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(VSD)
Small VSDs, the chest radiograph is usually normal
Large VSD: The presence of right ventricular hypertrophy, olegeimic lung fields
(pulmonary hypertension or an associated pulmonic stenosis), gross
cardiomegaly with prominence of both ventricles, the left atrium.
Ventricular Septal defects
30–50% of small defects close spontaneously, most
frequently during the 1st 2 yr of life.
Small muscular VSDs are more likely to close (up to 80%)
than membranous VSDs are (up to 35%).
infants with large defects have repeated episodes of
respiratory infection and heart failure despite optimal
medical management.
Surgical repair prior to development of an irreversible
increase in pulmonary vascular resistance (usually prior to
the patient's second birthday).
Atrial Septal Defect
Enlargement of the
right ventricle
Enlargement of atrium
Large pulmonary artery
increased pulmonary
vascularity is.
Primary Pulmonary Hypertension
Prominent pulmonary
artery.
Prominent right
ventricle
Prominent vascularity
in the hilar areas
Decreased vascualr
marking in the
periphery.
No treatment
Tetralogy of Fallot
Ventricular septal
defect
Pulmonic stenosis
Overriding aorta
Right ventricular
hypertrophy
Cyanotic
Congestive Cardiac Failure
Enlarged heart
Plethoric lung fields
specially at bases
Boot shaped heart in pt. with
TOF
Snow man or figure8 in pt. with TAPVR
Situs inverses
Aortic Coarctation
10-day old girl with CHF; 8 cc contrast,
3D CT
CT
Patent Ductus
Arteriosus
CT MR
Shunt Lesion: Septal Defects
ASD
ASD/VSD
Post ASD repair
normal frontal view
L
AV
MV
AV
MV
Source Undetermined
Enlarged
Right Atrium
Source Undetermined
Source Undetermined
Enlarged
Right Ventricle
Source Undetermined
Source Undetermined
Enlarged
Right Ventricle
(lateral CXR)
Source Undetermined
Source Undetermined
Enlarged
Left Atrium
Source Undetermined
Source Undetermined
Enlarged
Left Atrium
calcified
mitral Source Undetermined
annulus
Source Undetermined
Enlarged
Left Ventricle
Source Undetermined
Source Undetermined
54 year old with dyspnea on exertion
a) pulmonic stenosis
b) ASD
c) fluid overload
d) primary PHTN
Source Undetermined
24 year-old
with heart
murmur
a) pulmonic stenosis
b) ASD
c) fluid overload
d) primary PHTN
Source Undetermined
Small Heart
Pericardial or Myocardial
Disease?
Use physical exam to
Pericardial Calcifications
differentiate
Small Heart
Pericardial Effusion
– “Oreo” Sign
Fluid collection between epicardial and
retrosternal fat pads
Visible Borders of
Mediastinum
Pericardial Effusion
Myocardial Failure or Pericardial
Effusion?
Globally enlarged heart
Narrow VPW
Myocardial Failure
Gehlbach, Brian K., et al. The Pulmonary Manifestations of Left Heart Failure. Chest. 2004; 125: 669-682.
Valvular Disease =
Unequal chamber enlargement
Aortic Stenosis
flow with normal flow
distribution
– Narrow vascular pedicle
– Increased LVP (may have mild
LV enlargement)
– Post stenotic dilation of aorta
Atrial Septal Defect…Why?
LUL Oligemia
rrow VPW
Larger vessels
Small Vessels
Evaluate the Vessels:
Cardiogenic Edema
Cardiogenic edema occurs secondary to
hydrostatic forces and therefore
predominately occurs in the lower lobes
Most commonly secondary to left heart
failure (acute or chronic)
Vascular indistinctness
Which represents edema?
http://radiographics.rsnajnls.org/cgi/content/full/21/4/1047
Tags and Perfusion
Coronary Artery Imaging
More Angiograms
RCA: MR and Conventional
Fast Spin-Echo
Ct angio
(a) RAO view along the interventricular groove shows
LAD, with mixed atherosclerotic lesion (arrowhead)
with calcified components in the proximal course of
the vessel.
(b) LAO view in plane RCA with calcified nodules
(arrowheads) along the course of the vessel.
(c) LAO "spider" view shows (LAD and its diagonal
branches, with soft-tissue-attenuation plaque
(arrowhead) in the anterior aspect of the left main
coronary artery (LM) wall.