. Generally, a TIA happens
when platelets in the blood clump together in
your arteries (a blood clot) making blood flow
to a part of the brain be blocked or reduced.
After a short time, blood flows again and the
symptoms go away.
 CASE FOCUS:

MRS. J
 Personal History
 Mrs. J was born in Kapangan, Benguet on
December 19, 1950. She is 60 years old, a
Filipino citizen, married and has four children.
She lives with her family, residing at Pico, La
Trinidad, Benguet. She is a chronic smoker and
an occasional drinker. She is preoccupied with
heavy labor in the garden subjecting her to
consistent stress. She was admitted in the local
hospital on August 1, 2010 at 7:00 in the evening
with a chief complaint of sudden onset dyspnea
followed by vomiting and syncope.
 Family Health-Illness History
 Mrs. J’s reported that several of her relatives on
her paternal side including her father have
hypertension. Other than this, there were no
other reported illnesses within her family and
relatives.
 History of Past Illness
 Mrs. J was diagnosed with hypertension in her
late 40’s. She was prescribed with Losartan for
maintenance. Other than that, there were no
known related ailments and past illnesses with
the present one.
 History of Present Illness
 Chief Complaint: Sudden onset dyspnea
followed by vomiting and syncope.
 Sequence of appearance of S&S: During
dinner, Mrs. J suddenly experience
dyspnea. Shortly after, she vomited,
experienced left-sided weakness
followed by loss of consciousness. She was
brought to the hospital in a drowsy
state and was admitted at 7:00 in the evening.
PATHOPHYSIOLOGY
 Predisposing factors Precipitating factors

- Age - sex - cigarette smoking

- history of TIA/stroke & HTN on - stress

the family
- increased fatty food on diet
- personal HTN

Formation of plaque deposits Atherosclerosis

Thrombosis

Hypertension Occlusion of major vessels

Cerebral Hypoperfusion

Impaired distribution of oxygen and

glucose

Tissue hypoxia and cellular

starvation

Cerebral Ischemia (TIA)

PATHOPHYSIOLOGY
 IVF (PNSS 1L X 8HRS)
 D5H20 500mL + 2 amps Chlonidine
 Activityc