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Learning Outcomes
Summarize the process of evaluating ECG tracings and determining the presence of dysrhythmias. Identify criteria used for classification of dysrhythmias. Describe various rhythms and dysrhythmias. Identify the dysrhythmias using the criteria for classification.
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Explain how dysrhythmias may affect the patient. Discuss basic patient care and treatment for dysrhythmias. Describe an electronic pacemaker, its function, and the normal pacemaker rhythm characteristics. Identify the steps in evaluating electronic pacemaker function on an ECG tracing.
Chapter 5 3
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Heart Block Rhythms Bundle Branch Block Dysrhythmias Rhythms Originating from the Ventricles Electronic Pacemaker Rhythms
Chapter 5
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Occur when electrical activity has difficulty traveling along normal conduction pathway Ventricular depolarization is delayed or absent Three levels of heart blocks
1st degree heart block 2nd degree AV block, Mobitz I (Wenckebach) 2nd degree AV block, Mobitz II 3rd degree block
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Delay in electrical conduction around the AV node Electrical stimulus is prevented from traveling to ventricular conduction system
Chapter 5
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Rhythm regularity between P-P interval and R-R interval is constant. Rate normal, 60-100 beats per minute P wave configuration
Same configuration and shape P wave occurs before each QRS complex
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PR interval greater than 0.20 second QRS duration normal 0.06-0.10 second
Chapter 5
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Monitor and report further degeneration and development of other heart blocks
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Some electrical impulses are blocked/non-conducted at AV junction region AV node conducts electrical impulse to ventricular conduction pathway until it fails, then resets in a repetitious pattern The ventricular rhythm is irregular
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Rhythm
P-P interval regular R-R interval irregular due to blocked impulses Atrial rate within normal limits Ventricular rate slower than atrial rate
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Rate
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P wave configuration
Normal size, upright One P wave for every QRS, with additional P waves
PR interval varies, starts short, gets progressively longer until QRS wave is dropped, then cycle is restarted QRS duration normal 0.06-0.10 second
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Patient may not exhibit signs of low cardiac output until rate decreases to 40 beats per minute or lower Condition usually results from inflammation around AV node and is often temporary
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If patient exhibits signs of low cardiac output, notify licensed practitioner If no low cardiac output signs are exhibited, monitor patient for progression to third degree heart block
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Referred to as the classical heart block AV node selects which electrical impulses to block No pattern or reason for dropping QRS complex exists Frequently progresses to third degree AV block
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Rhythm
P-P interval is regular R-R interval is irregular Atrial rate is within normal limits Ventricular rate is slower than atrial rate
Rate
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P wave configuration
Normal size Upright One P wave for every QRS complex, but additional P waves
PR interval constant, even after QRS drop occurs QRS duration normal, 0.06-0.10 second
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Troubleshooting
In second degree type I, the PR interval in front of the dropped QRS complex is longer than the PR interval behind the dropped QRS complex In second degree type II, the interval is constant Both rhythms should be reported
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Observe patient for signs of low cardiac output Condition can progress quickly to third degree AV block or complete heart block
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Also known as complete heart block (CHB) All electrical impulses originating above the ventricles are blocked No correlation exists between atrial and ventricular depolarization
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Rhythm
P-P interval is regular R-R interval is regular, but different from P-P interval Atrial rate is 60 to 100 beats per minute Ventricular is 20 to 40 beats per minute
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Rate
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P wave configuration
Normal size and shape, but may be buried in QRS complex Number of P waves does not correlate to number of QRS complexes P waves outnumber QRS complexes
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PR interval
Intervals will vary A long PR interval followed by a short PR interval indicates complete heart block Constant May be within normal limits or wider depending on the location of the block
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QRS duration
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Patient will show signs of low cardiac output and may be unconscious Initiate Code Blue when patient shows first signs of low cardiac output Immediate medical intervention may be required temporary pacemaker Rhythm strips should be mounted and identified in patients record
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Occur when one or both ventricular pathways are damaged Ventricle with damaged pathway receives current one cell at a time Longer contraction time reflected in wider QRS complex
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Right side of the conduction pathway is blocked after the His bundle Conduction travels to left ventricle first, then moves to the right side Chapter 5
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Left conduction pathway is blocked Conduction travels to right ventricle first, then moves to the left side
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P wave configuration shape, configuration, deflection, and coordination with QRS complex depend on basic rhythm PR interval normal, 0.12-0.20 second QRS duration 0.12 second or greater
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Patient exhibits normal effects of basic rhythm Widening of QRS complex must be reported to a licensed practitioner immediately Condition may require pacemaker or Code Blue
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Rate of automaticity is 20-40 beats per minute Current initiated in Purkinje fibers QRS duration and configuration will be 0.12 second or greater, suggesting cell-by-cell stimulation
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Premature ventricular contractions (PVC) Idioventricular rhythm Accelerated idioventricular rhythm Ventricular tachycardia (V tach) Agonal rhythm Ventricular fibrillation (V fib) Asystole
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Caused by an ectopic beat that occurs early in the cycle and originates from the ventricles
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Rhythm
P-P and R-R intervals are regular with early QRS complexes Early complex has full compensatory pause Atrial and ventricular rates are the same for the underlying rhythm Early complexes make ventricular rhythm faster than normal rhythm
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Rate
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P wave configurations
Shape is that of the underlying rhythm P waves not identified on early ventricular complex Follows underlying rhythm P wave not present in early complex
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PR interval
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QRS duration
Follows underlying rhythm Duration of early complex greater than 0.12 second QRS shape is bizarre, with T wave in opposite direction from QRS wave
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Unifocal early beat (has similar shape, suggesting only one irritable focus present) Multifocal varied shapes and forms of the PVCs Interpolated PVC occurs during the normal R-R interval without interrupting the normal cycle. Occasional more than one to four PVCs per minute Frequent more than five to seven PVCs per minute
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Bigeminy every other beat is a PVC Trigeminy every third beat is a PVC Quadrigeminy every fourth beat is a PVC R on T PVCs PVC occurs on the T wave or the vulnerable period of the ventricular refractory period Coupling two PVCs occur back to back
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Significance depends on their frequency Can occur in normal hearts May feel dizziness or other symptoms of low cardiac output R on T PVCs and coupling increase risk of a more serious dysrhythmia Notify practitioner if symptoms of low cardiac output develop
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Idioventricular Rhythm
Impulse created by the ventricular pacemaker Presents with the classic wide QRS complex, slow ventricular rate and absent P waves
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Rhythm
R-R interval is regular, P-P interval cannot be determined Ventricular rate is 20-40 beats per minute
Rate
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Profound loss of cardiac output The patient will likely be unconscious Notify health care practitioner immediately Likely to require medication and/or pacing
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Impulse created by the ventricular pacemaker The heart rate is faster than an idioventricular rhythm QRS complex is wide and bizarre and P waves are absent
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Rhythm
R-R interval is regular. P-P interval cannot be determined Ventricular rate is 40-100 beats per minute
Rate
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Decrease in cardiac output due to slow ventricular rate Patient may be unconscious Notify health care practitioner May require medication and/or pacing
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Three or more PVCs occur in a row Ventricles are in continuous state of contraction-relaxation
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Rhythm
P-P interval usually not identifiable R-R interval usually regular, can be slightly irregular at times Atrial rate cannot be determined Ventricular rate 100-200 beats per minute
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Rate
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Greater than 0.12 second T wave in opposite direction (usually down) from QRS complex
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Lost atrial kick and decreased ventricular filling time results in decreased cardiac output Approximately 50% of patients become unconscious immediately Notify licensed practitioner
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If patient is unresponsive, issue Code Blue, begin CPR, use emergency equipment Mount rhythm strips in patients medical record If patient is responsive, licensed practitioner may initiate treatment plan
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Troubleshooting
Heart rates less than 60 beats per minute can cause escape beats Often PVCs occur in response to bradycardia
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Agonal Rhythm
Occurs when all of the pacemakers in the heart have failed Ventricular rate is less than 20 beats per minute
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Rhythm
Rate is less than 20 beats per minute P wave is absent PR interval cannot be determined QRS duration and configuration
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Profound loss of cardiac output Patient will be unconscious Notify health care practitioner immediately This is a medical emergency; BLS and ACLS interventions will be initiated
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Safety
Follow facilitys guidelines on stocking of crash cart for Code Blue emergencies
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Chaotic asynchronous electrical activity within ventricular tissue results in no cardiac output
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Rhythm
R-R interval, if able to determine, will be irregular Ventricular rate, if identifiable, will be greater than 300 beats per minute
Rate
P wave configurations not identifiable PR interval not identifiable QRS duration cannot be determined
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If patient is conscious and talking, leads are loose or detached Patient will be unresponsive Code Blue situation is present Begin CPR and emergency measures immediately Advanced cardiac life support (ACLS) should begin immediately
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Asystole
Often called straight line or flat line No electrical activity is present in the myocardium
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Asystole Criteria
Rhythm no waveforms are present Rate no atrial or ventricular rates are present P wave configurations no P waves are present PR interval none, since no waveforms are present QRS duration not measurable, no waveforms are present
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If family/friends are present, explain that there is an emergency and escort them out of immediate area
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Also known as artificial pacemakers Deliver electrical impulse to myocardium, causing cells to depolarize Can pace the atria, ventricles, or both Usually implanted under the skin
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Performing ECG on patient with pacemaker may require additional training Each pacemaker is individually set for the desired heart rate and electrical current Atrial pacing is used alone when the AV node and ventricular conduction are performing correctly
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Pacemaker Safety
Electrical current from pacemaker cannot be transmitted to those who encounter the patient
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Pacing spike
Thin spike on ECG tracing indicating electrical current from pacemaker After spike, either a P wave or QRS complex will appear
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AV delay
Similar to PR interval on normal rhythm tracing Measured from atrial spike to ventricular spike Normally 0.12-0.20 second If patient has normal P wave and pacerinduced ventricular complex, measure from beginning of P wave to ventricular spike
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Step 1 What are the rate and regularity of the paced rhythm? Step 2 What are the rate and regularity of the intrinsic rhythm? Step 3 Is the atrial lead sensing appropriate? Step 4 Is atrial capture present?
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Step 5 Is AV delay appropriate? Step 6 Is ventricular sensing appropriate? Step 7 Is ventricular capture present? Note: If patient does not have an AV sequential pacemaker, steps 3 through 5 can be eliminated.
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Slow firing rates Less effective sensing capabilities Less than predetermined electrical current Sensing capability too low for pacemaker to see normal contractions
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Program complication
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Malfunctioning failure to pace Malsensing failure to sense Loss of capture failure to depolarize Oversensing perceiving sources other than the heart
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Your Responsibility
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Chapter 5 Summary
Evaluating an ECG requires basic knowledge of the components of the rhythm The process of evaluating an ECG tracing includes determining the rhythm, rate, P wave configuration, PR interval, and QRS duration and configuration Sinus rhythm is a normally functioning rhythm Dysrhythmias are abnormal rhythms
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Many dysrhythmias can result in low cardiac output Each of the dysrhythmias may affect the patient with varying degrees of severity Pacemakers can pace the atria, ventricles or both and are seen as a spike on the ECG Evaluating a pacemaker involves seven steps, similar to a non-paced rhythm
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